Expression of Tissue Factor and Platelet/Leukocyte Markers on Extracellular Vesicles Reflect Platelet-Leukocyte Interaction in Severe COVID-19.

COVID-19 extracellular vesicles immunothrombosis platelet–leukocyte aggregates tissue factor

Journal

International journal of molecular sciences
ISSN: 1422-0067
Titre abrégé: Int J Mol Sci
Pays: Switzerland
ID NLM: 101092791

Informations de publication

Date de publication:
28 Nov 2023
Historique:
received: 11 10 2023
revised: 21 11 2023
accepted: 24 11 2023
medline: 9 12 2023
pubmed: 9 12 2023
entrez: 9 12 2023
Statut: epublish

Résumé

Severe COVID-19 is frequently associated with thromboembolic complications. Increased platelet activation and platelet-leukocyte aggregate formation can amplify thrombotic responses by inducing tissue factor (TF) expression on leukocytes. Here, we characterized TF-positive extracellular vesicles (EVs) and their cellular origin in 12 patients suffering from severe COVID-19 (time course, 134 samples overall) and 25 healthy controls. EVs exposing phosphatidylserine (PS) were characterized by flow cytometry. Their cellular origin was determined by staining with anti-CD41, anti-CD45, anti-CD235a, and anti-CD105 as platelet, leukocyte, red blood cell, and endothelial markers. We further investigated the association of EVs with TF, platelet factor 4 (PF4), C-reactive protein (CRP), and high mobility group box-1 protein (HMGB-1). COVID-19 patients showed higher levels of PS-exposing EVs compared to controls. The majority of these EVs originated from platelets. A higher amount of EVs in patient samples was associated with CRP, HMGB-1, PF4, and TF as compared to EVs from healthy donors. In COVID-19 samples, 16.5% of all CD41

Identifiants

pubmed: 38069209
pii: ijms242316886
doi: 10.3390/ijms242316886
pmc: PMC10707108
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Lower Austrian Society for Research Funding (GFF)
ID : K3-F-744/005-2019
Organisme : Technology Fund of Lower Austria
ID : WST3-F-5030664/032-2022

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Auteurs

Tanja Eichhorn (T)

Center for Biomedical Technology, Department for Biomedical Research, University for Continuing Education Krems, 3500 Krems, Austria.

René Weiss (R)

Center for Biomedical Technology, Department for Biomedical Research, University for Continuing Education Krems, 3500 Krems, Austria.

Silke Huber (S)

Institute of Hygiene and Medical Microbiology, Medical University of Innsbruck, 6020 Innsbruck, Austria.

Marie Ebeyer-Masotta (M)

Center for Biomedical Technology, Department for Biomedical Research, University for Continuing Education Krems, 3500 Krems, Austria.

Marwa Mostageer (M)

Center for Biomedical Technology, Department for Biomedical Research, University for Continuing Education Krems, 3500 Krems, Austria.

Robert Emprechtinger (R)

Faculty of Health and Medicine, University for Continuing Education Krems, 3500 Krems, Austria.

Ludwig Knabl (L)

Department of Internal Medicine, Hospital St. Vinzenz, 6511 Zams, Austria.

Ludwig Knabl (L)

TyrolPath, 6511 Zams, Austria.

Reinhard Würzner (R)

Institute of Hygiene and Medical Microbiology, Medical University of Innsbruck, 6020 Innsbruck, Austria.

Viktoria Weber (V)

Center for Biomedical Technology, Department for Biomedical Research, University for Continuing Education Krems, 3500 Krems, Austria.

Classifications MeSH