Impacts of high sensitivity troponin T reporting on care and outcomes in clinical practice: Interactions between low troponin concentrations and participant sex within two randomized clinical trials.


Journal

International journal of cardiology
ISSN: 1874-1754
Titre abrégé: Int J Cardiol
Pays: Netherlands
ID NLM: 8200291

Informations de publication

Date de publication:
15 12 2023
Historique:
received: 07 12 2022
revised: 10 09 2023
accepted: 22 09 2023
medline: 23 10 2023
pubmed: 29 9 2023
entrez: 28 9 2023
Statut: ppublish

Résumé

The impacts of high sensitivity cardiac troponin (hs-cTn) reporting on downstream interventions amongst suspected acute coronary syndrome (ACS) in the emergency department (ED), especially amongst those with newly identified hs-cTn elevations and in consideration of well-established sex-related disparities, has not been critically evaluated to date. This investigation explores the impact of hs-cTnT reporting on care and outcomes, particularly by participant sex. Two similarly ED-based randomized controlled trials conducted between July 2011 to March 2013 (n = 1988) and August 2015 to April 2019 (n = 3378) were comparatively evaluated. Clinical outcomes were adjudicated to the Fourth Universal Definition of MI. Changes in practice were assessed at 30 days, and death or MI were explored to 12 months. The HS-Troponin study demonstrated no difference in death or MI with unmasking amongst those with hs-cTnT <30 ng/L, whereas the RAPID TnT study demonstrated a significantly higher rate. In RAPID TnT, there was significant increase in death or MI associated with unmasking for females with hs-cTnT <30 ng/L (masked: 11[1.5%], unmasked: 25[3.4%],HR: 2.27,95%C.I.:1.87-2.77,P < 0.001). Less cardiac stress testing with unmasking amongst those <30 ng/L was observed in males in both studies, which was significant in RAPID TnT (masked: 92[12.0%], unmasked: 55[7.0%], P = 0.008). In RAPID TnT, significantly higher rates of angiography in males were observed with unmasking, with no such changes amongst females <30 ng/L (masked: 28[3.7%], unmasked: 51[6.5%],P = 0.01). Compared with males, there were no evident impacts on downstream practices for females with unmasking in RAPID TnT, likely representing missed opportunities to reduce late death or MI.

Sections du résumé

BACKGROUND
The impacts of high sensitivity cardiac troponin (hs-cTn) reporting on downstream interventions amongst suspected acute coronary syndrome (ACS) in the emergency department (ED), especially amongst those with newly identified hs-cTn elevations and in consideration of well-established sex-related disparities, has not been critically evaluated to date. This investigation explores the impact of hs-cTnT reporting on care and outcomes, particularly by participant sex.
METHODS
Two similarly ED-based randomized controlled trials conducted between July 2011 to March 2013 (n = 1988) and August 2015 to April 2019 (n = 3378) were comparatively evaluated. Clinical outcomes were adjudicated to the Fourth Universal Definition of MI. Changes in practice were assessed at 30 days, and death or MI were explored to 12 months.
RESULTS
The HS-Troponin study demonstrated no difference in death or MI with unmasking amongst those with hs-cTnT <30 ng/L, whereas the RAPID TnT study demonstrated a significantly higher rate. In RAPID TnT, there was significant increase in death or MI associated with unmasking for females with hs-cTnT <30 ng/L (masked: 11[1.5%], unmasked: 25[3.4%],HR: 2.27,95%C.I.:1.87-2.77,P < 0.001). Less cardiac stress testing with unmasking amongst those <30 ng/L was observed in males in both studies, which was significant in RAPID TnT (masked: 92[12.0%], unmasked: 55[7.0%], P = 0.008). In RAPID TnT, significantly higher rates of angiography in males were observed with unmasking, with no such changes amongst females <30 ng/L (masked: 28[3.7%], unmasked: 51[6.5%],P = 0.01).
CONCLUSION
Compared with males, there were no evident impacts on downstream practices for females with unmasking in RAPID TnT, likely representing missed opportunities to reduce late death or MI.

Identifiants

pubmed: 37769972
pii: S0167-5273(23)01389-X
doi: 10.1016/j.ijcard.2023.131396
pii:
doi:

Substances chimiques

Troponin T 0
Troponin I 0
Biomarkers 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

131396

Informations de copyright

Copyright © 2023. Published by Elsevier B.V.

Déclaration de conflit d'intérêts

Declaration of Competing Interest KL: None. EK: None. AM: None. CP: Unrestricted Grant: Roche Diagnostics. AC: None. YZ: None. JE: None. SR: None. SJL: None. AB: None. TB: None. JKF: None. SQ: None. LC: Institutional research grants: Siemens, Abbott Diagnostics, Beckman Coulter. Speaker honoraria: Abbott Diagnostics, Beckman Coulter, Siemens Healthineers. DPC: Speaker Honoraria Astra Zeneca Australia, Roche Diagnostic. Unrestricted Grant: Edwards Life Sciences, Roche Diagnostics.

Auteurs

Kristina Lambrakis (K)

College of Medicine & Public Health, Flinders University of South Australia, Adelaide, Australia; South Australian Department of Health, Adelaide, Australia. Electronic address: Kristina.Lambrakis@flinders.edu.au.

Ehsan Khan (E)

College of Medicine & Public Health, Flinders University of South Australia, Adelaide, Australia; South Australian Department of Health, Adelaide, Australia.

Anke van den Merkhof (A)

College of Medicine & Public Health, Flinders University of South Australia, Adelaide, Australia; Faculty of Medical Sciences, University of Groningen, the Netherlands.

Cynthia Papendick (C)

South Australian Department of Health, Adelaide, Australia; School of Medicine, University of Adelaide, Adelaide, Australia.

Anthony Chuang (A)

College of Medicine & Public Health, Flinders University of South Australia, Adelaide, Australia; South Australian Department of Health, Adelaide, Australia.

Yuze Zhai (Y)

College of Medicine & Public Health, Flinders University of South Australia, Adelaide, Australia.

Joanne Eng-Frost (J)

South Australian Department of Health, Adelaide, Australia.

Simon Rocheleau (S)

South Australian Department of Health, Adelaide, Australia.

Sam J Lehman (SJ)

College of Medicine & Public Health, Flinders University of South Australia, Adelaide, Australia; South Australian Department of Health, Adelaide, Australia.

Andrew Blyth (A)

College of Medicine & Public Health, Flinders University of South Australia, Adelaide, Australia; South Australian Department of Health, Adelaide, Australia.

Tom Briffa (T)

School of Population and Global Health, University of Western Australia, Perth, Australia.

Stephen Quinn (S)

Department of Statistics, Data Science and Epidemiology, Swinburne University of Technology, Melbourne, Australia.

John K French (JK)

Department of Cardiology, Liverpool Hospital, University of New South Wales, Sydney, Australia.

Louise Cullen (L)

Emergency and Trauma Centre, Royal Brisbane and Women's Hospital, Brisbane, Australia; School of Public Health, Queensland University of Technology, Brisbane, Australia; School of Medicine, University of Queensland, Brisbane, Australia.

Derek P Chew (DP)

College of Medicine & Public Health, Flinders University of South Australia, Adelaide, Australia; South Australian Department of Health, Adelaide, Australia.

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