Higher Infectivity of the Human Immunodeficiency Virus in Sensitive Cells with a Modification of the CCR5 Gene.


Journal

Doklady biological sciences : proceedings of the Academy of Sciences of the USSR, Biological sciences sections
ISSN: 1608-3105
Titre abrégé: Dokl Biol Sci
Pays: United States
ID NLM: 7505459

Informations de publication

Date de publication:
Aug 2023
Historique:
received: 03 02 2023
accepted: 10 03 2023
revised: 07 03 2023
medline: 1 11 2023
pubmed: 14 10 2023
entrez: 13 10 2023
Statut: ppublish

Résumé

As a natural mutation of the human ccr5 gene has been shown to confer resistance to human immunodeficiency virus type 1 (HIV-1) infection, a new avenue has opened in the development of alternative treatment approaches through genome editing. One of the two chemokine co-receptors of the plasma membrane is utilized by HIV-1 to infect CD4

Identifiants

pubmed: 37833581
doi: 10.1134/S0012496623700412
pii: 10.1134/S0012496623700412
doi:

Substances chimiques

CCR5 protein, human 0
Receptors, CCR5 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

251-254

Informations de copyright

© 2023. Pleiades Publishing, Ltd.

Références

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Auteurs

D N Nosik (DN)

Ivanovsky Institute of Virology, Gamaleya National Research Center of Epidemiology and Microbiology, Moscow, Russia. dnnosik@yandex.ru.

L B Kalnina (LB)

Ivanovsky Institute of Virology, Gamaleya National Research Center of Epidemiology and Microbiology, Moscow, Russia.

L M Selimova (LM)

Ivanovsky Institute of Virology, Gamaleya National Research Center of Epidemiology and Microbiology, Moscow, Russia.

A V Pronin (AV)

Ivanovsky Institute of Virology, Gamaleya National Research Center of Epidemiology and Microbiology, Moscow, Russia.

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