Neutrophil metalloproteinase driven spleen damage hampers infection control of trypanosomiasis.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
05 09 2023
Historique:
received: 13 01 2023
accepted: 18 08 2023
medline: 7 9 2023
pubmed: 6 9 2023
entrez: 5 9 2023
Statut: epublish

Résumé

Recent blood transcriptomic analysis of rhodesiense sleeping sickness patients has revealed that neutrophil signature genes and activation markers constitute the top indicators of trypanosomiasis-associated inflammation. Here, we show that Trypanosoma brucei infection results in expansion and differentiation of four splenic neutrophil subpopulations, including Mki67

Identifiants

pubmed: 37669943
doi: 10.1038/s41467-023-41089-w
pii: 10.1038/s41467-023-41089-w
pmc: PMC10480172
doi:

Substances chimiques

Metalloproteases EC 3.4.-

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

5418

Informations de copyright

© 2023. Springer Nature Limited.

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Auteurs

Hien Thi Thu Pham (HTT)

Laboratory for Biomedical Research, Department of Environmental Technology, Food Technology and Molecular Biotechnology KR01, Ghent University Global Campus, Incheon, South Korea.
Department of Biomedical Molecular Biology, Ghent University, Ghent, Belgium.
Laboratory of Cellular and Molecular Immunology, Vrije Universiteit Brussel, Brussels, Belgium.

Stefan Magez (S)

Laboratory for Biomedical Research, Department of Environmental Technology, Food Technology and Molecular Biotechnology KR01, Ghent University Global Campus, Incheon, South Korea.
Laboratory of Cellular and Molecular Immunology, Vrije Universiteit Brussel, Brussels, Belgium.
Department of Biochemistry and Microbiology, Ghent University, Ghent, Belgium.

Boyoon Choi (B)

Laboratory for Biomedical Research, Department of Environmental Technology, Food Technology and Molecular Biotechnology KR01, Ghent University Global Campus, Incheon, South Korea.
Laboratory of Cellular and Molecular Immunology, Vrije Universiteit Brussel, Brussels, Belgium.
Department of Biochemistry and Microbiology, Ghent University, Ghent, Belgium.

Bolortsetseg Baatar (B)

Laboratory for Biomedical Research, Department of Environmental Technology, Food Technology and Molecular Biotechnology KR01, Ghent University Global Campus, Incheon, South Korea.

Joohee Jung (J)

Duksung Women's University, Seoul, South Korea.

Magdalena Radwanska (M)

Laboratory for Biomedical Research, Department of Environmental Technology, Food Technology and Molecular Biotechnology KR01, Ghent University Global Campus, Incheon, South Korea. magdalena.radwanska@Ugent.be.
Department of Biomedical Molecular Biology, Ghent University, Ghent, Belgium. magdalena.radwanska@Ugent.be.

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