Ongoing substrate-driven atrial fibrillation "boxed" in the left atrial posterior wall with ablation: a case report.

ablation box lesion case report independent tachycardia posterior wall

Journal

Frontiers in cardiovascular medicine
ISSN: 2297-055X
Titre abrégé: Front Cardiovasc Med
Pays: Switzerland
ID NLM: 101653388

Informations de publication

Date de publication:
2023
Historique:
received: 02 07 2023
accepted: 31 08 2023
medline: 4 10 2023
pubmed: 4 10 2023
entrez: 4 10 2023
Statut: epublish

Résumé

Patients undergoing valve surgery for rheumatic heart disease are expected to develop significant atrial arrhythmogenic substrates outside of the pulmonary veins, which sometimes require complex ablation techniques for the treatment of symptomatic arrhythmias. We describe, herein, the case of a 76-year-old male undergoing endocardial ablation for the treatment of symptomatic persistent atrial fibrillation which developed after aortic and mitral valve replacement with a simultaneous tricuspid ring annuloplasty. Following pulmonary vein isolation, the patient's atrial fibrillation was converted into cavotricuspid isthmus-dependent atrial flutter. After a successful cavotricuspid isthmus ablation, the arrhythmia reverted back to a left atrial tachyarrhythmia originating from the posterior wall. A linear left atrial lesion led to the electrical isolation of a large area, which included the posterior wall, as well as the containment of the ongoing fibrillatory activity, while sinus rhythm was restored in the rest of the atria. In conclusion, successful left atrial posterior wall isolation can be achieved in the setting of severe scarring due to previous atriotomy by creating a linear lesion on the atrial roof, in conjunction with pulmonary vein isolation, sparing the patient from requiring bottom-line ablation, and avoiding possible esophageal injury. Such compartmentalization of the left atrium may effectively contain local fibrillatory activity, while allowing for the restoration of sinus rhythm.

Identifiants

pubmed: 37790595
doi: 10.3389/fcvm.2023.1251874
pmc: PMC10542893
doi:

Types de publication

Case Reports

Langues

eng

Pagination

1251874

Informations de copyright

© 2023 Traykov, Marchov, Martinov, El Abbady, Gelev and Dichtl.

Déclaration de conflit d'intérêts

The authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.

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Auteurs

Vassil Traykov (V)

Clinic of Cardiology, Acibadem City Clinic Tokuda Hospital, Sofia, Bulgaria.

Daniel Marchov (D)

Clinic of Cardiology, Acibadem City Clinic Tokuda Hospital, Sofia, Bulgaria.

Emiliyan Martinov (E)

Clinic of Cardiology, Acibadem City Clinic Tokuda Hospital, Sofia, Bulgaria.

Asmaa El Abbady (A)

Cardiac Electrophysiology Department, National Heart Institute, Giza, Egypt.

Valeri Gelev (V)

Clinic of Cardiology, Acibadem City Clinic Tokuda Hospital, Sofia, Bulgaria.

Wolfgang Dichtl (W)

University Hospital for Internal Medicine III (Cardiology and Angiology), Medical University of Innsbruck, Innsbruck, Austria.

Classifications MeSH