Evaluation of the HBV liver reservoir with fine needle aspirates.

FNA HBV Liver biopsy cccDNA

Journal

JHEP reports : innovation in hepatology
ISSN: 2589-5559
Titre abrégé: JHEP Rep
Pays: Netherlands
ID NLM: 101761237

Informations de publication

Date de publication:
Oct 2023
Historique:
received: 21 04 2023
revised: 19 06 2023
accepted: 22 06 2023
medline: 7 9 2023
pubmed: 7 9 2023
entrez: 7 9 2023
Statut: epublish

Résumé

Finite duration of treatment associated with HBsAg loss is the current goal for improved therapeutic approaches against chronic HBV infection, as it indicates elimination or durable inactivation of intrahepatic covalently closed circular DNA (cccDNA). To assist drug development, the definition of early predictive markers of HBsAg loss by assessing their value in reflecting intrahepatic cccDNA levels and transcriptional activity is essential. Fine needle aspirates (FNAs) have recently emerged as a less invasive alternative to core liver biopsy (CLB) and showed to be useful for investigating intrahepatic immune responses. The aim of this study was to optimise and validate the use of FNA Paired FNA/CLB samples were obtained from patients with HBeAg+ chronic hepatitis (n = 4), HBeAg- chronic hepatitis (n = 4), and HBeAg- chronic infection (n = 1). One HBeAg+ patient was undergoing tenofovir treatment. HBV 3.5-kb RNA and cccDNA were quantified by droplet digital PCR. cccDNA was quantifiable in all but one FNA/CLB pair, showing the highest levels in untreated HBeAg+ patients, except for the tenofovir-treated patient. Similarly, 3.5-kb RNA was detectable in all but one FNA sample and showed higher levels in HBeAg+ patients. When comparing cccDNA and 3.5-kb RNA quantification in FNA These results demonstrate the possibility to quantify cccDNA and assess its transcriptional activity in patients with chronic hepatitis B by combining FNA and droplet digital PCR. This supports the use of FNA in clinical trials to evaluate the intrahepatic viral reservoir during the development of new antivirals and immunomodulatory agents. Chronic hepatitis B infection is characterised by a complex interplay between immune responses and viral replication in the liver, which determines the long-term outcome of the disease. In this study, we show that fine needle aspiration of the liver, a less-invasive alternative to core biopsies, allows the assessment of the hepatic viral reservoir.

Sections du résumé

Background & Aims UNASSIGNED
Finite duration of treatment associated with HBsAg loss is the current goal for improved therapeutic approaches against chronic HBV infection, as it indicates elimination or durable inactivation of intrahepatic covalently closed circular DNA (cccDNA). To assist drug development, the definition of early predictive markers of HBsAg loss by assessing their value in reflecting intrahepatic cccDNA levels and transcriptional activity is essential. Fine needle aspirates (FNAs) have recently emerged as a less invasive alternative to core liver biopsy (CLB) and showed to be useful for investigating intrahepatic immune responses. The aim of this study was to optimise and validate the use of FNA
Methods UNASSIGNED
Paired FNA/CLB samples were obtained from patients with HBeAg+ chronic hepatitis (n = 4), HBeAg- chronic hepatitis (n = 4), and HBeAg- chronic infection (n = 1). One HBeAg+ patient was undergoing tenofovir treatment. HBV 3.5-kb RNA and cccDNA were quantified by droplet digital PCR.
Results UNASSIGNED
cccDNA was quantifiable in all but one FNA/CLB pair, showing the highest levels in untreated HBeAg+ patients, except for the tenofovir-treated patient. Similarly, 3.5-kb RNA was detectable in all but one FNA sample and showed higher levels in HBeAg+ patients. When comparing cccDNA and 3.5-kb RNA quantification in FNA
Conclusions UNASSIGNED
These results demonstrate the possibility to quantify cccDNA and assess its transcriptional activity in patients with chronic hepatitis B by combining FNA and droplet digital PCR. This supports the use of FNA in clinical trials to evaluate the intrahepatic viral reservoir during the development of new antivirals and immunomodulatory agents.
Impact and implications UNASSIGNED
Chronic hepatitis B infection is characterised by a complex interplay between immune responses and viral replication in the liver, which determines the long-term outcome of the disease. In this study, we show that fine needle aspiration of the liver, a less-invasive alternative to core biopsies, allows the assessment of the hepatic viral reservoir.

Identifiants

pubmed: 37675272
doi: 10.1016/j.jhepr.2023.100841
pii: S2589-5559(23)00172-6
pmc: PMC10477677
doi:

Types de publication

Journal Article

Langues

eng

Pagination

100841

Informations de copyright

© 2023 The Authors.

Déclaration de conflit d'intérêts

FZ received grants from Assembly, Beam Therapeutics, and Evotec; FZ had consulting activities with Assembly, Blue Jay, Gilead, and GSK. Please refer to the accompanying ICMJE disclosure forms for further details.

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Auteurs

Barbara Testoni (B)

INSERM U1052, CNRS UMR-5286, Cancer Research Center of Lyon (CRCL), Lyon, France.
University of Lyon, Université Claude-Bernard (UCBL), Lyon, France.
Hepatology Institute of Lyon, Lyon, France.

Armando Andres Roca Suarez (AA)

INSERM U1052, CNRS UMR-5286, Cancer Research Center of Lyon (CRCL), Lyon, France.
University of Lyon, Université Claude-Bernard (UCBL), Lyon, France.
Hepatology Institute of Lyon, Lyon, France.

Arianna Battisti (A)

Barts Liver Centre, Immunobiology, Blizard Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, London, UK.

Marie-Laure Plissonnier (ML)

INSERM U1052, CNRS UMR-5286, Cancer Research Center of Lyon (CRCL), Lyon, France.
University of Lyon, Université Claude-Bernard (UCBL), Lyon, France.
Hepatology Institute of Lyon, Lyon, France.

Marintha Heil (M)

Roche Molecular Diagnostics, Pleasanton, CA, USA.

Thierry Fontanges (T)

Department of Hepatology, Croix Rousse Hospital, Hospices Civils de Lyon, Lyon, France.

François Villeret (F)

Department of Hepatology, Croix Rousse Hospital, Hospices Civils de Lyon, Lyon, France.

Yasmina Chouik (Y)

Department of Hepatology, Croix Rousse Hospital, Hospices Civils de Lyon, Lyon, France.

Massimo Levrero (M)

INSERM U1052, CNRS UMR-5286, Cancer Research Center of Lyon (CRCL), Lyon, France.
Hepatology Institute of Lyon, Lyon, France.
Department of Hepatology, Croix Rousse Hospital, Hospices Civils de Lyon, Lyon, France.
Department of Internal Medicine - DMISM and the IIT Center for Life Nanoscience (CLNS), Sapienza University, Rome, Italy.

Upkar Gill (U)

Barts Liver Centre, Immunobiology, Blizard Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, London, UK.

Patrick Kennedy (P)

Barts Liver Centre, Immunobiology, Blizard Institute, Barts and The London School of Medicine and Dentistry, Queen Mary University of London, London, UK.

Fabien Zoulim (F)

INSERM U1052, CNRS UMR-5286, Cancer Research Center of Lyon (CRCL), Lyon, France.
University of Lyon, Université Claude-Bernard (UCBL), Lyon, France.
Hepatology Institute of Lyon, Lyon, France.
Department of Hepatology, Croix Rousse Hospital, Hospices Civils de Lyon, Lyon, France.

Classifications MeSH