Association of urinary arsenic with the oxidative DNA damage marker 8-hydroxy-2 deoxyguanosine: A meta-analysis.


Journal

The Science of the total environment
ISSN: 1879-1026
Titre abrégé: Sci Total Environ
Pays: Netherlands
ID NLM: 0330500

Informations de publication

Date de publication:
15 Dec 2023
Historique:
received: 14 05 2023
revised: 24 08 2023
accepted: 24 08 2023
medline: 6 11 2023
pubmed: 3 9 2023
entrez: 2 9 2023
Statut: ppublish

Résumé

The International Agency for Research on Cancer has classified arsenic as a class I carcinogen. Oxidative DNA damage is a typical early precursor to recognized malignancies. The most sensitive early independent marker of oxidative DNA damage is believed to be 8-hydroxy-2 deoxyguanosine (8-OHdG). To date, research on the link between urinary arsenic and 8-OHdG has not been consistent. This study was aimed at exploring the effects of urinary arsenic on 8-OHdG in human urine. A literature search until January 2023 was performed on the PubMed, Cochrane Library, Web of Science, Embase, and Scopus databases through a combination of computer and manual retrieval. Stata 12.0 was used to examine the degree of heterogeneity among included studies. The percentage change and 95 % confidence interval (95 % CI) of 8-OHdG were calculated between populations exposed to different doses. We used a random effect model because the degree of heterogeneity exceeded 50 %. Sensitivity analysis and testing for publication bias were performed. This meta-analysis included nine studies, most of which were performed in China. After exposure to arsenic, urinary arsenic (per 10 μg/g creatinine increase) was associated with the increased 8-OHdG (% change = 41.49 %, 95 % CI: 19.73 %, 63.25 %). Subgroup analysis indicated that the percentage change in 8-OHdG in urine was more pronounced in people exposed to arsenic <50 μg/L (% change = 24.60 %, 95 % CI: 17.35 %, 37.85 %). In studies using total urinary arsenic content as an indicator, the percentage change in 8-OHdG in urine was more significant (% change = 60.38 %, 95 % CI: 15.08 %, 105.68 %). The 8-OHdG levels in human urine significantly increased after exposure to environmental arsenic, thus suggesting that arsenic exposure is correlated with oxidative DNA damage.

Sections du résumé

BACKGROUND BACKGROUND
The International Agency for Research on Cancer has classified arsenic as a class I carcinogen. Oxidative DNA damage is a typical early precursor to recognized malignancies. The most sensitive early independent marker of oxidative DNA damage is believed to be 8-hydroxy-2 deoxyguanosine (8-OHdG). To date, research on the link between urinary arsenic and 8-OHdG has not been consistent.
OBJECTIVE OBJECTIVE
This study was aimed at exploring the effects of urinary arsenic on 8-OHdG in human urine.
METHODS METHODS
A literature search until January 2023 was performed on the PubMed, Cochrane Library, Web of Science, Embase, and Scopus databases through a combination of computer and manual retrieval. Stata 12.0 was used to examine the degree of heterogeneity among included studies. The percentage change and 95 % confidence interval (95 % CI) of 8-OHdG were calculated between populations exposed to different doses. We used a random effect model because the degree of heterogeneity exceeded 50 %. Sensitivity analysis and testing for publication bias were performed.
RESULTS RESULTS
This meta-analysis included nine studies, most of which were performed in China. After exposure to arsenic, urinary arsenic (per 10 μg/g creatinine increase) was associated with the increased 8-OHdG (% change = 41.49 %, 95 % CI: 19.73 %, 63.25 %). Subgroup analysis indicated that the percentage change in 8-OHdG in urine was more pronounced in people exposed to arsenic <50 μg/L (% change = 24.60 %, 95 % CI: 17.35 %, 37.85 %). In studies using total urinary arsenic content as an indicator, the percentage change in 8-OHdG in urine was more significant (% change = 60.38 %, 95 % CI: 15.08 %, 105.68 %).
CONCLUSION CONCLUSIONS
The 8-OHdG levels in human urine significantly increased after exposure to environmental arsenic, thus suggesting that arsenic exposure is correlated with oxidative DNA damage.

Identifiants

pubmed: 37659570
pii: S0048-9697(23)05225-7
doi: 10.1016/j.scitotenv.2023.166600
pii:
doi:

Substances chimiques

8-Hydroxy-2'-Deoxyguanosine 88847-89-6
Arsenic N712M78A8G
Deoxyguanosine G9481N71RO
Biomarkers 0

Types de publication

Meta-Analysis Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

166600

Informations de copyright

Copyright © 2023. Published by Elsevier B.V.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors declare that they have no known competing financial interest or personal relationship.

Auteurs

Donglei Wang (D)

School of Public Health, Baotou Medical College, Baotou 014030, Inner Mongolia, China.

Qingqing Liang (Q)

School of Public Health, Baotou Medical College, Baotou 014030, Inner Mongolia, China.

Dapeng Tai (D)

School of Public Health, Baotou Medical College, Baotou 014030, Inner Mongolia, China.

Yali Wang (Y)

School of Public Health, Baotou Medical College, Baotou 014030, Inner Mongolia, China.

Hongyu Hao (H)

School of Public Health, Baotou Medical College, Baotou 014030, Inner Mongolia, China.

Zhengran Liu (Z)

School of Public Health, Baotou Medical College, Baotou 014030, Inner Mongolia, China. Electronic address: 101995083@btmc.edu.cn.

Lihua Huang (L)

School of Public Health, Baotou Medical College, Baotou 014030, Inner Mongolia, China. Electronic address: huanglihua@btmc.edu.cn.

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Classifications MeSH