Subtyping of Group 3/4 medulloblastoma as a potential prognostic biomarker among patients treated with reduced dose of craniospinal irradiation: a Japanese Pediatric Molecular Neuro-Oncology Group study.


Journal

Acta neuropathologica communications
ISSN: 2051-5960
Titre abrégé: Acta Neuropathol Commun
Pays: England
ID NLM: 101610673

Informations de publication

Date de publication:
25 09 2023
Historique:
received: 30 07 2023
accepted: 08 09 2023
medline: 27 9 2023
pubmed: 26 9 2023
entrez: 25 9 2023
Statut: epublish

Résumé

One of the most significant challenges in patients with medulloblastoma is reducing the dose of craniospinal irradiation (CSI) to minimize neurological sequelae in survivors. Molecular characterization of patients receiving lower than standard dose of CSI therapy is important to facilitate further reduction of treatment burden. We conducted DNA methylation analysis using an Illumina Methylation EPIC array to investigate molecular prognostic markers in 38 patients with medulloblastoma who were registered in the Japan Pediatric Molecular Neuro-Oncology Group and treated with reduced-dose CSI. Among the patients, 23 were classified as having a standard-risk and 15 as high-risk according to the classic classification based on tumor resection rate and presence of metastasis, respectively. The median follow-up period was 71.5 months (12.0-231.0). The median CSI dose was 18 Gy (15.0-24.0) in both groups, and 5 patients in the high-risk group received a CSI dose of 18.0 Gy. Molecular subgrouping revealed that the standard-risk cohort included 5 WNT, 2 SHH, and 16 Group 3/4 cases; all 15 patients in the high-risk cohort had Group 3/4 medulloblastoma. Among the patients with Group 3/4 medulloblastoma, 9 of the 31 Group 3/4 cases were subclassified as subclass II, III, and V, which were known to an association with poor prognosis according to the novel subtyping among the subgroups. Patients with poor prognostic subtype showed worse prognosis than that of others (5-year progression survival rate 90.4% vs. 22.2%; p < 0.0001). The result was replicated in the multivariate analysis (hazard ratio12.77, 95% confidence interval for hazard ratio 2.38-99.21, p value 0.0026 for progression-free survival, hazard ratio 5.02, 95% confidence interval for hazard ratio 1.03-29.11, p value 0.044 for overall survival). Although these findings require validation in a larger cohort, the present findings suggest that novel subtyping of Group 3/4 medulloblastoma may be a promising prognostic biomarker even among patients treated with lower-dose CSI than standard treatment.

Sections du résumé

BACKGROUND
One of the most significant challenges in patients with medulloblastoma is reducing the dose of craniospinal irradiation (CSI) to minimize neurological sequelae in survivors. Molecular characterization of patients receiving lower than standard dose of CSI therapy is important to facilitate further reduction of treatment burden.
METHODS
We conducted DNA methylation analysis using an Illumina Methylation EPIC array to investigate molecular prognostic markers in 38 patients with medulloblastoma who were registered in the Japan Pediatric Molecular Neuro-Oncology Group and treated with reduced-dose CSI.
RESULTS
Among the patients, 23 were classified as having a standard-risk and 15 as high-risk according to the classic classification based on tumor resection rate and presence of metastasis, respectively. The median follow-up period was 71.5 months (12.0-231.0). The median CSI dose was 18 Gy (15.0-24.0) in both groups, and 5 patients in the high-risk group received a CSI dose of 18.0 Gy. Molecular subgrouping revealed that the standard-risk cohort included 5 WNT, 2 SHH, and 16 Group 3/4 cases; all 15 patients in the high-risk cohort had Group 3/4 medulloblastoma. Among the patients with Group 3/4 medulloblastoma, 9 of the 31 Group 3/4 cases were subclassified as subclass II, III, and V, which were known to an association with poor prognosis according to the novel subtyping among the subgroups. Patients with poor prognostic subtype showed worse prognosis than that of others (5-year progression survival rate 90.4% vs. 22.2%; p < 0.0001). The result was replicated in the multivariate analysis (hazard ratio12.77, 95% confidence interval for hazard ratio 2.38-99.21, p value 0.0026 for progression-free survival, hazard ratio 5.02, 95% confidence interval for hazard ratio 1.03-29.11, p value 0.044 for overall survival).
CONCLUSION
Although these findings require validation in a larger cohort, the present findings suggest that novel subtyping of Group 3/4 medulloblastoma may be a promising prognostic biomarker even among patients treated with lower-dose CSI than standard treatment.

Identifiants

pubmed: 37749662
doi: 10.1186/s40478-023-01652-4
pii: 10.1186/s40478-023-01652-4
pmc: PMC10521425
doi:

Substances chimiques

Biomarkers, Tumor 0

Types de publication

Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

153

Informations de copyright

© 2023. BioMed Central Ltd., part of Springer Nature.

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Auteurs

Kohei Fukuoka (K)

Department of Hematology/Oncology, Saitama Children's Medical Center, 1-2, Shin-Toshin, Saitama, 330-8777, Japan. kohfukuoka@gmail.com.

Jun Kurihara (J)

Department of Neurosurgery, Saitama Children's Medical Center, Saitama, Japan.

Tomoko Shofuda (T)

Department of Biomedical Research and Innovation, Institute for Clinical Research, Osaka National Hospital, National Hospital Organization, Osaka, Japan.

Naoki Kagawa (N)

Department of Neurosurgery, Osaka University Graduate School of Medicine, Osaka, Japan.

Kai Yamasaki (K)

Department of Pediatric Hematology and Oncology, Osaka City General Hospital, Osaka, Japan.

Ryo Ando (R)

Department of Neurosurgery, Chiba Children's Hospital, Chiba, Japan.

Joji Ishida (J)

Department of Neurological Surgery, Okayama University Graduate School, Okayama, Japan.

Masayuki Kanamori (M)

Department of Neurosurgery, Tohoku University Graduate School of Medicine, Sendai, Japan.

Atsufumi Kawamura (A)

Department of Neurosurgery, Hyogo Prefectural Kobe Children's Hospital, Kobe, Japan.

Young-Soo Park (YS)

Department of Neurosurgery, Nara Medical University, Kashihara, Japan.

Chikako Kiyotani (C)

Children's Cancer Center, National Center for Child Health and Development, Tokyo, Japan.

Takuya Akai (T)

Departments of Neurosurgery, Graduate School of Medicine and Pharmaceutical Science, University of Toyama, Toyama, Japan.

Dai Keino (D)

Division of Hematology/Oncology, Kanagawa Children's Medical Center, Yokohama, Japan.

Yosuke Miyairi (Y)

Department of Neurosurgery, Nagano Children's Hospital, Azumino, Japan.

Atsushi Sasaki (A)

Department of Pathology, Saitama Medical University, Saitama, Japan.

Junko Hirato (J)

Department of Pathology, Public Tomioka General Hospital, Gunma, Japan.

Takeshi Inoue (T)

Department of Pathology, Osaka City General Hospital, Osaka, Japan.

Atsuko Nakazawa (A)

Department of Clinical Research, Saitama Children's Medical Center, Saitama, Japan.

Katsuyoshi Koh (K)

Department of Hematology/Oncology, Saitama Children's Medical Center, 1-2, Shin-Toshin, Saitama, 330-8777, Japan.

Ryo Nishikawa (R)

Department of Neuro-Oncology/Neurosurgery, Saitama Medical University International Medical Center, Saitama, Japan.

Isao Date (I)

Department of Neurological Surgery, Okayama University Graduate School, Okayama, Japan.

Motoo Nagane (M)

Department of Neurosurgery, Kyorin University Faculty of Medicine, Mitaka, Japan.

Koichi Ichimura (K)

Department of Brain Disease Translational Research, Juntendo University Faculty of Medicine, Tokyo, Japan.

Yonehiro Kanemura (Y)

Department of Biomedical Research and Innovation, Institute for Clinical Research, Osaka National Hospital, National Hospital Organization, Osaka, Japan.

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Classifications MeSH