Bartonella infections are prevalent in rodents despite efficient immune responses.

Antigen escape Bacterial dynamics Disease ecology Ecoimmunology Host–pathogen interactions Microbial ecology Recurrent bacteremia

Journal

Parasites & vectors
ISSN: 1756-3305
Titre abrégé: Parasit Vectors
Pays: England
ID NLM: 101462774

Informations de publication

Date de publication:
04 Sep 2023
Historique:
received: 26 06 2023
accepted: 06 08 2023
medline: 6 9 2023
pubmed: 5 9 2023
entrez: 4 9 2023
Statut: epublish

Résumé

Pathogens face strong selection from host immune responses, yet many host populations support pervasive pathogen populations. We investigated this puzzle in a model system of Bartonella and rodents from Israel's northwestern Negev Desert. We chose to study this system because, in this region, 75-100% of rodents are infected with Bartonella at any given time, despite an efficient immunological response. In this region, Bartonella species circulate in three rodent species, and we tested the hypothesis that at least one of these hosts exhibits a waning immune response to Bartonella, which allows reinfections. We inoculated captive animals of all three rodent species with the same Bartonella strain, and we quantified the bacterial dynamics and Bartonella-specific immunoglobulin G antibody kinetics over a period of 139 days after the primary inoculation, and then for 60 days following reinoculation with the same strain. Contrary to our hypothesis, we found a strong, long-lasting immunoglobulin G antibody response, with protective immunological memory in all three rodent species. That response prevented reinfection upon exposure of the rodents to the same Bartonella strain. This study constitutes an initial step toward understanding how the interplay between traits of Bartonella and their hosts influences the epidemiological dynamics of these pathogens in nature.

Sections du résumé

BACKGROUND BACKGROUND
Pathogens face strong selection from host immune responses, yet many host populations support pervasive pathogen populations. We investigated this puzzle in a model system of Bartonella and rodents from Israel's northwestern Negev Desert. We chose to study this system because, in this region, 75-100% of rodents are infected with Bartonella at any given time, despite an efficient immunological response. In this region, Bartonella species circulate in three rodent species, and we tested the hypothesis that at least one of these hosts exhibits a waning immune response to Bartonella, which allows reinfections.
METHODS METHODS
We inoculated captive animals of all three rodent species with the same Bartonella strain, and we quantified the bacterial dynamics and Bartonella-specific immunoglobulin G antibody kinetics over a period of 139 days after the primary inoculation, and then for 60 days following reinoculation with the same strain.
RESULTS RESULTS
Contrary to our hypothesis, we found a strong, long-lasting immunoglobulin G antibody response, with protective immunological memory in all three rodent species. That response prevented reinfection upon exposure of the rodents to the same Bartonella strain.
CONCLUSIONS CONCLUSIONS
This study constitutes an initial step toward understanding how the interplay between traits of Bartonella and their hosts influences the epidemiological dynamics of these pathogens in nature.

Identifiants

pubmed: 37667323
doi: 10.1186/s13071-023-05918-7
pii: 10.1186/s13071-023-05918-7
pmc: PMC10478473
doi:

Substances chimiques

Immunoglobulin G 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

315

Subventions

Organisme : National Science Foundation
ID : DEB-1813069
Organisme : Israel Science Foundation
ID : 1391/15

Informations de copyright

© 2023. BioMed Central Ltd., part of Springer Nature.

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Auteurs

Ruth Rodríguez-Pastor (R)

Jacob Blaustein Center for Scientific Cooperation, The Jacob Blaustein Institutes for Desert Research, Ben-Gurion University of the Negev, Midreshet Ben-Gurion, Israel.

Adam Z Hasik (AZ)

Jacob Blaustein Center for Scientific Cooperation, The Jacob Blaustein Institutes for Desert Research, Ben-Gurion University of the Negev, Midreshet Ben-Gurion, Israel.
Institute of Evolutionary Biology, University of Edinburgh, Edinburgh, UK.

Nadav Knossow (N)

The Mitrani Department of Desert Ecology, Swiss Institute for Dryland Environmental and Energy Research, The Jacob Blaustein Institutes for Desert Research, Ben-Gurion University of the Negev, 849900, Midreshet Ben-Gurion, Israel.

Enav Bar-Shira (E)

Section of Immunology, Department of Animal Sciences, Faculty of Agricultural, Nutritional and Environmental Sciences, The Hebrew University of Jerusalem, Rehovot, Israel.

Naama Shahar (N)

The Mitrani Department of Desert Ecology, Swiss Institute for Dryland Environmental and Energy Research, The Jacob Blaustein Institutes for Desert Research, Ben-Gurion University of the Negev, 849900, Midreshet Ben-Gurion, Israel.

Ricardo Gutiérrez (R)

National Reference Center for Bacteriology, Costa Rican Institute for Research and Teaching in Nutrition and Health (INCIENSA), Cartago, Costa Rica.

Luis Zaman (L)

Department of Ecology and Evolutionary Biology, Center for the Study of Complex Systems (CSCS), University of Michigan, Ann Arbor, MI, USA.

Shimon Harrus (S)

Koret School of Veterinary Medicine, Faculty of Agricultural, Nutritional and Environmental Sciences, The Hebrew University of Jerusalem, Rehovot, Israel.

Richard E Lenski (RE)

Department of Microbiology and Molecular Genetics, Michigan State University, East Lansing, MI, USA.

Jeffrey E Barrick (JE)

Department of Molecular Biosciences, The University of Texas at Austin, Austin, TX, USA.

Hadas Hawlena (H)

The Mitrani Department of Desert Ecology, Swiss Institute for Dryland Environmental and Energy Research, The Jacob Blaustein Institutes for Desert Research, Ben-Gurion University of the Negev, 849900, Midreshet Ben-Gurion, Israel. hadashaw@bgu.ac.il.

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