Pubertal patterns in children with sickle cell anemia: A case-control study in Cameroon.


Journal

Archives de pediatrie : organe officiel de la Societe francaise de pediatrie
ISSN: 1769-664X
Titre abrégé: Arch Pediatr
Pays: France
ID NLM: 9421356

Informations de publication

Date de publication:
Oct 2023
Historique:
received: 25 07 2022
revised: 19 03 2023
accepted: 26 03 2023
medline: 27 9 2023
pubmed: 14 9 2023
entrez: 13 9 2023
Statut: ppublish

Résumé

Puberty may be impaired in children with sickle cell anemia (SCA). Therefore, we aimed to explore the clinical and hormonal features of puberty in Cameroonian children. In a case-control study, we included 64 children aged 8-18 years with SCA matched to healthy controls. We assessed height, weight, body mass index, body composition, and Tanner stages. Hormonal measurements included anti-mullerian hormone, follicle-stimulating hormone, luteinizing hormone, and sex hormones (estrogens/testosterone). We used the Mann-Whitney Wilcoxon test to compare the median values between cases and controls. We looked for associations between the severity criteria of SCA and delayed puberty through multivariate analysis. Delayed puberty was reported in 27.3% of girls and 10% of boys with SCA. The median age of menarche was delayed by 2 years compared to controls. SCA patients had a low lean body mass compared to controls (p = 0.03). Anti-mullerian hormone levels were significantly higher in boys with SCA than those of controls (45.9 ng/mL vs. 17.65 ng/mL; p = 0.018). A history of severe infection, acute chest syndrome, and low hemoglobin level was associated with delayed sexual maturation in children with SCA. Our study revealed delayed puberty in children with SCA. Moreover, puberty is affected by the severity of the disease. This highlights the importance of regular monitoring of puberty during the follow-up of these children.

Sections du résumé

BACKGROUND BACKGROUND
Puberty may be impaired in children with sickle cell anemia (SCA). Therefore, we aimed to explore the clinical and hormonal features of puberty in Cameroonian children.
METHODS METHODS
In a case-control study, we included 64 children aged 8-18 years with SCA matched to healthy controls. We assessed height, weight, body mass index, body composition, and Tanner stages. Hormonal measurements included anti-mullerian hormone, follicle-stimulating hormone, luteinizing hormone, and sex hormones (estrogens/testosterone). We used the Mann-Whitney Wilcoxon test to compare the median values between cases and controls. We looked for associations between the severity criteria of SCA and delayed puberty through multivariate analysis.
RESULTS RESULTS
Delayed puberty was reported in 27.3% of girls and 10% of boys with SCA. The median age of menarche was delayed by 2 years compared to controls. SCA patients had a low lean body mass compared to controls (p = 0.03). Anti-mullerian hormone levels were significantly higher in boys with SCA than those of controls (45.9 ng/mL vs. 17.65 ng/mL; p = 0.018). A history of severe infection, acute chest syndrome, and low hemoglobin level was associated with delayed sexual maturation in children with SCA.
CONCLUSION CONCLUSIONS
Our study revealed delayed puberty in children with SCA. Moreover, puberty is affected by the severity of the disease. This highlights the importance of regular monitoring of puberty during the follow-up of these children.

Identifiants

pubmed: 37704520
pii: S0929-693X(23)00135-5
doi: 10.1016/j.arcped.2023.03.015
pii:
doi:

Substances chimiques

Anti-Mullerian Hormone 80497-65-0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

466-470

Informations de copyright

Copyright © 2023. Published by Elsevier Masson SAS.

Déclaration de conflit d'intérêts

Declaration of Competing Interest None.

Auteurs

Ritha Mbono Betoko (RM)

Department of Pediatrics, Faculty of Medicine and Pharmaceutical Sciences, Cameroon. Electronic address: mbonobetoko@yahoo.fr.

Suzanne Sap (S)

Department of Pediatrics, Faculty of Medicine and Biomedical Sciences, Cameroon; Mother and Child Centre, Chantal BIYA Foundation, Yaounde, Cameroon.

Anastasie Yanda Alima (AY)

Mother and Child Centre, Chantal BIYA Foundation, Yaounde, Cameroon.

David Chelo (D)

Department of Pediatrics, Faculty of Medicine and Biomedical Sciences, Cameroon; Mother and Child Centre, Chantal BIYA Foundation, Yaounde, Cameroon.

Jocelyn Tony Nengom (JT)

Mother and Child Centre, Chantal BIYA Foundation, Yaounde, Cameroon.

Dominique Simon (D)

Department of Pediatric Endocrinology and Diabetes, Robert Debré Hospital, Paris, France.

Didier Chevenne (D)

Department of Pediatric Endocrinology and Diabetes, Robert Debré Hospital, Paris, France.

Paul Koki Ndombo (PK)

Department of Pediatrics, Faculty of Medicine and Biomedical Sciences, Cameroon; Mother and Child Centre, Chantal BIYA Foundation, Yaounde, Cameroon.

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