Over diagnosis of bradykinin angioedema in patients treated with angiotensin-converting enzyme inhibitors or angiotensin II receptor blockers.

Angioedema Angiotensin converting enzyme inhibitors Bradykinin Mast-cell Urticaria

Journal

The World Allergy Organization journal
ISSN: 1939-4551
Titre abrégé: World Allergy Organ J
Pays: United States
ID NLM: 101481283

Informations de publication

Date de publication:
Aug 2023
Historique:
received: 24 04 2023
revised: 31 07 2023
accepted: 31 07 2023
medline: 28 8 2023
pubmed: 28 8 2023
entrez: 28 8 2023
Statut: epublish

Résumé

Bradykinin angioedemas are a potentially serious side effect of angiotensin-converting enzyme inhibitors (ACEI) and more controversially of angiotensin II receptor blockers (ARB). Their challenging diagnosis is based on the absence of any recurrence after more than 6 months of drug discontinuation; otherwise mast-cell driven angioedemas as a differential diagnosis must be considered. The aim of this study was to determine the prevalence of recurrent angioedema in patients referred for ACEI/ARB-induced bradykinin angioedema, after more than 6 months of drug discontinuation. We included ACEI/ARB-treated patients referred for angioedema(s) without hives and unresponsive to antihistamines, after they discontinued ACEI/ARB for at least 6 months. Any C1-inhibitor deficiency was excluded. The primary endpoint was the prevalence of patients with recurrent angioedema after more than 6 months of drug discontinuation and/or developing hives during follow-up. The secondary endpoint was the identification of epidemiological factors associated with any final diagnosis. Thirty-eight of 93 patients (41%) with a suspicion of ACEI/ARB-induced bradykinin angioedema still had recurrent angioedema (n = 27) or developed hives (n = 2) or both (n = 9) after 6 months of drug discontinuation. Good response to icatibant and facial but not oral localization were predictive for the final diagnosis of ACEI/ARB-induced bradykinin angioedema and mast-cell driven angioedema, respectively. In patients referred for acquired angioedema without wheals occurring during ACEI/ARB therapy, 59% finally had a diagnosis of ACEI/ARB-induced bradykinin angioedema whereas 41% were rather diagnosed with mast-cell driven angioedema. The overdiagnosis of ACEI/ARB-induced bradykinin angioedema may deteriorate the management of severe cardiovascular conditions.

Sections du résumé

Background UNASSIGNED
Bradykinin angioedemas are a potentially serious side effect of angiotensin-converting enzyme inhibitors (ACEI) and more controversially of angiotensin II receptor blockers (ARB). Their challenging diagnosis is based on the absence of any recurrence after more than 6 months of drug discontinuation; otherwise mast-cell driven angioedemas as a differential diagnosis must be considered.
Objective UNASSIGNED
The aim of this study was to determine the prevalence of recurrent angioedema in patients referred for ACEI/ARB-induced bradykinin angioedema, after more than 6 months of drug discontinuation.
Methods UNASSIGNED
We included ACEI/ARB-treated patients referred for angioedema(s) without hives and unresponsive to antihistamines, after they discontinued ACEI/ARB for at least 6 months. Any C1-inhibitor deficiency was excluded. The primary endpoint was the prevalence of patients with recurrent angioedema after more than 6 months of drug discontinuation and/or developing hives during follow-up. The secondary endpoint was the identification of epidemiological factors associated with any final diagnosis.
Results UNASSIGNED
Thirty-eight of 93 patients (41%) with a suspicion of ACEI/ARB-induced bradykinin angioedema still had recurrent angioedema (n = 27) or developed hives (n = 2) or both (n = 9) after 6 months of drug discontinuation. Good response to icatibant and facial but not oral localization were predictive for the final diagnosis of ACEI/ARB-induced bradykinin angioedema and mast-cell driven angioedema, respectively.
Conclusion UNASSIGNED
In patients referred for acquired angioedema without wheals occurring during ACEI/ARB therapy, 59% finally had a diagnosis of ACEI/ARB-induced bradykinin angioedema whereas 41% were rather diagnosed with mast-cell driven angioedema. The overdiagnosis of ACEI/ARB-induced bradykinin angioedema may deteriorate the management of severe cardiovascular conditions.

Identifiants

pubmed: 37638360
doi: 10.1016/j.waojou.2023.100809
pii: S1939-4551(23)00069-8
pmc: PMC10458346
doi:

Types de publication

Journal Article

Langues

eng

Pagination

100809

Informations de copyright

© 2023 Published by Elsevier Inc. on behalf of World Allergy Organization.

Déclaration de conflit d'intérêts

None.

Références

Eur J Emerg Med. 2017 Oct;24(5):318-325
pubmed: 28059860
Front Genet. 2022 Jul 18;13:914376
pubmed: 35923707
N Engl J Med. 2015 Jan 29;372(5):418-25
pubmed: 25629740
Chest. 2006 Jan;129(1 Suppl):169S-173S
pubmed: 16428706
J Community Hosp Intern Med Perspect. 2019 Dec 14;9(6):453-459
pubmed: 32002148
Medicine (Baltimore). 2015 Nov;94(45):e1939
pubmed: 26559262
N Engl J Med. 2010 Aug 5;363(6):532-41
pubmed: 20818888
Hypertension. 2009 Sep;54(3):516-23
pubmed: 19581505
J Allergy Clin Immunol Pract. 2017 Sep - Oct;5(5):1402-1409.e3
pubmed: 28552382
Intern Med J. 2015 Aug;45(8):821-7
pubmed: 25944565
Hypertension. 2008 Jun;51(6):1624-30
pubmed: 18413488
JAMA. 1997 Jul 16;278(3):232-3
pubmed: 9218671
Hypertension. 2001 Jan;37(1):46-51
pubmed: 11208755
Am J Cardiovasc Drugs. 2012 Aug 1;12(4):263-77
pubmed: 22587776
J Allergy Clin Immunol Pract. 2017 May - Jun;5(3):744-749
pubmed: 28377081
J Vet Intern Med. 2019 Mar;33(2):363-382
pubmed: 30806496
Allergy. 2014 May;69(5):602-16
pubmed: 24673465
PLoS One. 2019 Nov 11;14(11):e0224858
pubmed: 31710633
Clin Mol Allergy. 2021 Dec 6;19(1):24
pubmed: 34872575
Ann Intern Med. 2017 Jul 18;167(2):142-143
pubmed: 28554195
Hypertension. 2008 Jan;51(1):141-7
pubmed: 18025295
Arch Intern Med. 2004 Apr 26;164(8):910-3
pubmed: 15111379
CMAJ. 2006 Oct 24;175(9):1065-70
pubmed: 17060655
Rev Med Interne. 2013 Apr;34(4):209-13
pubmed: 23380506
N Engl J Med. 2008 Apr 10;358(15):1547-59
pubmed: 18378520
J Community Hosp Intern Med Perspect. 2020 Feb 10;10(1):16-18
pubmed: 32128053
Clin Exp Allergy. 2010 Jan;40(1):50-61
pubmed: 19659669
J Card Fail. 2022 May;28(5):e1-e167
pubmed: 35378257
Am J Cardiol. 2012 Aug 1;110(3):383-91
pubmed: 22521308
Allergy. 2014 Jul;69(7):868-87
pubmed: 24785199
J Allergy Clin Immunol. 2004 Sep;114(3 Suppl):S51-131
pubmed: 15356535
J Allergy Clin Immunol. 2017 Jul;140(1):242-248.e2
pubmed: 27913306
Am J Hypertens. 2004 Feb;17(2):103-11
pubmed: 14751650
Ann Allergy Asthma Immunol. 2008 Nov;101(5):495-9
pubmed: 19055203
Ann Emerg Med. 2010 Sep;56(3):278-82
pubmed: 20447725
Curr Vasc Pharmacol. 2014 Mar;12(2):223-48
pubmed: 24568157
J Intern Med. 2019 May;285(5):553-561
pubmed: 30618189
Orphanet J Rare Dis. 2018 May 4;13(1):73
pubmed: 29728119

Auteurs

Marie Douillard (M)

Department of Dermatology, St Eloi Hospital, 34000, Montpellier, France.

Zineb Deheb (Z)

Médecine Sorbonne Université, Service de Dermatologie et Allergologie, Hôpital Tenon, Assistance Publique-Hôpitaux de Paris (AP-HP), Paris, France.

Agathe Bozon (A)

Department of Dermatology, St Eloi Hospital, 34000, Montpellier, France.

Nadia Raison-Peyron (N)

Department of Dermatology, St Eloi Hospital, 34000, Montpellier, France.

Olivier Dereure (O)

Department of Dermatology, St Eloi Hospital, 34000, Montpellier, France.

Lionel Moulis (L)

Clinical and Epidemiological Research Unit, CHU, Montpellier, 34000, France.

Angèle Soria (A)

Médecine Sorbonne Université, Service de Dermatologie et Allergologie, Hôpital Tenon, Assistance Publique-Hôpitaux de Paris (AP-HP), Paris, France.
Cimi-Paris, INSERM 1135, Paris, France.

Aurélie Du-Thanh (A)

Department of Dermatology, St Eloi Hospital, 34000, Montpellier, France.

Classifications MeSH