Regulatory T Cell Inhibition by P60 Combined with Adenoviral AFP Transduced Dendritic Cells for Immunotherapy of Hepatocellular Carcinoma.


Journal

Immunological investigations
ISSN: 1532-4311
Titre abrégé: Immunol Invest
Pays: England
ID NLM: 8504629

Informations de publication

Date de publication:
Nov 2023
Historique:
medline: 27 11 2023
pubmed: 17 10 2023
entrez: 17 10 2023
Statut: ppublish

Résumé

Vaccination with tumor-associated antigen-pulsed dendritic cells leads to specific T-cell response against hepatocellular carcinoma. However, clinical response has been shown to be limited. High regulatory T-cell count is associated with poor prognosis and seems to mediate immune tolerance in hepatocellular carcinoma. Forkhead box P3-peptide inhibitor P60 has been shown to specifically inhibit regulatory T-cell function in murine models. Aim of this study was to investigate whether P60 can improve the immune response induced by vaccination with adenovirus-transduced dendritic cells expressing alpha-fetoprotein in subcutaneous and orthotopic murine models for hepatocellular carcinoma. Mice developing subcutaneous or orthotopic HCC received daily treatment with P60 starting at different tumor stages. Additionally, mice were vaccinated twice with dendritic cells expressing alpha-fetoprotein. In a preventive setting prior to tumor engraftment, vaccination with alpha-fetoprotein-expressing dendritic cells significantly decreased tumor growth in a subcutaneous model ( In this study, we showed that vaccination with alpha-fetoprotein-expressing dendritic cells in combination with a specific inhibition of regulatory T-cells by using P60 leads to synergistic tumor inhibition and prolonged survival. This emphasizes the importance of regulatory T-cells inhibition for obtaining an effective antitumoral immune response in hepatocellular carcinoma.

Sections du résumé

BACKGROUND & AIMS UNASSIGNED
Vaccination with tumor-associated antigen-pulsed dendritic cells leads to specific T-cell response against hepatocellular carcinoma. However, clinical response has been shown to be limited. High regulatory T-cell count is associated with poor prognosis and seems to mediate immune tolerance in hepatocellular carcinoma. Forkhead box P3-peptide inhibitor P60 has been shown to specifically inhibit regulatory T-cell function in murine models. Aim of this study was to investigate whether P60 can improve the immune response induced by vaccination with adenovirus-transduced dendritic cells expressing alpha-fetoprotein in subcutaneous and orthotopic murine models for hepatocellular carcinoma.
METHODS UNASSIGNED
Mice developing subcutaneous or orthotopic HCC received daily treatment with P60 starting at different tumor stages. Additionally, mice were vaccinated twice with dendritic cells expressing alpha-fetoprotein.
RESULTS UNASSIGNED
In a preventive setting prior to tumor engraftment, vaccination with alpha-fetoprotein-expressing dendritic cells significantly decreased tumor growth in a subcutaneous model (
CONCLUSION UNASSIGNED
In this study, we showed that vaccination with alpha-fetoprotein-expressing dendritic cells in combination with a specific inhibition of regulatory T-cells by using P60 leads to synergistic tumor inhibition and prolonged survival. This emphasizes the importance of regulatory T-cells inhibition for obtaining an effective antitumoral immune response in hepatocellular carcinoma.

Identifiants

pubmed: 37846958
doi: 10.1080/08820139.2023.2261980
doi:

Substances chimiques

alpha-Fetoproteins 0
Cancer Vaccines 0

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

966-984

Auteurs

Farsaneh Sadeghlar (F)

Department of Medicine I, University Hospital of Bonn, Bonn, Germany.

Julia Seelemann (J)

Department of Medicine I, University Hospital of Bonn, Bonn, Germany.

Annabelle Vogt (A)

Department of Medicine I, University Hospital of Bonn, Bonn, Germany.

Christian Möhring (C)

Department of Medicine I, University Hospital of Bonn, Bonn, Germany.

Taotao Zhou (T)

Department of Medicine I, University Hospital of Bonn, Bonn, Germany.

Robert Mahn (R)

Department of Medicine I, University Hospital of Bonn, Bonn, Germany.

Miroslaw Kornek (M)

Department of Medicine I, University Hospital of Bonn, Bonn, Germany.

Veronika Lukacs-Kornek (V)

Institute for Molecular Medicine and Experimental Immunology, University Hospital of the Rheinische Friedrich-Wilhelms-University, Bonn, Germany.

Noelia Casares (N)

Program of Immunology and Immunotherapy, Center for Applied Medical Research, University of Navarra, Pamplona, Spain.

Juan José Lasarte (JJ)

Program of Immunology and Immunotherapy, Center for Applied Medical Research, University of Navarra, Pamplona, Spain.

Pablo Sarobe (P)

Program of Immunology and Immunotherapy, Center for Applied Medical Research, University of Navarra, Pamplona, Spain.

Cornelius van Beekum (C)

Program of Immunology and Immunotherapy, Center for Applied Medical Research, University of Navarra, Pamplona, Spain.

Hanno Matthaei (H)

Department of Visceral Surgery, University Hospital of Bonn, Bonn, Germany.

Steffen Manekeller (S)

Department of Visceral Surgery, University Hospital of Bonn, Bonn, Germany.

Jörg Kalff (J)

Department of Visceral Surgery, University Hospital of Bonn, Bonn, Germany.

Ingo G H Schmidt-Wolf (IGH)

Department of Medicine I, University Hospital of Bonn, Bonn, Germany.
Department of Integrated Oncology (CIO), University Hospital of Bonn, Bonn, Germany.

Christian P Strassburg (CP)

Department of Medicine I, University Hospital of Bonn, Bonn, Germany.

Maria A Gonzalez-Carmona (MA)

Department of Medicine I, University Hospital of Bonn, Bonn, Germany.

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Classifications MeSH