Human 'knockouts' of CSF3 display severe congenital neutropenia.

GCSF neutropenia recurrent infection

Journal

British journal of haematology
ISSN: 1365-2141
Titre abrégé: Br J Haematol
Pays: England
ID NLM: 0372544

Informations de publication

Date de publication:
Nov 2023
Historique:
revised: 07 08 2023
received: 12 06 2023
accepted: 08 08 2023
pubmed: 24 8 2023
medline: 24 8 2023
entrez: 23 8 2023
Statut: ppublish

Résumé

Colony-stimulating factor 3 (CSF3) is a key factor in neutrophil production and function, and recombinant forms have been used clinically for decades to treat congenital and acquired neutropenia. Although biallelic inactivation of its receptor CSF3R is a well-established cause of severe congenital neutropenia (SCN), no corresponding Mendelian disease has been ascribed to date to CSF3. Here, we describe three patients from two families each segregating a different biallelic inactivating variant in CSF3 with SCN. Complete deficiency of CSF3 as a result of nonsense-mediated decay (NMD) could be demonstrated on RT-PCR using skin fibroblasts-derived RNA. The phenotype observed in this cohort mirrors that documented in mouse and zebrafish models of CSF3 deficiency. Our results suggest that CSF3 deficiency in humans causes a novel autosomal recessive form of SCN.

Identifiants

pubmed: 37612131
doi: 10.1111/bjh.19054
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

477-480

Informations de copyright

© 2023 British Society for Haematology and John Wiley & Sons Ltd.

Références

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Auteurs

Ebtissal Khouj (E)

Department of Translational Genomics, Center for Genomic Medicine, King Faisal Specialist Hospital and Research Center, Riyadh, Saudi Arabia.

Dana Marafi (D)

Department of Pediatrics, Faculty of Medicine, Kuwait University, Safat, Kuwait.
Department of Pediatrics, Adan Hospital, Ministry of Health, Hadiya, Kuwait.
Kuwait Medical Genetics Centre, Ministry of Health, Sulaibikhat, Kuwait.

Bayan Aljamal (B)

Department of Translational Genomics, Center for Genomic Medicine, King Faisal Specialist Hospital and Research Center, Riyadh, Saudi Arabia.

Anwar Hajiya (A)

Department of Pediatrics, Adan Hospital, Ministry of Health, Hadiya, Kuwait.

Reem M Elshafie (RM)

Kuwait Medical Genetics Centre, Ministry of Health, Sulaibikhat, Kuwait.

Mais O Hashem (MO)

Department of Translational Genomics, Center for Genomic Medicine, King Faisal Specialist Hospital and Research Center, Riyadh, Saudi Arabia.

Firdous Abdulwahab (F)

Department of Translational Genomics, Center for Genomic Medicine, King Faisal Specialist Hospital and Research Center, Riyadh, Saudi Arabia.

Amal Jaafar (A)

Department of Translational Genomics, Center for Genomic Medicine, King Faisal Specialist Hospital and Research Center, Riyadh, Saudi Arabia.

Tarfa Alshidi (T)

Department of Translational Genomics, Center for Genomic Medicine, King Faisal Specialist Hospital and Research Center, Riyadh, Saudi Arabia.

Ashraf H Aboelanine (AH)

Kuwait Medical Genetics Centre, Ministry of Health, Sulaibikhat, Kuwait.

Ali Awaji (A)

Genetic Center, Prince Mohammed bin Nasser Hospital, Jazan, Saudi Arabia.

Fowzan S Alkuraya (FS)

Department of Translational Genomics, Center for Genomic Medicine, King Faisal Specialist Hospital and Research Center, Riyadh, Saudi Arabia.

Classifications MeSH