DDAH1 Protects against Cardiotoxin-Induced Muscle Injury and Regeneration.
DDAH1
cardiotoxin
muscle regeneration
oxidative stress
Journal
Antioxidants (Basel, Switzerland)
ISSN: 2076-3921
Titre abrégé: Antioxidants (Basel)
Pays: Switzerland
ID NLM: 101668981
Informations de publication
Date de publication:
13 Sep 2023
13 Sep 2023
Historique:
received:
03
08
2023
revised:
07
09
2023
accepted:
11
09
2023
medline:
28
9
2023
pubmed:
28
9
2023
entrez:
28
9
2023
Statut:
epublish
Résumé
Nitric oxide (NO) is an important biological signaling molecule affecting muscle regeneration. The activity of NO synthase (NOS) is regulated by dimethylarginine dimethylaminohydrolase 1 (DDAH1) through degradation of the endogenous NOS inhibitor asymmetric dimethylarginine (ADMA). To investigate the role of DDAH1 in muscle injury and regeneration, muscle-specific
Identifiants
pubmed: 37760057
pii: antiox12091754
doi: 10.3390/antiox12091754
pmc: PMC10525962
pii:
doi:
Types de publication
Journal Article
Langues
eng
Subventions
Organisme : National Key R&D Program of China
ID : 2020YFF0304503
Organisme : National Natural Science Foundation of China
ID : 82070250
Organisme : Beijing Natural Science Foundation
ID : 5222029
Organisme : Fundamental Research Funds for the Central Universities
ID : E2ET0605X2
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