The stem region of group A transferase is crucial for its specificity, and its alteration promotes heterologous Forssman synthase activity.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
26 08 2023
Historique:
received: 15 02 2023
accepted: 17 08 2023
medline: 28 8 2023
pubmed: 27 8 2023
entrez: 26 8 2023
Statut: epublish

Résumé

Some stem region mutants of human blood group A transferase (hAT) possess Forssman synthase (FS) activity, but very little is known about the mechanisms responsible for this enzymatic crosstalk. We performed confocal microscopy and image analysis to determine whether different intra-Golgi localization was accountable for this acquired activity. We also performed structural modeling and mutational and normal mode analyses. We introduced new mutations in the stem region and tested its FS and AT activities. No differences in subcellular localization were found between hAT and FS-positive mutants. AlphaFold models of hAT and mFS (mouse Forssman synthase) showed that the hAT stem region has a tether-like stem region, while in mFS, it encircles its catalytic domain. In silico analysis of FS-positive mutants indicated that stem region mutations induced structural changes, decreasing interatomic interactions and mobility of hAT that correlated with FS activity. Several additional mutations introduced in that region also bestowed FS activity without altering the AT activity: hAT 37-55 aa substitution by mFS 34-52, 37-55 aa deletion, and missense mutations: S46P, Q278Y, and Q286M. Stem region structure, mobility, and interactions are crucial for hAT specificity. Moreover, stem region mutations can lead to heterologous Forssman activity without changes in the catalytic machinery.

Identifiants

pubmed: 37634031
doi: 10.1038/s41598-023-40900-4
pii: 10.1038/s41598-023-40900-4
pmc: PMC10460411
doi:

Substances chimiques

globoside alpha-N-acetylgalactosaminyltransferase EC 2.4.1.88

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

13996

Informations de copyright

© 2023. Springer Nature Limited.

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Auteurs

Emili Cid (E)

Laboratory of Immunohematology and Glycobiology, Josep Carreras Leukaemia Research Institute, Ctra. de Can Ruti, Cami de Les Escoles S/N, 08916, Badalona, Spain. ecid@carrerasresearch.org.

Miyako Yamamoto (M)

Laboratory of Immunohematology and Glycobiology, Josep Carreras Leukaemia Research Institute, Ctra. de Can Ruti, Cami de Les Escoles S/N, 08916, Badalona, Spain.

Laura Barrero (L)

Laboratory of Immunohematology and Glycobiology, Josep Carreras Leukaemia Research Institute, Ctra. de Can Ruti, Cami de Les Escoles S/N, 08916, Badalona, Spain.

Fumiichiro Yamamoto (F)

Laboratory of Immunohematology and Glycobiology, Josep Carreras Leukaemia Research Institute, Ctra. de Can Ruti, Cami de Les Escoles S/N, 08916, Badalona, Spain.

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Classifications MeSH