Costunolide prevents renal ischemia-reperfusion injury in rats by reducing autophagy, apoptosis, inflammation, and DNA damage.

Apoptosis Autophagy Costunolide Rat Renal ischemia-reperfusion

Journal

Iranian journal of basic medical sciences
ISSN: 2008-3866
Titre abrégé: Iran J Basic Med Sci
Pays: Iran
ID NLM: 101517966

Informations de publication

Date de publication:
2023
Historique:
received: 14 04 2023
accepted: 30 05 2023
medline: 22 9 2023
pubmed: 22 9 2023
entrez: 22 9 2023
Statut: ppublish

Résumé

Renal ischemia-reperfusion (I/R) is a vital health condition leading to acute kidney injury. Costunolide (COST) is an actively used molecule clinically for its anti-inflammatory, antioxidant, and immunomodulatory properties. In the present study, we searched for the possible protective effects of COST against renal ischemia/reperfusion (I/R) injury in rats. We established a renal I/R rat model. We divided forty rats into four groups: group I (sham), group II (I/R), group III (I/R+COST 5 mg/kg), and group IV (I/R+COST 10 mg/kg). We collected blood, kidney, and lung samples for analysis. COST administration performed anti-oxidant and anti-inflammatory activity by reducing oxidant parameters and proinflammatory cytokine levels. COST alleviated DNA damage through declining 8-hydroxydeoxyguanosine (8-OHdG) levels. In addition, COST diminished tubular damage and inflammation by reducing kidney injury molecule-1 (KIM-1) production. COST administration also ameliorated apoptosis and autophagy by decreasing caspase-3 and microtubule-associated protein light chain 3B (MAPLC3, LC3B) expression. COST demonstrated protective effects against renal I/R-induced injury.

Identifiants

pubmed: 37736519
doi: 10.22038/IJBMS.2023.71779.15596
pmc: PMC10510491
doi:

Types de publication

Journal Article

Langues

eng

Pagination

1168-1176

Déclaration de conflit d'intérêts

The authors reported no potential conflicts of interest.

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Auteurs

Mustafa Can Güler (MC)

Department of Physiology, Faculty of Medicine, Atatürk University, Erzurum, Turkey.

Erol Akpinar (E)

Department of Pharmacology, Faculty of Medicine, Atatürk University, Erzurum, Turkey.

Ayhan Tanyeli (A)

Department of Physiology, Faculty of Medicine, Atatürk University, Erzurum, Turkey.

Selim Çomakli (S)

Department of Pathology, Faculty of Veterinary, Atatürk University, Erzurum, Turkey.

Yasin Bayir (Y)

Department of Biochemistry, Faculty of Pharmacy, Atatürk University, Erzurum, Turkey.

Classifications MeSH