Chromosomal rearrangement in the 22q11.2 region: a critical locus for sociability and attentional skills.


Journal

Psychiatric genetics
ISSN: 1473-5873
Titre abrégé: Psychiatr Genet
Pays: England
ID NLM: 9106748

Informations de publication

Date de publication:
01 10 2023
Historique:
medline: 15 9 2023
pubmed: 14 9 2023
entrez: 14 9 2023
Statut: ppublish

Résumé

Rearrangements of 22q11.2 region, most often deletions and duplications, are responsible for multiple congenital disorders. These rearrangements are involved in syndromes that share some phenotypic similarities. To date, 22q11.2 triplication remains very rare, with few cases described in the literature. Here, we report for the first time the clinical, neurocognitive, social cognition and psychiatric properties of a 6-year-old child with 22q11.2 triplication, in comparison with a patient with 22q11.2 duplication and 16 cases of patients with 22q11.2 deletion. Chromosomal region 22q11.2 seems to be a critical locus for sociability and attentional skills and rearrangements could be interpreted as a predisposing factor for the development of psychotic symptoms (22q11.2 deletion), a protective factor (22q11.2 duplication) or a tendency factor for hypersociability (22q11.2 triplication).

Identifiants

pubmed: 37706496
doi: 10.1097/YPG.0000000000000351
pii: 00041444-202310000-00004
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

202-205

Informations de copyright

Copyright © 2023 Wolters Kluwer Health, Inc. All rights reserved.

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Auteurs

Marie-Noëlle Babinet (MN)

Centre de Référence Maladies Rares Troubles du Comportement d'Origine Génétique (GénoPsy Lyon), Centre d'excellence autisme iMIND, Centre Hospitalier Le Vinatier, 95 Boulevard Pinel, 69500 Bron, France et UMR 5229, CNRS & Université Lyon 1.
Unité de Recherche Étude des Mécanismes Cognitifs, Université Lumière Lyon 2, Université de Lyon, 5 avenue Pierre Mendes-France, 69676 Bron cedex, France.

Nadine Thomas (N)

Centre de Référence Maladies Rares Troubles du Comportement d'Origine Génétique (GénoPsy Lyon), Centre d'excellence autisme iMIND, Centre Hospitalier Le Vinatier, 95 Boulevard Pinel, 69500 Bron, France et UMR 5229, CNRS & Université Lyon 1.

Linda Pons (L)

Centre de Référence Maladies Rares Troubles du Comportement d'Origine Génétique (GénoPsy Lyon), Centre d'excellence autisme iMIND, Centre Hospitalier Le Vinatier, 95 Boulevard Pinel, 69500 Bron, France et UMR 5229, CNRS & Université Lyon 1.
Unité Fonctionnelle de Cytogénétique, Laboratoire de Biologie Médicale, Centre Hospitalier de Valence, Valence, France.

Caroline Schluth-Bolard (C)

Genetics Department, GH Est, Hospices Civils de Lyon, Lyon, France.
Université Claude Bernard Lyon 1, CNRS, INSERM, Physiopathologie et Génétique du Neurone et du Muscle, UMR5261, U1315, Institut NeuroMyoGène, 69008 Lyon, France.
Laboratoire de Diagnostic Génétique, Institut de Génétique Médicale d'Alsace, Hôpitaux Universitaires de Strasbourg, Strasbourg, France.

Damien Sanlaville (D)

Université Claude Bernard Lyon 1, CNRS, INSERM, Physiopathologie et Génétique du Neurone et du Muscle, UMR5261, U1315, Institut NeuroMyoGène, 69008 Lyon, France.

Caroline Demily (C)

Centre de Référence Maladies Rares Troubles du Comportement d'Origine Génétique (GénoPsy Lyon), Centre d'excellence autisme iMIND, Centre Hospitalier Le Vinatier, 95 Boulevard Pinel, 69500 Bron, France et UMR 5229, CNRS & Université Lyon 1.

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