Differential longitudinal changes of hippocampal subfields in patients with anorexia nervosa.

Anorexia nervosa FreeSurfer Hippocampus Leptin Longitudinal study

Journal

Psychiatry and clinical neurosciences
ISSN: 1440-1819
Titre abrégé: Psychiatry Clin Neurosci
Pays: Australia
ID NLM: 9513551

Informations de publication

Date de publication:
28 Nov 2023
Historique:
revised: 31 10 2023
received: 11 08 2023
accepted: 26 11 2023
medline: 29 11 2023
pubmed: 29 11 2023
entrez: 29 11 2023
Statut: aheadofprint

Résumé

Anorexia nervosa (AN) is a mental disorder characterized by dietary restriction, fear of gaining weight, and distorted body image. Recent studies indicate the hippocampus, crucial for learning and memory, may be affected in AN, yet subfield-specific effects remain unclear. Here we investigated hippocampal subfield alterations in acute AN, changes following weight-restoration and their associations with leptin levels. T1-weighted magnetic resonance imaging scans were processed using FreeSurfer. We compared 22 left and right hemispheric hippocampal subfield volumes cross-sectionally and longitudinally in females with acute AN (n = 165 at baseline, n = 110 after partial weight-restoration), healthy females (n = 271, HC), and females after long-term recovery from AN (n = 79) using linear models. We found that most hippocampal subfield volumes were significantly reduced in AN compared to HC (~ - 3.9%). Certain areas like the subiculum exhibited no significant reduction in the acute state of AN, while other areas, like the hippocampal tail, showed strong decreases (~ - 9%). Following short-term weight-recovery, most subfields increased in volume. Comparisons between participants after long-term weight-recovery and HC yielded no differences. The hippocampal tail volume was positively associated with leptin levels in AN independent of BMI. Our study provides evidence of differential volumetric differences in hippocampal subfields between individuals with AN and HC and almost complete normalization after weight rehabilitation. These alterations are spatially inhomogeneous and more pronounced compared to other major mental disorders (e.g., major depressive disorder, schizophrenia). We provide novel insights linking hypoleptinemia to hippocampal subfield alterations hinting towards clinical relevance of leptin normalization in AN recovery. This article is protected by copyright. All rights reserved.

Sections du résumé

BACKGROUND BACKGROUND
Anorexia nervosa (AN) is a mental disorder characterized by dietary restriction, fear of gaining weight, and distorted body image. Recent studies indicate the hippocampus, crucial for learning and memory, may be affected in AN, yet subfield-specific effects remain unclear. Here we investigated hippocampal subfield alterations in acute AN, changes following weight-restoration and their associations with leptin levels.
METHODS METHODS
T1-weighted magnetic resonance imaging scans were processed using FreeSurfer. We compared 22 left and right hemispheric hippocampal subfield volumes cross-sectionally and longitudinally in females with acute AN (n = 165 at baseline, n = 110 after partial weight-restoration), healthy females (n = 271, HC), and females after long-term recovery from AN (n = 79) using linear models.
RESULTS RESULTS
We found that most hippocampal subfield volumes were significantly reduced in AN compared to HC (~ - 3.9%). Certain areas like the subiculum exhibited no significant reduction in the acute state of AN, while other areas, like the hippocampal tail, showed strong decreases (~ - 9%). Following short-term weight-recovery, most subfields increased in volume. Comparisons between participants after long-term weight-recovery and HC yielded no differences. The hippocampal tail volume was positively associated with leptin levels in AN independent of BMI.
CONCLUSIONS CONCLUSIONS
Our study provides evidence of differential volumetric differences in hippocampal subfields between individuals with AN and HC and almost complete normalization after weight rehabilitation. These alterations are spatially inhomogeneous and more pronounced compared to other major mental disorders (e.g., major depressive disorder, schizophrenia). We provide novel insights linking hypoleptinemia to hippocampal subfield alterations hinting towards clinical relevance of leptin normalization in AN recovery. This article is protected by copyright. All rights reserved.

Identifiants

pubmed: 38018338
doi: 10.1111/pcn.13626
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

This article is protected by copyright. All rights reserved.

Auteurs

Klaas Bahnsen (K)

Translational Developmental Neuroscience Section, Division of Psychological and Social Medicine and Developmental Neuroscience, Faculty of Medicine, Technische Universität Dresden, Dresden, Germany.

Marie-Louis Wronski (ML)

Translational Developmental Neuroscience Section, Division of Psychological and Social Medicine and Developmental Neuroscience, Faculty of Medicine, Technische Universität Dresden, Dresden, Germany.
Neuroendocrine Unit, Department of Medicine, Massachusetts General Hospital and Harvard Medical School, Boston, Massachusetts, USA.

Johanna Louise Keeler (JL)

Translational Developmental Neuroscience Section, Division of Psychological and Social Medicine and Developmental Neuroscience, Faculty of Medicine, Technische Universität Dresden, Dresden, Germany.
Department of Psychological Medicine, Institute of Psychiatry, Psychology & Neuroscience, King's College London, London, UK.

Joseph A King (JA)

Translational Developmental Neuroscience Section, Division of Psychological and Social Medicine and Developmental Neuroscience, Faculty of Medicine, Technische Universität Dresden, Dresden, Germany.

Quirina Preusker (Q)

Translational Developmental Neuroscience Section, Division of Psychological and Social Medicine and Developmental Neuroscience, Faculty of Medicine, Technische Universität Dresden, Dresden, Germany.

Theresa Kolb (T)

Translational Developmental Neuroscience Section, Division of Psychological and Social Medicine and Developmental Neuroscience, Faculty of Medicine, Technische Universität Dresden, Dresden, Germany.

Kerstin Weidner (K)

Department of Psychotherapy and Psychosomatic Medicine, Faculty of Medicine, Technische Universität Dresden, Dresden, Germany.

Veit Roessner (V)

Department of Child and Adolescent Psychiatry, Faculty of Medicine, Technische Universität Dresden, Dresden, Germany.

Fabio Bernardoni (F)

Translational Developmental Neuroscience Section, Division of Psychological and Social Medicine and Developmental Neuroscience, Faculty of Medicine, Technische Universität Dresden, Dresden, Germany.

Stefan Ehrlich (S)

Translational Developmental Neuroscience Section, Division of Psychological and Social Medicine and Developmental Neuroscience, Faculty of Medicine, Technische Universität Dresden, Dresden, Germany.
Eating Disorder Research and Treatment Center, Department of Child and Adolescent Psychiatry, Faculty of Medicine, Technische Universität Dresden, Dresden, Germany.

Classifications MeSH