Mmp2 Deficiency Leads to Defective Parturition and High Dystocia Rates in Mice.
MMP2
MMP9
double knockout
dystocia
fibrosis
gelatinase
matrix metalloproteinase
myometrium
uterine
Journal
International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791
Informations de publication
Date de publication:
27 Nov 2023
27 Nov 2023
Historique:
received:
01
10
2023
revised:
14
11
2023
accepted:
22
11
2023
medline:
9
12
2023
pubmed:
9
12
2023
entrez:
9
12
2023
Statut:
epublish
Résumé
Parturition is the final and essential step for mammalian reproduction. While the uterus is quiescent during pregnancy, fundamental changes arise in the myometrial contractility, inducing fetal expulsion. Extracellular matrix (ECM) remodeling is fundamental for these events. The gelatinases subgroup of matrix metalloproteinases (MMPs), MMP2 and MMP9, participate in uterine ECM remodeling throughout pregnancy and parturition. However, their loss-of-function effect is unknown. Here, we determined the result of eliminating
Identifiants
pubmed: 38069145
pii: ijms242316822
doi: 10.3390/ijms242316822
pmc: PMC10706207
pii:
doi:
Types de publication
Journal Article
Langues
eng
Sous-ensembles de citation
IM
Subventions
Organisme : Israel Science Foundation
ID : 1252/19
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