Association of Hip Dysplasia With Trochlear Dysplasia in Skeletally Mature Patients.

hip dysplasia lateral trochlear inclination trochlear depth trochlear dysplasia

Journal

Orthopaedic journal of sports medicine
ISSN: 2325-9671
Titre abrégé: Orthop J Sports Med
Pays: United States
ID NLM: 101620522

Informations de publication

Date de publication:
Oct 2023
Historique:
received: 07 05 2023
accepted: 19 05 2023
medline: 12 10 2023
pubmed: 12 10 2023
entrez: 12 10 2023
Statut: epublish

Résumé

Developmental dysplasia of the hip (DDH) and trochlear dysplasia (TD) are distinct pathologies with several important features in common. In addition to shared risk factors, both forms of dysplasia cause abnormal joint kinematics and force transmission, predisposing patients to pain, injuries to cartilage and soft tissue stabilizers, and ultimately arthritis. To evaluate for an association between hip dysplasia and TD in skeletally mature patients with symptomatic hip dysplasia. Cross-sectional study; Level of evidence, 3. A total of 48 patients with DDH who underwent periacetabular osteotomy were compared with 48 sex-matched patients who underwent hip arthroscopy for femoroacetabular impingement (FAI) between July 2014 and February 2021. All patients were skeletally mature. The Tönnis angle and lateral center-edge angle were measured on preoperative pelvis radiographs. Femoral version, trochlear depth, lateral trochlear inclination (LTI), tibial tubercle-trochlear groove distance (TTTG-d), and posterior lateral condylar angle (PLCA) were measured on preoperative magnetic resonance imaging scans of the symptomatic hip and ipsilateral knee. Continuous variables were compared between the patient groups using 2-sample Patients with DDH demonstrated a reduced trochlear depth compared with patients with FAI (3.6 vs 4.6 mm; Patients with DDH had reduced trochlear depth compared with patients with FAI, demonstrating a higher incidence of dysplastic trochlear features that may predispose patients to patellofemoral joint disease. Further research is needed to determine whether screening at-risk patients and treating TD will help to prevent symptomatic patellofemoral disease.

Sections du résumé

Background UNASSIGNED
Developmental dysplasia of the hip (DDH) and trochlear dysplasia (TD) are distinct pathologies with several important features in common. In addition to shared risk factors, both forms of dysplasia cause abnormal joint kinematics and force transmission, predisposing patients to pain, injuries to cartilage and soft tissue stabilizers, and ultimately arthritis.
Purpose UNASSIGNED
To evaluate for an association between hip dysplasia and TD in skeletally mature patients with symptomatic hip dysplasia.
Study Design UNASSIGNED
Cross-sectional study; Level of evidence, 3.
Methods UNASSIGNED
A total of 48 patients with DDH who underwent periacetabular osteotomy were compared with 48 sex-matched patients who underwent hip arthroscopy for femoroacetabular impingement (FAI) between July 2014 and February 2021. All patients were skeletally mature. The Tönnis angle and lateral center-edge angle were measured on preoperative pelvis radiographs. Femoral version, trochlear depth, lateral trochlear inclination (LTI), tibial tubercle-trochlear groove distance (TTTG-d), and posterior lateral condylar angle (PLCA) were measured on preoperative magnetic resonance imaging scans of the symptomatic hip and ipsilateral knee. Continuous variables were compared between the patient groups using 2-sample
Results UNASSIGNED
Patients with DDH demonstrated a reduced trochlear depth compared with patients with FAI (3.6 vs 4.6 mm;
Conclusion UNASSIGNED
Patients with DDH had reduced trochlear depth compared with patients with FAI, demonstrating a higher incidence of dysplastic trochlear features that may predispose patients to patellofemoral joint disease. Further research is needed to determine whether screening at-risk patients and treating TD will help to prevent symptomatic patellofemoral disease.

Identifiants

pubmed: 37822419
doi: 10.1177/23259671231200805
pii: 10.1177_23259671231200805
pmc: PMC10563471
doi:

Types de publication

Journal Article

Langues

eng

Pagination

23259671231200805

Informations de copyright

© The Author(s) 2023.

Déclaration de conflit d'intérêts

One or more of the authors has declared the following potential conflict of interest or source of funding: A.T.F. has received education payments from Evolution Surgical. S.L.S. has received education payments from Evolution Surgical and Elite Orthopedics; consulting fees from Bioventus, DJO, Kinamed, Linvatec, Olympus America, Ceterix Orthopaedics, Pacira Therapeutics, Smith & Nephew, Vericel, JRF Ortho, LifeNet Health, and Flexion Therapeutics; nonconsulting fees from Arthrex, Linvatec, Smith & Nephew, and Synthes GmbH; royalties from Linvatec and ConMed; hospitality payments from Aesculap Biologics; and honoraria from Flexion Therapeutics, JRF Ortho, and Vericel. K.G.S. has received education payments from Evolution Surgical and hospitality payments from Arthrex. AOSSM checks author disclosures against the Open Payments Database (OPD). AOSSM has not conducted an independent investigation on the OPD and disclaims any liability or responsibility relating thereto. Ethical approval for this study was obtained from Stanford University (No. 5136).

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Auteurs

Andrew T Fithian (AT)

Department of Orthopaedic Surgery, Stanford University, Redwood City, California, USA.

Ann E Richey (AE)

Department of Orthopaedic Surgery, Stanford University, Redwood City, California, USA.

Seth L Sherman (SL)

Department of Orthopaedic Surgery, Stanford University, Redwood City, California, USA.

Kevin G Shea (KG)

Department of Orthopaedic Surgery, Stanford University, Redwood City, California, USA.

Stephanie Y Pun (SY)

Department of Orthopaedic Surgery, Stanford University, Redwood City, California, USA.

Classifications MeSH