Implications of Ventricular Arrhythmia After Cardiac Resynchronization Therapy.

Anti-tachycardia pacing heart failure paced conduction delay shock therapy ventricular arrhythmia

Journal

Heart rhythm
ISSN: 1556-3871
Titre abrégé: Heart Rhythm
Pays: United States
ID NLM: 101200317

Informations de publication

Date de publication:
20 Sep 2023
Historique:
received: 28 06 2023
revised: 02 09 2023
accepted: 16 09 2023
medline: 23 9 2023
pubmed: 23 9 2023
entrez: 22 9 2023
Statut: aheadofprint

Résumé

Conflicting data are available on whether ventricular arrhythmia (VA) or shock therapy increases mortality. Though cardiac resynchronization therapy (CRT) reduces the risk of VA, little is known about the prognostic value of VA among patients with CRT devices. This study aimed to evaluate the implications of VA as a prognostic marker for CRT. We investigated 330 CRT patients within one year after CRT device implantation. The primary endpoint was the composite endpoint of all-cause death or hospitalization for HF. Forty-three patients had VA events. These patients had a significantly higher risk of the primary endpoint, even among CRT responders (p = 0.009). Fast VA compared to slow VA was associated with an increased risk of the primary endpoint (hazard ratio [HR]: 2.14; 95% confidence interval [CI]: 1.06-4.34, p = 0.035). Shock therapy was not associated with a primary endpoint (shock therapy vs. anti-tachycardia pacing, HR: 1.49; 95% CI: 0.73-3.03, p = 0.269). The patients with VA had a lower prevalence of response to CRT (23 [53%] vs. 202 [70%], p = 0.031) and longer LV-paced conduction time (174 ± 23 ms vs. 143 ± 36 ms, p = 0.003) than the patients without VA. VA occurrence within one year was related to paced electrical delay and poor response to CRT. VA could be associated with poor prognosis among CRT patients.

Sections du résumé

BACKGROUND BACKGROUND
Conflicting data are available on whether ventricular arrhythmia (VA) or shock therapy increases mortality. Though cardiac resynchronization therapy (CRT) reduces the risk of VA, little is known about the prognostic value of VA among patients with CRT devices.
OBJECTIVES OBJECTIVE
This study aimed to evaluate the implications of VA as a prognostic marker for CRT.
METHODS METHODS
We investigated 330 CRT patients within one year after CRT device implantation. The primary endpoint was the composite endpoint of all-cause death or hospitalization for HF.
RESULTS RESULTS
Forty-three patients had VA events. These patients had a significantly higher risk of the primary endpoint, even among CRT responders (p = 0.009). Fast VA compared to slow VA was associated with an increased risk of the primary endpoint (hazard ratio [HR]: 2.14; 95% confidence interval [CI]: 1.06-4.34, p = 0.035). Shock therapy was not associated with a primary endpoint (shock therapy vs. anti-tachycardia pacing, HR: 1.49; 95% CI: 0.73-3.03, p = 0.269). The patients with VA had a lower prevalence of response to CRT (23 [53%] vs. 202 [70%], p = 0.031) and longer LV-paced conduction time (174 ± 23 ms vs. 143 ± 36 ms, p = 0.003) than the patients without VA.
CONCLUSION CONCLUSIONS
VA occurrence within one year was related to paced electrical delay and poor response to CRT. VA could be associated with poor prognosis among CRT patients.

Identifiants

pubmed: 37739199
pii: S1547-5271(23)02705-4
doi: 10.1016/j.hrthm.2023.09.014
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2023. Published by Elsevier Inc.

Auteurs

Nobuhiko Ueda (N)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan. Electronic address: rabbitueda2003@ncvc.go.jp.

Kohei Ishibashi (K)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Takashi Noda (T)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Satoshi Oka (S)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Yuichiro Miyazaki (Y)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Keiko Shimamoto (K)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Akinori Wakamiya (A)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Kenzaburo Nakajima (K)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Tsukasa Kamakura (T)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Mitsuru Wada (M)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Yuko Inoue (Y)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Koji Miyamoto (K)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Satoshi Nagase (S)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Takeshi Aiba (T)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Hideaki Kanzaki (H)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Chisato Izumi (C)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Teruo Noguchi (T)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Kengo Kusano (K)

Department of Cardiovascular Medicine, National Cerebral and Cardiovascular Center, Suita, Japan.

Classifications MeSH