Possible Molecular Targeting of Biofilm-Associated Genes by Nano-Ag in Candida albicans.

Biofilms Gene expression Nanoparticles

Journal

Applied biochemistry and biotechnology
ISSN: 1559-0291
Titre abrégé: Appl Biochem Biotechnol
Pays: United States
ID NLM: 8208561

Informations de publication

Date de publication:
03 Nov 2023
Historique:
accepted: 17 10 2023
medline: 3 11 2023
pubmed: 3 11 2023
entrez: 3 11 2023
Statut: aheadofprint

Résumé

The treatment of candidiasis infections is hindered by the presence of biofilms. Here, we report the biofilm-associated genes as potential molecular targets by silver nanoparticles (nano-Ag) in Candida albicans. Nano-Ag was biosynthesized using Bacillus licheniformis, Bacillus cereus, and Fusarium oxysporum. The physicochemical properties of the microbial-synthesized of nano-Ag are widely characterized by visual observation, ultraviolet-visible spectroscopy, scanning electron microscopy, X-ray diffraction spectroscopy, and Fourier transform infrared spectroscopy. Characterization results revealed the formation of nano-Ag. Antiplanktonic cells and antibiofilm activities of nano-Ag were also demonstrated by minimum inhibition concentrations (MIC), minimum fungicidal concentration (MFC), MFC/MIC ratio, crystal violet staining, 2,3-bis (2-methoxy-4-nitro-5 sulfophenyl)-5-[(phenylamino) carbonyl]-2H-tetrazolium hydroxide (XTT), and microscopic image analysis. We have analyzed the expressions of biofilm-associated genes in C. albicans treated with different concentrations of nano-Ag based on MIC. The expression profile of BCR1, ALS1, ALS3, HWP1, and ECE1 showed downregulated genes involved in these pathways by the treatment with nanoparticles. Negative regulators, TUP1, NRG1, and TOR1, were upregulated by the treatment of nano-Ag. Our study suggests that nano-Ag affects gene expression and may subsequently decrease the pathogenesis of C. albicans by inhibiting biofilm formation. Molecular targeting of biofilm-associated genes involved in biofilm formation by nano-Ag may be an effective treatment strategy for candidiasis infections.

Identifiants

pubmed: 37922031
doi: 10.1007/s12010-023-04758-6
pii: 10.1007/s12010-023-04758-6
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© 2023. The Author(s), under exclusive licence to Springer Science+Business Media, LLC, part of Springer Nature.

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Auteurs

Mahbobeh Baghiat Esfahani (MB)

Department of Microbiology, Shiraz Branch, Islamic Azad University, Shiraz, Iran.

Alireza Khodavandi (A)

Department of Biology, Gachsaran Branch, Islamic Azad University, Gachsaran, Iran. alireza_Khodavandi@yahoo.com.

Fahimeh Alizadeh (F)

Department of Biology, Gachsaran Branch, Islamic Azad University, Gachsaran, Iran.

Nima Bahador (N)

Department of Microbiology, Shiraz Branch, Islamic Azad University, Shiraz, Iran.

Classifications MeSH