n-6 fatty acid biomarkers and incident atrial fibrillation: an individual participant-level pooled analysis of 11 international prospective studies.

Fatty Acids and Outcomes Research Consortium atrial arrhythmia cardiovascular disease polyunsaturated fatty acids primary prevention prospective cohorts

Journal

The American journal of clinical nutrition
ISSN: 1938-3207
Titre abrégé: Am J Clin Nutr
Pays: United States
ID NLM: 0376027

Informations de publication

Date de publication:
11 2023
Historique:
received: 12 02 2023
revised: 02 09 2023
accepted: 18 09 2023
medline: 6 11 2023
pubmed: 29 9 2023
entrez: 28 9 2023
Statut: ppublish

Résumé

The presence of atrial fibrillation (AF) is associated with an over 2-fold increased risk of stroke, heart failure, and cardiovascular mortality. Long chain n-6 PUFAs have been suggested to have a variety of beneficial biologic effects that may reduce AF development; however, prior studies evaluating this relationship are limited. We prospectively evaluated the association between circulating levels of linoleic acid (LA) and arachidonic acid (AA) with incident AF. We used participant-level data from a global consortium of 11 prospective cohort studies with measurements of LA and AA in adults (aged ≥18 y). Participating studies conducted de novo analyses using a prespecified analytical plan with harmonized definitions for exposures, outcomes, covariates, and subgroups. Associations were pooled using inverse-variance weighted meta-analysis. Among 41,335 participants, 6173 incident cases of AF were ascertained, with median follow-up time of 14 y. In multivariable analysis, per interquintile range (difference between the 10th and 90th percentiles for each fatty acid), circulating n-6 levels were not associated with incident AF. For LA, the hazard ratio per interquintile range was 0.96 (95% confidence interval [CI]: 0.89, 1.04), and for AA, 1.02 (95% CI: 0.94, 1.10), with little evidence of heterogeneity between cohorts. Associations were similarly nonsignificant across subgroups of age, race, and biomarker fraction. Biomarkers of n-6 fatty acids including LA and AA are not associated with incident AF. These findings suggest that overall effects of n-6 PUFAs on influencing AF development are neutral.

Sections du résumé

BACKGROUND
The presence of atrial fibrillation (AF) is associated with an over 2-fold increased risk of stroke, heart failure, and cardiovascular mortality. Long chain n-6 PUFAs have been suggested to have a variety of beneficial biologic effects that may reduce AF development; however, prior studies evaluating this relationship are limited.
OBJECTIVES
We prospectively evaluated the association between circulating levels of linoleic acid (LA) and arachidonic acid (AA) with incident AF.
METHODS
We used participant-level data from a global consortium of 11 prospective cohort studies with measurements of LA and AA in adults (aged ≥18 y). Participating studies conducted de novo analyses using a prespecified analytical plan with harmonized definitions for exposures, outcomes, covariates, and subgroups. Associations were pooled using inverse-variance weighted meta-analysis.
RESULTS
Among 41,335 participants, 6173 incident cases of AF were ascertained, with median follow-up time of 14 y. In multivariable analysis, per interquintile range (difference between the 10th and 90th percentiles for each fatty acid), circulating n-6 levels were not associated with incident AF. For LA, the hazard ratio per interquintile range was 0.96 (95% confidence interval [CI]: 0.89, 1.04), and for AA, 1.02 (95% CI: 0.94, 1.10), with little evidence of heterogeneity between cohorts. Associations were similarly nonsignificant across subgroups of age, race, and biomarker fraction.
CONCLUSIONS
Biomarkers of n-6 fatty acids including LA and AA are not associated with incident AF. These findings suggest that overall effects of n-6 PUFAs on influencing AF development are neutral.

Identifiants

pubmed: 37769813
pii: S0002-9165(23)66154-4
doi: 10.1016/j.ajcnut.2023.09.008
pii:
doi:

Substances chimiques

Fatty Acids, Omega-6 0
Fatty Acids, Unsaturated 0
Linoleic Acid 9KJL21T0QJ
Arachidonic Acid 27YG812J1I
Biomarkers 0

Types de publication

Meta-Analysis Journal Article Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

921-929

Informations de copyright

Copyright © 2023 American Society for Nutrition. Published by Elsevier Inc. All rights reserved.

Déclaration de conflit d'intérêts

Conflict of interest The authors report no conflicts of interest.

Auteurs

Parveen K Garg (PK)

Division of Cardiology, University of Southern California Keck School of Medicine, Los Angeles, CA, United States.

Weihua Guan (W)

Division of Biostatistics, University of Minnesota, Minneapolis, MN, United States.

Sarah Nomura (S)

Department of Lab Medicine and Pathology, University of Minnesota, Minneapolis, MN, United States.

Natalie L Weir (NL)

Department of Lab Medicine and Pathology, University of Minnesota, Minneapolis, MN, United States.

Nathan Tintle (N)

Department of Population Health Sciences, College of Nursing, University of Illinois-Chicago, Chicago, IL, United States.

Jyrki K Virtanen (JK)

Institute of Public Health and Clinical Nutrition, University of Eastern Finland, Kuopio, Finland.

Yoichiro Hirakawa (Y)

Department of Medicine and Clinical Science, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.

Frank Qian (F)

Department of Nutrition, Harvard T.H. Chan School of Public Health, Boston, MA, United States; Department of Medicine, Beth Israel Deaconess Medical Center, Harvard Medical School, Boston, MA, United States.

Qi Sun (Q)

Department of Nutrition, Harvard T.H. Chan School of Public Health, Boston, MA, United States.

Eric Rimm (E)

Department of Nutrition, Harvard T.H. Chan School of Public Health, Boston, MA, United States.

Rozenn N Lemaitre (RN)

Cardiovascular Health Research Unit, Department of Medicine, University of Washington, Seattle, WA, United States.

Paul N Jensen (PN)

Cardiovascular Health Research Unit, Department of Medicine, University of Washington, Seattle, WA, United States.

Susan R Heckbert (SR)

Department of Epidemiology, University of Washington, Seattle, WA, United States.

Fumiaki Imamura (F)

MRC Epidemiology Unit, University of Cambridge School of Clinical Medicine, Cambridge, United Kingdom.

Marinka Steur (M)

Department of Epidemiology, Erasmus MC, University Medical Center, Rotterdam, the Netherlands.

Karin Leander (K)

Unit of Cardiovascular and Nutritional Epidemiology, Institute of Environmental Medicine, Karolinska Institutet, Stockholm, Sweden.

Federica Laguzzi (F)

Unit of Cardiovascular and Nutritional Epidemiology, Institute of Environmental Medicine, Karolinska Institutet, Stockholm, Sweden.

Trudy Voortman (T)

Department of Epidemiology, Erasmus MC, University Medical Center, Rotterdam, the Netherlands.

Toshiharu Ninomiya (T)

Department of Epidemiology and Public Health, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan; Center for Cohort Studies, Graduate School of Medical Sciences, Kyushu University, Fukuoka, Japan.

Dariush Mozaffarian (D)

Friedman School of Nutrition Science and Policy, Tufts University, Boston, MA, United States; Division of Cardiology, Tufts Medical Center, Boston, MA, United States.

William S Harris (WS)

Department of Internal Medicine, Sanford School of Medicine, University of South Dakota, Sioux Falls, SD, United States.

David S Siscovick (DS)

The New York Academy of Medicine, New York, NY, United States.

Michael Y Tsai (MY)

Department of Lab Medicine and Pathology, University of Minnesota, Minneapolis, MN, United States. Electronic address: tsaix001@umn.edu.

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Classifications MeSH