Carbon dioxide and MAPK signalling: towards therapy for inflammation.
ERK1/2
Hypercapnia
Hypercarbia
Hypocapnia
JNK
p38
Journal
Cell communication and signaling : CCS
ISSN: 1478-811X
Titre abrégé: Cell Commun Signal
Pays: England
ID NLM: 101170464
Informations de publication
Date de publication:
10 10 2023
10 10 2023
Historique:
received:
13
06
2023
accepted:
05
09
2023
medline:
12
10
2023
pubmed:
11
10
2023
entrez:
10
10
2023
Statut:
epublish
Résumé
Inflammation, although necessary to fight infections, becomes a threat when it exceeds the capability of the immune system to control it. In addition, inflammation is a cause and/or symptom of many different disorders, including metabolic, neurodegenerative, autoimmune and cardiovascular diseases. Comorbidities and advanced age are typical predictors of more severe cases of seasonal viral infection, with COVID-19 a clear example. The primary importance of mitogen-activated protein kinases (MAPKs) in the course of COVID-19 is evident in the mechanisms by which cells are infected with SARS-CoV-2; the cytokine storm that profoundly worsens a patient's condition; the pathogenesis of diseases, such as diabetes, obesity, and hypertension, that contribute to a worsened prognosis; and post-COVID-19 complications, such as brain fog and thrombosis. An increasing number of reports have revealed that MAPKs are regulated by carbon dioxide (CO
Identifiants
pubmed: 37817178
doi: 10.1186/s12964-023-01306-x
pii: 10.1186/s12964-023-01306-x
pmc: PMC10566067
doi:
Substances chimiques
Carbon Dioxide
142M471B3J
p38 Mitogen-Activated Protein Kinases
EC 2.7.11.24
Mitogen-Activated Protein Kinases
EC 2.7.11.24
Types de publication
Video-Audio Media
Journal Article
Review
Research Support, Non-U.S. Gov't
Langues
eng
Sous-ensembles de citation
IM
Pagination
280Informations de copyright
© 2023. BioMed Central Ltd., part of Springer Nature.
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