The Ca2+/Calmodulin-dependent Calcineurin/NFAT Signaling Pathway in the Pathogenesis of Insulin Resistance in Skeletal Muscle.
Humans
Insulin Resistance
Calmodulin
/ metabolism
Calcineurin
/ metabolism
Calcium
/ metabolism
Muscle Fibers, Fast-Twitch
/ metabolism
Muscle Fibers, Slow-Twitch
/ metabolism
Muscle, Skeletal
/ metabolism
NFATC Transcription Factors
/ metabolism
Signal Transduction
/ physiology
Insulin
/ metabolism
Glucose
/ metabolism
Journal
Experimental and clinical endocrinology & diabetes : official journal, German Society of Endocrinology [and] German Diabetes Association
ISSN: 1439-3646
Titre abrégé: Exp Clin Endocrinol Diabetes
Pays: Germany
ID NLM: 9505926
Informations de publication
Date de publication:
Nov 2023
Nov 2023
Historique:
medline:
16
11
2023
pubmed:
25
10
2023
entrez:
24
10
2023
Statut:
ppublish
Résumé
Skeletal muscle is the tissue directly involved in insulin-stimulated glucose uptake. Glucose is the primary energy substrate for contracting muscles, and proper metabolism of glucose is essential for health. Contractile activity and the associated Ca
Substances chimiques
Calmodulin
0
Calcineurin
EC 3.1.3.16
Calcium
SY7Q814VUP
NFATC Transcription Factors
0
Insulin
0
Glucose
IY9XDZ35W2
Types de publication
Review
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Pagination
589-594Informations de copyright
Thieme. All rights reserved.
Déclaration de conflit d'intérêts
The authors declare that they have no conflict of interest.