Effects of novel androgen receptor signaling inhibitors on PSMA PET signal intensity in patients with castrate-resistant prostate cancer: a prospective exploratory serial imaging study.

Androgen receptor Hormonal treatment PSMA PET PSMA flare Prostate cancer

Journal

EJNMMI research
ISSN: 2191-219X
Titre abrégé: EJNMMI Res
Pays: Germany
ID NLM: 101560946

Informations de publication

Date de publication:
30 Oct 2023
Historique:
received: 15 08 2023
accepted: 22 10 2023
medline: 30 10 2023
pubmed: 30 10 2023
entrez: 30 10 2023
Statut: epublish

Résumé

PSMA expression is influenced by hormonal status. We evaluated changes in PSA and whole-body 68Ga-PSMA-11 PET/CT (WB-PSMA PET) after initiation of androgen receptor signaling inhibitors (ARSi). Prospectively enrolled patients with metastatic castration-resistant prostate cancer (mCRPC) initiating ARSi underwent serial PSA measurements and WB-PSMA PET at baseline, 1-week, and 3-months post-ARSi. We correlated WB-PSMA PET metrics and PSA kinetics after ARSi to 1-year clinical outcome. Due to low enrollment rate, the study was closed before reaching the recruitment goal of 30 patients. Nine patients were enrolled. At 1-year, unfavorable outcome was documented in 6/9 (66%) patients. Nine/9 patients completed PSMA PET at 1-week, 5/9 at 3-months. Changes in PSA, PSMA-VOL, SUVmean and SUVmax were - 12%, + 5%, + 3%, and + 10% at 1-week, - 42%, - 16%, - 15% and - 17% at 3-months, respectively. Our prospective trial involving 9 mCRPC patients initiating ARSi did not show significant modulation of PSMA expression measured on WB-PSMA PET at 1-week. This study was registered on clinicaltrials.gov (NCT04279561).

Sections du résumé

BACKGROUND BACKGROUND
PSMA expression is influenced by hormonal status. We evaluated changes in PSA and whole-body 68Ga-PSMA-11 PET/CT (WB-PSMA PET) after initiation of androgen receptor signaling inhibitors (ARSi).
METHODS METHODS
Prospectively enrolled patients with metastatic castration-resistant prostate cancer (mCRPC) initiating ARSi underwent serial PSA measurements and WB-PSMA PET at baseline, 1-week, and 3-months post-ARSi. We correlated WB-PSMA PET metrics and PSA kinetics after ARSi to 1-year clinical outcome.
RESULTS RESULTS
Due to low enrollment rate, the study was closed before reaching the recruitment goal of 30 patients. Nine patients were enrolled. At 1-year, unfavorable outcome was documented in 6/9 (66%) patients. Nine/9 patients completed PSMA PET at 1-week, 5/9 at 3-months. Changes in PSA, PSMA-VOL, SUVmean and SUVmax were - 12%, + 5%, + 3%, and + 10% at 1-week, - 42%, - 16%, - 15% and - 17% at 3-months, respectively.
CONCLUSIONS CONCLUSIONS
Our prospective trial involving 9 mCRPC patients initiating ARSi did not show significant modulation of PSMA expression measured on WB-PSMA PET at 1-week. This study was registered on clinicaltrials.gov (NCT04279561).

Identifiants

pubmed: 37902861
doi: 10.1186/s13550-023-01048-4
pii: 10.1186/s13550-023-01048-4
pmc: PMC10616012
doi:

Banques de données

ClinicalTrials.gov
['NCT04279561']

Types de publication

Journal Article

Langues

eng

Pagination

95

Informations de copyright

© 2023. The Author(s).

Références

Proc Natl Acad Sci U S A. 2011 Jun 7;108(23):9578-82
pubmed: 21606347
Eur J Nucl Med Mol Imaging. 2023 Jan;50(2):613-621
pubmed: 36161511
Cancer Res. 1994 Apr 1;54(7):1807-11
pubmed: 7511053
BJU Int. 2021 Aug;128(2):127-130
pubmed: 34318983
J Nucl Med. 2019 Sep;60(9):1277-1283
pubmed: 30850484
J Natl Compr Canc Netw. 2019 May 1;17(5):479-505
pubmed: 31085757
Clin Nucl Med. 2018 Nov;43(11):e404-e406
pubmed: 30222680
Nucl Med Mol Imaging. 2017 Sep;51(3):202-211
pubmed: 28878845
J Nucl Med. 2019 Jul;60(7):950-954
pubmed: 30552200
EJNMMI Res. 2018 Oct 29;8(1):96
pubmed: 30374743
Eur Urol Oncol. 2018 May;1(1):78-82
pubmed: 31100231

Auteurs

Ida Sonni (I)

Department of Molecular and Medical Pharmacology, University of California, Los Angeles, Los Angeles, CA, USA. isonni@mednet.ucla.edu.
Department of Radiological Sciences, David Geffen School of Medicine, University of California, Los Angeles, Los Angeles, CA, 90095-7370, USA. isonni@mednet.ucla.edu.
Department of Experimental and Clinical Medicine, University Magna Graecia, Catanzaro, Italy. isonni@mednet.ucla.edu.

Andrei Gafita (A)

Department of Molecular and Medical Pharmacology, University of California, Los Angeles, Los Angeles, CA, USA.

Lena M Unterrainer (LM)

Department of Molecular and Medical Pharmacology, University of California, Los Angeles, Los Angeles, CA, USA.
Department of Nuclear Medicine, Ludwig Maximilian University of Munich LMU, Munich, Germany.

Rejah M Alano (RM)

Department of Molecular and Medical Pharmacology, University of California, Los Angeles, Los Angeles, CA, USA.

Stephanie Lira (S)

Department of Molecular and Medical Pharmacology, University of California, Los Angeles, Los Angeles, CA, USA.

John Shen (J)

Department of Medical Oncology, University of California, Los Angeles, Los Angeles, CA, USA.

Alexandra Drakaki (A)

Department of Medical Oncology, University of California, Los Angeles, Los Angeles, CA, USA.

Tristan Grogan (T)

Department of Medicine Statistics Core, University of California, Los Angeles, Los Angeles, CA, USA.

Matthew B Rettig (MB)

Department of Medical Oncology, University of California, Los Angeles, Los Angeles, CA, USA.

Johannes Czernin (J)

Department of Molecular and Medical Pharmacology, University of California, Los Angeles, Los Angeles, CA, USA.

Jeremie Calais (J)

Department of Molecular and Medical Pharmacology, University of California, Los Angeles, Los Angeles, CA, USA.

Classifications MeSH