Structural basis of promiscuous substrate transport by Organic Cation Transporter 1.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
11 10 2023
Historique:
received: 06 04 2023
accepted: 29 09 2023
medline: 1 11 2023
pubmed: 12 10 2023
entrez: 11 10 2023
Statut: epublish

Résumé

Organic Cation Transporter 1 (OCT1) plays a crucial role in hepatic metabolism by mediating the uptake of a range of metabolites and drugs. Genetic variations can alter the efficacy and safety of compounds transported by OCT1, such as those used for cardiovascular, oncological, and psychological indications. Despite its importance in drug pharmacokinetics, the substrate selectivity and underlying structural mechanisms of OCT1 remain poorly understood. Here, we present cryo-EM structures of full-length human OCT1 in the inward-open conformation, both ligand-free and drug-bound, indicating the basis for its broad substrate recognition. Comparison of our structures with those of outward-open OCTs provides molecular insight into the alternating access mechanism of OCTs. We observe that hydrophobic gates stabilize the inward-facing conformation, whereas charge neutralization in the binding pocket facilitates the release of cationic substrates. These findings provide a framework for understanding the structural basis of the promiscuity of drug binding and substrate translocation in OCT1.

Identifiants

pubmed: 37821493
doi: 10.1038/s41467-023-42086-9
pii: 10.1038/s41467-023-42086-9
pmc: PMC10567722
doi:

Substances chimiques

Organic Cation Transporter 1 0
Organic Cation Transport Proteins 0
Organic Cation Transporter 2 0

Types de publication

Journal Article Research Support, N.I.H., Extramural Research Support, Non-U.S. Gov't

Langues

eng

Sous-ensembles de citation

IM

Pagination

6374

Subventions

Organisme : NIGMS NIH HHS
ID : U24 GM129547
Pays : United States

Informations de copyright

© 2023. Springer Nature Limited.

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Auteurs

Yi C Zeng (YC)

Molecular, Structural and Computational Biology Division, The Victor Chang Cardiac Research Institute, Darlinghurst, NSW, Australia. y.zeng@victorchang.edu.au.
School of Clinical Medicine, Faculty of Medicine and Health, UNSW Sydney, Sydney, NSW, Australia. y.zeng@victorchang.edu.au.

Meghna Sobti (M)

Molecular, Structural and Computational Biology Division, The Victor Chang Cardiac Research Institute, Darlinghurst, NSW, Australia.
School of Clinical Medicine, Faculty of Medicine and Health, UNSW Sydney, Sydney, NSW, Australia.

Ada Quinn (A)

Australian Institute of Bioengineering and Nanotechnology, University of Queensland, Brisbane, QLD, Australia.

Nicola J Smith (NJ)

School of Biomedical Sciences, Faculty of Medicine & Health, UNSW Sydney, Kensington, NSW, Australia.

Simon H J Brown (SHJ)

School of Chemistry and Molecular Bioscience, Molecular Horizons, and Australian Research Council Centre for Cryo-electron Microscopy of Membrane Proteins, University of Wollongong, Wollongong, NSW, Australia.

Jamie I Vandenberg (JI)

School of Clinical Medicine, Faculty of Medicine and Health, UNSW Sydney, Sydney, NSW, Australia.
Molecular Cardiology and Biophysics Division, The Victor Chang Cardiac Research Institute, Darlinghurst, NSW, Australia.

Renae M Ryan (RM)

School of Medical Sciences, Faculty of Medicine and Health, University of Sydney, Sydney, NSW, Australia.

Megan L O'Mara (ML)

Australian Institute of Bioengineering and Nanotechnology, University of Queensland, Brisbane, QLD, Australia.

Alastair G Stewart (AG)

Molecular, Structural and Computational Biology Division, The Victor Chang Cardiac Research Institute, Darlinghurst, NSW, Australia. a.stewart@victorchang.edu.au.
School of Clinical Medicine, Faculty of Medicine and Health, UNSW Sydney, Sydney, NSW, Australia. a.stewart@victorchang.edu.au.

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