Mechanisms and consequences of weight gain after deep brain stimulation of the subthalamic nucleus in patients with Parkinson's disease.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
30 08 2023
Historique:
received: 16 11 2022
accepted: 08 08 2023
medline: 1 9 2023
pubmed: 31 8 2023
entrez: 30 8 2023
Statut: epublish

Résumé

Body weight gain in combination with metabolic alterations has been observed after deep brain stimulation (DBS) of subthalamic nucleus (STN) in patients with Parkinson's disease (PD), which potentially counteracts the positive effects of motor improvement. We aimed to identify stimulation-dependent effects on motor activities, body weight, body composition, energy metabolism, and metabolic blood parameters and to determine if these alterations are associated with the local impact of DBS on different STN parcellations. We assessed 14 PD patients who underwent STN DBS (PD-DBS) before as well as 6- and 12-months post-surgery. For control purposes, 18 PD patients under best medical treatment (PD-CON) and 25 healthy controls (H-CON) were also enrolled. Wrist actigraphy, body composition, hormones, and energy expenditure measurements were applied. Electrode placement in the STN was localized, and the local impact of STN DBS was estimated. We found that STN DBS improved motor function by ~ 40% (DBS ON, Med ON). Weight and fat mass increased by ~ 3 kg and ~ 3% in PD-DBS (all P ≤ 0.005). fT3 (P = 0.001) and insulin levels (P = 0.048) increased solely in PD-DBS, whereas growth hormone levels (P = 0.001), daily physical activity, and VO

Identifiants

pubmed: 37648732
doi: 10.1038/s41598-023-40316-0
pii: 10.1038/s41598-023-40316-0
pmc: PMC10468527
doi:

Substances chimiques

Insulins 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

14202

Informations de copyright

© 2023. Springer Nature Limited.

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Auteurs

Julia Steinhardt (J)

Department of Neurology, University of Lübeck, Ratzeburger Allee 160, 23538, Lübeck, Germany.
Institute of Endocrinology and Diabetes, University of Lübeck, Lübeck, Germany.
Center of Brain, Behavior and Metabolism, University of Lübeck, Lübeck, Germany.

Laura Lokowandt (L)

Department of Neurology, University of Lübeck, Ratzeburger Allee 160, 23538, Lübeck, Germany.
Center of Brain, Behavior and Metabolism, University of Lübeck, Lübeck, Germany.

Dirk Rasche (D)

Department of Neurosurgery, University of Lübeck, Lübeck, Germany.

Andreas Koch (A)

Section Maritime Medicine, Naval Medical Institute, Kiel, Germany.

Volker Tronnier (V)

Department of Neurosurgery, University of Lübeck, Lübeck, Germany.

Thomas F Münte (TF)

Department of Neurology, University of Lübeck, Ratzeburger Allee 160, 23538, Lübeck, Germany.
Center of Brain, Behavior and Metabolism, University of Lübeck, Lübeck, Germany.

Sebastian M Meyhöfer (SM)

Institute of Endocrinology and Diabetes, University of Lübeck, Lübeck, Germany.
Center of Brain, Behavior and Metabolism, University of Lübeck, Lübeck, Germany.
German Center for Diabetes Research (DZD), Neuherberg, Germany.

Britta Wilms (B)

Institute of Endocrinology and Diabetes, University of Lübeck, Lübeck, Germany.
Center of Brain, Behavior and Metabolism, University of Lübeck, Lübeck, Germany.
Section Maritime Medicine, Naval Medical Institute, Kiel, Germany.

Norbert Brüggemann (N)

Department of Neurology, University of Lübeck, Ratzeburger Allee 160, 23538, Lübeck, Germany. norbert.brueggemann@uni-luebeck.de.
Center of Brain, Behavior and Metabolism, University of Lübeck, Lübeck, Germany. norbert.brueggemann@uni-luebeck.de.

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