The Protective Effect of Chronic Intermittent Hypobaric Hypoxia on Preventing the Destruction of CD34

Aplastic anaemia Apoptosis Chronic intermittent hypobaric hypoxia Hematopoietic stem cells Th1/Th2 balance

Journal

Stem cell reviews and reports
ISSN: 2629-3277
Titre abrégé: Stem Cell Rev Rep
Pays: United States
ID NLM: 101752767

Informations de publication

Date de publication:
13 Oct 2023
Historique:
accepted: 11 09 2023
medline: 13 10 2023
pubmed: 13 10 2023
entrez: 13 10 2023
Statut: aheadofprint

Résumé

Aplastic anaemia (AA) is a haematopoietic disorder caused by immune-mediated attack on haematopoietic stem cells (HSCs). Stem cell transplantation and immunosuppressive therapy remain the major treatment choice for AA patients but have limited benefits and undesired side effects. The aim of our study was to clarify the protective role of immunity of chronic intermittent hypobaric hypoxia (CIHH) and the underlying mechanism in AA. Our integrative analysis demonstrated that CIHH pre-treatment significantly improved haematopoiesis and survival in an AA rat model. We further confirmed that CIHH pre-treatment was closely associated with the Th1/Th2 balance and a large number of negative regulatory haematopoietic factors, such as TNF-α and IFN-γ, produced by hyperactive Th1 lymphocytes released in AA rats, which induced the death program in a large number of CD34

Identifiants

pubmed: 37831395
doi: 10.1007/s12015-023-10631-0
pii: 10.1007/s12015-023-10631-0
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : National Natural Science Foundation of China
ID : 81900249
Organisme : Natural Science Foundation of Zhejiang Province
ID : LY23H020001
Organisme : Steel and Iron Foundation of Hebei Province
ID : H2020206003
Organisme : Natural Science Foundation of Liaoning Province
ID : 2022-MS-230

Informations de copyright

© 2023. The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature.

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Auteurs

Hui Guo (H)

Key Laboratory of Aging and Cancer Biology of Zhejiang Province, Hangzhou, China.
Key Laboratory of Infection and Immunity of CAS, Institute of Biophysics, Chinese Academy of Sciences, Beijing, China.

Yilin Zhao (Y)

Key Laboratory of Aging and Cancer Biology of Zhejiang Province, Hangzhou, China.
Department of Pathology and Pathophysiology, School of Basic Medical Sciences, Hangzhou Normal University, Hangzhou, China.

Rui Mu (R)

College of Basic Medicine, Hebei Medical University, Shijiazhuang, China.

Guangdao Zhang (G)

Department of Prosthodontics, School of Stomatology, China Medical University, Shenyang, China.

Shuxian Chen (S)

College of Basic Medicine, Hebei Medical University, Shijiazhuang, China.

Xinwei Cao (X)

College of Basic Medicine, Hebei Medical University, Shijiazhuang, China.

Kangcan Liu (K)

College of Basic Medicine, Hebei Medical University, Shijiazhuang, China.

Yiran Liu (Y)

College of Basic Medicine, Hebei Medical University, Shijiazhuang, China.

Baiyun Dai (B)

Key Laboratory of Aging and Cancer Biology of Zhejiang Province, Hangzhou, China.
Department of Pathology and Pathophysiology, School of Basic Medical Sciences, Hangzhou Normal University, Hangzhou, China.

Yuan Zhou (Y)

State Key Laboratory of Experimental Hematology, Institute of Hematology and Blood Diseases Hospital, Chinese Academy of Medical Sciences and Peking Union Medical College, Tianjin, China. yuanzhou@ihcams.ac.cn.

Chuan Wang (C)

Department of Pharmacology, Hebei Medical University, Shijiazhuang, China. wchpha@hotmail.com.

Jing Yang (J)

Key Laboratory of Aging and Cancer Biology of Zhejiang Province, Hangzhou, China. yjphy@126.com.
Department of Pathology and Pathophysiology, School of Basic Medical Sciences, Hangzhou Normal University, Hangzhou, China. yjphy@126.com.

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