Metformin Can Attenuate Beta-Cell Hypersecretion-Implications for Treatment of Children with Obesity.

OGTT childhood obesity free fatty acids glucose-stimulated insulin secretion human islets hyperinsulinemia metformin

Journal

Metabolites
ISSN: 2218-1989
Titre abrégé: Metabolites
Pays: Switzerland
ID NLM: 101578790

Informations de publication

Date de publication:
04 Aug 2023
Historique:
received: 06 07 2023
revised: 01 08 2023
accepted: 01 08 2023
medline: 25 8 2023
pubmed: 25 8 2023
entrez: 25 8 2023
Statut: epublish

Résumé

In children with obesity, insulin hypersecretion is proposed to precede insulin resistance. We investigated if metformin could be used to attenuate insulin secretion from palmitate-treated isolated islets and its implication for children with obesity. Human islets were exposed to palmitate for 0.5 or 1 day, when metformin was introduced. After culture, glucose-stimulated insulin secretion (GSIS) was measured. Children with obesity, who had received metformin for over six months (

Identifiants

pubmed: 37623862
pii: metabo13080917
doi: 10.3390/metabo13080917
pmc: PMC10456302
pii:
doi:

Types de publication

Journal Article

Langues

eng

Subventions

Organisme : European Commission's Seventh Framework Programme
ID : 279153
Organisme : Sweden's innovation agency
ID : 2020-02417
Organisme : EXODIAB (Swedish Research Council)

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Auteurs

Quan Wen (Q)

Department of Medical Cell Biology, Uppsala University, 75123 Uppsala, Sweden.
Department of Women's and Children's Health, Uppsala University, 75185 Uppsala, Sweden.

Rasmus Stenlid (R)

Department of Medical Cell Biology, Uppsala University, 75123 Uppsala, Sweden.
Department of Women's and Children's Health, Uppsala University, 75185 Uppsala, Sweden.
Overweight Unit, Academic Children's Hospital, Uppsala University, 75185 Uppsala, Sweden.

Azazul Islam Chowdhury (AI)

Department of Medical Cell Biology, Uppsala University, 75123 Uppsala, Sweden.

Iris Ciba (I)

Department of Medical Cell Biology, Uppsala University, 75123 Uppsala, Sweden.
Department of Women's and Children's Health, Uppsala University, 75185 Uppsala, Sweden.
Overweight Unit, Academic Children's Hospital, Uppsala University, 75185 Uppsala, Sweden.

Banu Aydin (B)

Department of Medical Cell Biology, Uppsala University, 75123 Uppsala, Sweden.
Department of Women's and Children's Health, Uppsala University, 75185 Uppsala, Sweden.

Sara Y Cerenius (SY)

Department of Medical Cell Biology, Uppsala University, 75123 Uppsala, Sweden.
Department of Women's and Children's Health, Uppsala University, 75185 Uppsala, Sweden.

Hannes Manell (H)

Department of Women's and Children's Health, Uppsala University, 75185 Uppsala, Sweden.
Overweight Unit, Academic Children's Hospital, Uppsala University, 75185 Uppsala, Sweden.

Anders Forslund (A)

Department of Medical Cell Biology, Uppsala University, 75123 Uppsala, Sweden.
Department of Women's and Children's Health, Uppsala University, 75185 Uppsala, Sweden.
Overweight Unit, Academic Children's Hospital, Uppsala University, 75185 Uppsala, Sweden.

Peter Bergsten (P)

Department of Medical Cell Biology, Uppsala University, 75123 Uppsala, Sweden.
Department of Women's and Children's Health, Uppsala University, 75185 Uppsala, Sweden.
Overweight Unit, Academic Children's Hospital, Uppsala University, 75185 Uppsala, Sweden.

Classifications MeSH