Human leukocyte immunoglobulin-like receptors in health and disease.
LILR
autoimmunity
cancer
immune tolerance
immunomodulation
immunotherapy
infection
Journal
Frontiers in immunology
ISSN: 1664-3224
Titre abrégé: Front Immunol
Pays: Switzerland
ID NLM: 101560960
Informations de publication
Date de publication:
2023
2023
Historique:
received:
25
08
2023
accepted:
20
09
2023
medline:
1
12
2023
pubmed:
29
11
2023
entrez:
29
11
2023
Statut:
epublish
Résumé
Human leukocyte immunoglobulin (Ig)-like receptors (LILR) are a family of 11 innate immunomodulatory receptors, primarily expressed on lymphoid and myeloid cells. LILRs are either activating (LILRA) or inhibitory (LILRB) depending on their associated signalling domains (D). With the exception of the soluble LILRA3, LILRAs mediate immune activation, while LILRB1-5 primarily inhibit immune responses and mediate tolerance. Abnormal expression and function of LILRs is associated with a range of pathologies, including immune insufficiency (infection and malignancy) and overt immune responses (autoimmunity and alloresponses), suggesting LILRs may be excellent candidates for targeted immunotherapies. This review will discuss the biology and clinical relevance of this extensive family of immune receptors and will summarise the recent developments in targeting LILRs in disease settings, such as cancer, with an update on the clinical trials investigating the therapeutic targeting of these receptors.
Identifiants
pubmed: 38022598
doi: 10.3389/fimmu.2023.1282874
pmc: PMC10679719
doi:
Substances chimiques
Receptors, Immunologic
0
Immunoglobulins
0
LILRA3 protein, human
0
Types de publication
Journal Article
Review
Langues
eng
Sous-ensembles de citation
IM
Pagination
1282874Informations de copyright
Copyright © 2023 Redondo-García, Barritt, Papagregoriou, Yeboah, Frendeus, Cragg and Roghanian.
Déclaration de conflit d'intérêts
BF is an employee of BioInvent International. AR and MSC receive institutional support from BioInvent International. AR acts as a consultant for Epsilogen. MSC has consulted for BioInvent International, Boehringer Ingelheim, GSK, Radiant, iteos, Surrozen, Hanall and Mestag and received research funding from BioInvent International, GSK, UCB and iTeos. He is a member of the GSK Immunology Network. The remaining authors declare that the research was conducted in the absence of any commercial or financial relationships that could be construed as a potential conflict of interest.
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