Orbitofrontal sulcal patterns in catatonia.

catatonic syndrome neurodevelopment disorder prefrontal cortex schizophrenia sulcogyral patterns

Journal

European psychiatry : the journal of the Association of European Psychiatrists
ISSN: 1778-3585
Titre abrégé: Eur Psychiatry
Pays: England
ID NLM: 9111820

Informations de publication

Date de publication:
19 Oct 2023
Historique:
pubmed: 19 10 2023
medline: 19 10 2023
entrez: 19 10 2023
Statut: epublish

Résumé

Catatonia is a psychomotor syndrome frequently observed in disorders with neurodevelopmental impairments, including psychiatric disorders such as schizophrenia. The orbitofrontal cortex (OFC) has been repeatedly associated with catatonia. It presents with an important interindividual morphological variability, with three distinct H-shaped sulcal patterns, types I, II, and III, based on the continuity of the medial and lateral orbital sulci. Types II and III have been identified as neurodevelopmental risk factors for schizophrenia. The sulcal pattern of the OFC has never been investigated in catatonia despite the role of the OFC in the pathophysiology and the neurodevelopmental component of catatonia. In this context, we performed a retrospective analysis of the OFC sulcal pattern in carefully selected homogeneous and matched subgroups of schizophrenia patients with catatonia ( Logistic regression analyses revealed a group effect on OFC sulcal pattern in the left ( Because the sulcal patterns are indirect markers of early brain development, our findings support a neurodevelopmental origin of catatonia and may shed light on the pathophysiology of this syndrome.

Sections du résumé

BACKGROUND BACKGROUND
Catatonia is a psychomotor syndrome frequently observed in disorders with neurodevelopmental impairments, including psychiatric disorders such as schizophrenia. The orbitofrontal cortex (OFC) has been repeatedly associated with catatonia. It presents with an important interindividual morphological variability, with three distinct H-shaped sulcal patterns, types I, II, and III, based on the continuity of the medial and lateral orbital sulci. Types II and III have been identified as neurodevelopmental risk factors for schizophrenia. The sulcal pattern of the OFC has never been investigated in catatonia despite the role of the OFC in the pathophysiology and the neurodevelopmental component of catatonia.
METHODS METHODS
In this context, we performed a retrospective analysis of the OFC sulcal pattern in carefully selected homogeneous and matched subgroups of schizophrenia patients with catatonia (
RESULTS RESULTS
Logistic regression analyses revealed a group effect on OFC sulcal pattern in the left (
CONCLUSION CONCLUSIONS
Because the sulcal patterns are indirect markers of early brain development, our findings support a neurodevelopmental origin of catatonia and may shed light on the pathophysiology of this syndrome.

Identifiants

pubmed: 37853748
doi: 10.1192/j.eurpsy.2023.2461
pii: S0924933823024616
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

e6

Subventions

Organisme : Fonds d'Etudes et de Recherche du Corps Médical

Auteurs

Mylène Moyal (M)

GHU Paris Psychiatrie et Neurosciences, Hôpital Sainte Anne, Paris, France.
Université Paris Cité, Institute of Psychiatry and Neuroscience of Paris (IPNP), INSERM U1266, IMA-Brain, Paris, France.

Alexandre Haroche (A)

GHU Paris Psychiatrie et Neurosciences, Hôpital Sainte Anne, Paris, France.
Université Paris Cité, Institute of Psychiatry and Neuroscience of Paris (IPNP), INSERM U1266, IMA-Brain, Paris, France.

David Attali (D)

GHU Paris Psychiatrie et Neurosciences, Hôpital Sainte Anne, Paris, France.
Physics for Medicine Paris, Inserm U1273, CNRS UMR 8063, ESPCI Paris, PSL University, Paris, France.

Ghita Dadi (G)

GHU Paris Psychiatrie et Neurosciences, Hôpital Sainte Anne, Paris, France.

Matthieu Raoelison (M)

Université Paris Cité, Laboratory for the Psychology of Child Development and Education, CNRS UMR 8240, Sorbonne, Paris, France.

Alice Le Berre (A)

GHU Paris Psychiatrie et Neurosciences, Hôpital Sainte Anne, Paris, France.
Université Paris Cité, Institute of Psychiatry and Neuroscience of Paris (IPNP), INSERM U1266, IMA-Brain, Paris, France.

Anton Iftimovici (A)

GHU Paris Psychiatrie et Neurosciences, Hôpital Sainte Anne, Paris, France.
Université Paris Cité, Institute of Psychiatry and Neuroscience of Paris (IPNP), INSERM U1266, IMA-Brain, Paris, France.
NeuroSpin, Atomic Energy Commission, Gif sur Yvette, France.

Boris Chaumette (B)

GHU Paris Psychiatrie et Neurosciences, Hôpital Sainte Anne, Paris, France.
Université Paris Cité, Institute of Psychiatry and Neuroscience of Paris (IPNP), INSERM U1266, IMA-Brain, Paris, France.
Department of Psychiatry, McGill University, Montreal, QC, Canada.

Sylvain Leroy (S)

GHU Paris Psychiatrie et Neurosciences, Hôpital Sainte Anne, Paris, France.

Sylvain Charron (S)

GHU Paris Psychiatrie et Neurosciences, Hôpital Sainte Anne, Paris, France.
Université Paris Cité, Institute of Psychiatry and Neuroscience of Paris (IPNP), INSERM U1266, IMA-Brain, Paris, France.

Clément Debacker (C)

GHU Paris Psychiatrie et Neurosciences, Hôpital Sainte Anne, Paris, France.
Université Paris Cité, Institute of Psychiatry and Neuroscience of Paris (IPNP), INSERM U1266, IMA-Brain, Paris, France.

Catherine Oppenheim (C)

GHU Paris Psychiatrie et Neurosciences, Hôpital Sainte Anne, Paris, France.
Université Paris Cité, Institute of Psychiatry and Neuroscience of Paris (IPNP), INSERM U1266, IMA-Brain, Paris, France.

Arnaud Cachia (A)

Université Paris Cité, Institute of Psychiatry and Neuroscience of Paris (IPNP), INSERM U1266, IMA-Brain, Paris, France.
Université Paris Cité, Laboratory for the Psychology of Child Development and Education, CNRS UMR 8240, Sorbonne, Paris, France.

Marion Plaze (M)

GHU Paris Psychiatrie et Neurosciences, Hôpital Sainte Anne, Paris, France.
Université Paris Cité, Institute of Psychiatry and Neuroscience of Paris (IPNP), INSERM U1266, IMA-Brain, Paris, France.

Classifications MeSH