Risk of Hepatocellular Carcinoma in People with HIV in the United States, 2001-2019.


Journal

Journal of the National Cancer Institute
ISSN: 1460-2105
Titre abrégé: J Natl Cancer Inst
Pays: United States
ID NLM: 7503089

Informations de publication

Date de publication:
23 Aug 2023
Historique:
received: 22 03 2023
revised: 06 07 2023
accepted: 17 08 2023
medline: 23 8 2023
pubmed: 23 8 2023
entrez: 23 8 2023
Statut: aheadofprint

Résumé

People with HIV (PWH) have higher risk of hepatocellular carcinoma (HCC) than the general population, partly due to higher prevalence of coinfection with hepatitis B virus (HBV) or C virus (HCV). We calculated standardized incidence ratios for HCC in PWH by comparing rates from PWH in the HIV/AIDS Cancer Match Study, a population-based HIV and cancer registry linkage, to those in the general population. We used multivariable Poisson regression to estimate adjusted incidence rate ratios (aIRRs) among PWH and linked the Texas HIV registry with medical claims data to estimate adjusted odds ratios (aORs) of HBV and HCV in HCC cases with logistic regression. Compared to the general population, HCC rates in PWH were elevated 2.79-fold (n = 1,736; 95%CI 2.66-2.92). HCC rates decreased significantly from 2001-2004 to 2015-2019 (p < 0.001). Compared to men who had sex with men (MSM), HCC risk was elevated 4.28-fold among men who injected drugs (95%CI 3.72-4.93) and 1.83-fold among women who injected drugs (95%CI 1.49-2.26). In Texas, 146 HCC cases amongst PWH were linked to claims data; 25% HBV-positive, 59% HCV-positive, and 13% co-infected with HBV and HCV. Compared to MSM, people who inject drugs (PWID) had 82% decreased odds of HBV (aOR 0.18; 95%CI (0.05 - 0.63), and 2-times the odds of HCV (aOR 20.4; 95%CI 3.32-125.3). During 2001-2019, HCC risk declined among PWH, though rates remain significantly elevated compared to the general population, particularly among PWID. Prevention and treatment of HBV/HCV are needed to reduce HCC risk among PWH.

Sections du résumé

BACKGROUND BACKGROUND
People with HIV (PWH) have higher risk of hepatocellular carcinoma (HCC) than the general population, partly due to higher prevalence of coinfection with hepatitis B virus (HBV) or C virus (HCV).
METHODS METHODS
We calculated standardized incidence ratios for HCC in PWH by comparing rates from PWH in the HIV/AIDS Cancer Match Study, a population-based HIV and cancer registry linkage, to those in the general population. We used multivariable Poisson regression to estimate adjusted incidence rate ratios (aIRRs) among PWH and linked the Texas HIV registry with medical claims data to estimate adjusted odds ratios (aORs) of HBV and HCV in HCC cases with logistic regression.
RESULTS RESULTS
Compared to the general population, HCC rates in PWH were elevated 2.79-fold (n = 1,736; 95%CI 2.66-2.92). HCC rates decreased significantly from 2001-2004 to 2015-2019 (p < 0.001). Compared to men who had sex with men (MSM), HCC risk was elevated 4.28-fold among men who injected drugs (95%CI 3.72-4.93) and 1.83-fold among women who injected drugs (95%CI 1.49-2.26). In Texas, 146 HCC cases amongst PWH were linked to claims data; 25% HBV-positive, 59% HCV-positive, and 13% co-infected with HBV and HCV. Compared to MSM, people who inject drugs (PWID) had 82% decreased odds of HBV (aOR 0.18; 95%CI (0.05 - 0.63), and 2-times the odds of HCV (aOR 20.4; 95%CI 3.32-125.3).
CONCLUSIONS CONCLUSIONS
During 2001-2019, HCC risk declined among PWH, though rates remain significantly elevated compared to the general population, particularly among PWID. Prevention and treatment of HBV/HCV are needed to reduce HCC risk among PWH.

Identifiants

pubmed: 37610358
pii: 7248913
doi: 10.1093/jnci/djad172
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Published by Oxford University Press 2023.

Auteurs

Jennifer K McGee-Avila (JK)

Infections and Immunoepidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, MD, USA.

Ilona Argirion (I)

Infections and Immunoepidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, MD, USA.

Eric A Engels (EA)

Infections and Immunoepidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, MD, USA.

Thomas R O'Brien (TR)

Infections and Immunoepidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, MD, USA.

Marie-Josèphe Horner (MJ)

Infections and Immunoepidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, MD, USA.
Trans- Divisional Research Program, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, MD, USA.

Baozhen Qiao (B)

Bureau of Cancer Epidemiology, New York State Department of Health, Albany, NY, USA.

Analise Monterosso (A)

HIV/STD/HCV Epidemiology and Surveillance Unit, Texas Department of State Health Services, Austin, TX, USA.

Qianlai Luo (Q)

Infections and Immunoepidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, MD, USA.

Meredith S Shiels (MS)

Infections and Immunoepidemiology Branch, Division of Cancer Epidemiology and Genetics, National Cancer Institute, Rockville, MD, USA.

Classifications MeSH