Longitudinal Relationship Between Reduced Modic Change Edema and Disability and Pain in Patients With Chronic Low Back Pain.


Journal

Spine
ISSN: 1528-1159
Titre abrégé: Spine (Phila Pa 1976)
Pays: United States
ID NLM: 7610646

Informations de publication

Date de publication:
15 Dec 2023
Historique:
received: 15 03 2023
accepted: 18 09 2023
medline: 28 11 2023
pubmed: 28 9 2023
entrez: 28 9 2023
Statut: ppublish

Résumé

Secondary analyses of a randomized trial [Antibiotics In Modic changes (MCs) study]. To assess whether or not reduced MC edema over time is related to reduced disability and pain in patients with chronic low back pain (LBP). It is not clear whether or not reduced MC edema implies improved clinical outcomes. Linear regression was conducted separately in 2 subgroups with MC edema at baseline on short tau inversion recovery (STIR) or T1/T2-weighted magnetic resonance imaging, respectively. Independent variable: reduced edema (yes/no) at 1 year on STIR or T1/T2-series, respectively. Dependent variable: 1-year score on the Roland-Morris Disability Questionnaire (RMDQ), Oswestry Disability Index (ODI), or 0 to 10 numeric rating scale for LBP intensity, adjusted for the baseline score, age, smoking, body mass index, physical workload, and baseline edema on STIR (STIR analysis only). Post hoc, we, in addition, adjusted all analyses for baseline edema on STIR, treatment group (amoxicillin/placebo), and prior disc surgery-or for disc degeneration. Among patients with MC edema on STIR at baseline (n = 162), reduced edema on STIR was not significantly related to the RMDQ ( B : -1.0, 95% CI: -2.8, 0.8; P = 0.27), ODI ( B :-1.4, 95% CI: -5.4, 2.6; P = 0.50), or LBP intensity scores ( B : -0.05, 95% CI: -0.8, 0.7; P = 0.90) after 1 year. Among patients with MC edema on T1/T2-series at baseline (n = 116), reduced edema on T1/T2 ( i.e ., reduced volume of the type 1 part of MCs) was not significantly related to RMDQ ( B: -1.7, 95% CI: -3.8, 0.3; P = 0.10) or ODI score ( B : -2.3, 95% CI: -7.1, 2.5; P = 0.34) but was significantly related to LBP intensity at 1 year ( B : -0.9, 95% CI: -1.8, -0.04; P = 0.04; correlation coefficient: 0.24). The post hoc analyses supported these results. Reduced MC edema over 1 year was not significantly associated with pain-related disability but was (on T1/T2-series) significantly but weakly related to reduced LBP intensity. Level 3.

Sections du résumé

STUDY DESIGN METHODS
Secondary analyses of a randomized trial [Antibiotics In Modic changes (MCs) study].
OBJECTIVE OBJECTIVE
To assess whether or not reduced MC edema over time is related to reduced disability and pain in patients with chronic low back pain (LBP).
SUMMARY OF BACKGROUND DATA BACKGROUND
It is not clear whether or not reduced MC edema implies improved clinical outcomes.
PATIENTS AND METHODS METHODS
Linear regression was conducted separately in 2 subgroups with MC edema at baseline on short tau inversion recovery (STIR) or T1/T2-weighted magnetic resonance imaging, respectively. Independent variable: reduced edema (yes/no) at 1 year on STIR or T1/T2-series, respectively. Dependent variable: 1-year score on the Roland-Morris Disability Questionnaire (RMDQ), Oswestry Disability Index (ODI), or 0 to 10 numeric rating scale for LBP intensity, adjusted for the baseline score, age, smoking, body mass index, physical workload, and baseline edema on STIR (STIR analysis only). Post hoc, we, in addition, adjusted all analyses for baseline edema on STIR, treatment group (amoxicillin/placebo), and prior disc surgery-or for disc degeneration.
RESULTS RESULTS
Among patients with MC edema on STIR at baseline (n = 162), reduced edema on STIR was not significantly related to the RMDQ ( B : -1.0, 95% CI: -2.8, 0.8; P = 0.27), ODI ( B :-1.4, 95% CI: -5.4, 2.6; P = 0.50), or LBP intensity scores ( B : -0.05, 95% CI: -0.8, 0.7; P = 0.90) after 1 year. Among patients with MC edema on T1/T2-series at baseline (n = 116), reduced edema on T1/T2 ( i.e ., reduced volume of the type 1 part of MCs) was not significantly related to RMDQ ( B: -1.7, 95% CI: -3.8, 0.3; P = 0.10) or ODI score ( B : -2.3, 95% CI: -7.1, 2.5; P = 0.34) but was significantly related to LBP intensity at 1 year ( B : -0.9, 95% CI: -1.8, -0.04; P = 0.04; correlation coefficient: 0.24). The post hoc analyses supported these results.
CONCLUSION CONCLUSIONS
Reduced MC edema over 1 year was not significantly associated with pain-related disability but was (on T1/T2-series) significantly but weakly related to reduced LBP intensity.
LEVEL OF EVIDENCE METHODS
Level 3.

Identifiants

pubmed: 37759351
doi: 10.1097/BRS.0000000000004837
pii: 00007632-990000000-00470
pmc: PMC10662590
doi:

Substances chimiques

Amoxicillin 804826J2HU
Anti-Bacterial Agents 0

Types de publication

Journal Article Randomized Controlled Trial

Langues

eng

Sous-ensembles de citation

IM

Pagination

1699-1708

Informations de copyright

Copyright © 2023 The Author(s). Published by Wolters Kluwer Health, Inc.

Déclaration de conflit d'intérêts

The authors report no conflicts of interest.

Références

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Auteurs

Ansgar Espeland (A)

Department of Radiology, Haukeland University Hospital, Bergen, Norway.
Department of Clinical Medicine, University of Bergen, Bergen, Norway.

Per Martin Kristoffersen (PM)

Department of Radiology, Haukeland University Hospital, Bergen, Norway.
Department of Clinical Medicine, University of Bergen, Bergen, Norway.

Lars Christian Haugli Bråten (LCH)

Department of Research and Innovation, Division of Clinical Neuroscience, Oslo University Hospital, Nydalen, Oslo, Norway.

Lars Grøvle (L)

Department of Rheumatology, Østfold Hospital Trust, Grålum, Norway.

Margreth Grotle (M)

Department of Research and Innovation, Division of Clinical Neuroscience, Oslo University Hospital, Nydalen, Oslo, Norway.
Department of Rehabilitation Science and Health Technology, Centre for Intelligent Musculoskeletal Health, Oslo Metropolitan University, Oslo, Norway.

Anne Julsrud Haugen (AJ)

Department of Rheumatology, Østfold Hospital Trust, Grålum, Norway.

Mads Peder Rolfsen (MP)

Faculty of Medicine, University of Oslo, Blindern, Oslo, Norway.
Department of Orthopedic Surgery, Oslo University Hospital, Nydalen, Oslo, Norway.

Christian Hellum (C)

Department of Orthopedic Surgery, Oslo University Hospital, Nydalen, Oslo, Norway.

John-Anker Zwart (JA)

Department of Research and Innovation, Division of Clinical Neuroscience, Oslo University Hospital, Nydalen, Oslo, Norway.
Faculty of Medicine, University of Oslo, Blindern, Oslo, Norway.

Kjersti Storheim (K)

Department of Research and Innovation, Division of Clinical Neuroscience, Oslo University Hospital, Nydalen, Oslo, Norway.
Department of Rehabilitation Science and Health Technology, Centre for Intelligent Musculoskeletal Health, Oslo Metropolitan University, Oslo, Norway.

Jörg Assmus (J)

Centre for Clinical Research, Haukeland University Hospital, Bergen, Norway.

Nils Vetti (N)

Department of Radiology, Haukeland University Hospital, Bergen, Norway.
Department of Clinical Medicine, University of Bergen, Bergen, Norway.

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