Usefulness of thalamic beta activity for closed-loop therapy in essential tremor.


Journal

Scientific reports
ISSN: 2045-2322
Titre abrégé: Sci Rep
Pays: England
ID NLM: 101563288

Informations de publication

Date de publication:
15 Dec 2023
Historique:
received: 18 05 2023
accepted: 08 12 2023
medline: 16 12 2023
pubmed: 16 12 2023
entrez: 15 12 2023
Statut: epublish

Résumé

A partial loss of effectiveness of deep brain stimulation of the ventral intermediate nucleus of the thalamus (VIM) has been reported in some patients with essential tremor (ET), possibly due to habituation to permanent stimulation. This study focused on the evolution of VIM local-field potentials (LFPs) data over time to assess the long-term feasibility of closed-loop therapy based on thalamic activity. We performed recordings of thalamic LFPs in 10 patients with severe ET using the ACTIVA™ PC + S (Medtronic plc.) allowing both recordings and stimulation in the same region. Particular attention was paid to describing the evolution of LFPs over time from 3 to 24 months after surgery when the stimulation was Off. We demonstrated a significant decrease in high-beta LFPs amplitude during movements inducing tremor in comparison to the rest condition 3 months after surgery (1.91 ± 0.89 at rest vs. 1.27 ± 1.37 µV

Identifiants

pubmed: 38102180
doi: 10.1038/s41598-023-49511-5
pii: 10.1038/s41598-023-49511-5
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

22332

Subventions

Organisme : Medtronic
ID : material

Informations de copyright

© 2023. The Author(s).

Références

Louis, E. D. & Ferreira, J. J. How common is the most common adult movement disorder? Update on the worldwide prevalence of essential tremor. Mov. Disord. 25, 534–541 (2010).
pubmed: 20175185 doi: 10.1002/mds.22838
Sharifi, S., Nederveen, A. J., Booij, J. & van Rootselaar, A.-F. Neuroimaging essentials in essential tremor: A systematic review. Neuroimage Clin. 5, 217–231 (2014).
pubmed: 25068111 pmcid: 4110352 doi: 10.1016/j.nicl.2014.05.003
Louis, E. D. & Faust, P. L. Essential tremor pathology: Neurodegeneration and reorganization of neuronal connections. Nat. Rev. Neurol. 16, 69–83 (2020).
pubmed: 31959938 doi: 10.1038/s41582-019-0302-1
Lee, M. et al. Decreased EAAT2 protein expression in the essential tremor cerebellar cortex. Acta Neuropathol. Commun. 2, 157 (2014).
pubmed: 25391854 pmcid: 4239402 doi: 10.1186/s40478-014-0157-z
Paris-Robidas, S. et al. Defective dentate nucleus GABA receptors in essential tremor. Brain 135, 105–116 (2012).
pubmed: 22120148 doi: 10.1093/brain/awr301
Buijink, A. W. G. et al. Motor network disruption in essential tremor: A functional and effective connectivity study. Brain 138, 2934–2947 (2015).
pubmed: 26248468 doi: 10.1093/brain/awv225
Binder, D. K., Shah, B. B. & Elias, W. J. Focused ultrasound and other lesioning in the treatment of tremor. J. Neurol. Sci. 435, 120193 (2022).
pubmed: 35259650 doi: 10.1016/j.jns.2022.120193
Rehncrona, S. et al. Long-term efficacy of thalamic deep brain stimulation for tremor: Double-blind assessments. Mov. Disord. 18, 163–170 (2003).
pubmed: 12539209 doi: 10.1002/mds.10309
Blomstedt, P., Hariz, G.-M., Hariz, M. I. & Koskinen, L.-O.D. Thalamic deep brain stimulation in the treatment of essential tremor: A long-term follow-up. Br. J. Neurosurg. 21, 504–509 (2007).
pubmed: 17922323 doi: 10.1080/02688690701552278
Shih, L. C., LaFaver, K., Lim, C., Papavassiliou, E. & Tarsy, D. Loss of benefit in VIM thalamic deep brain stimulation (DBS) for essential tremor (ET): How prevalent is it?. Parkinsonism Relat. Disord. 19, 676–679 (2013).
pubmed: 23582712 doi: 10.1016/j.parkreldis.2013.03.006
Velisar, A. et al. Dual threshold neural closed loop deep brain stimulation in Parkinson disease patients. Brain Stimul. 12, 868–876 (2019).
pubmed: 30833216 doi: 10.1016/j.brs.2019.02.020
Swann, N. C. et al. Adaptive deep brain stimulation for Parkinson’s disease using motor cortex sensing. J. Neural Eng. 15, 046006 (2018).
pubmed: 29741160 pmcid: 6021210 doi: 10.1088/1741-2552/aabc9b
Guidetti, M. et al. Clinical perspectives of adaptive deep brain stimulation. Brain Stimul. 14, 1238–1247 (2021).
pubmed: 34371211 doi: 10.1016/j.brs.2021.07.063
Diesburg, D. A., Greenlee, J. D. & Wessel, J. R. Cortico-subcortical β burst dynamics underlying movement cancellation in humans. Elife 10, e70270 (2021).
pubmed: 34874267 pmcid: 8691838 doi: 10.7554/eLife.70270
Graupe, D., Basu, I., Tuninetti, D., Vannemreddy, P. & Slavin, K. V. Adaptively controlling deep brain stimulation in essential tremor patient via surface electromyography. Neurol. Res. 32, 899–904 (2010).
pubmed: 20712926 doi: 10.1179/016164110X12767786356354
Yamamoto, T. et al. On-demand control system for deep brain stimulation for treatment of intention tremor. Neuromodulation 16, 230–235 (2013).
pubmed: 23094990 doi: 10.1111/j.1525-1403.2012.00521.x
Cagnan, H. et al. Stimulating at the right time: Phase-specific deep brain stimulation. Brain 140, 132–145 (2017).
pubmed: 28007997 doi: 10.1093/brain/aww286
Paradiso, G. et al. Involvement of human thalamus in the preparation of self-paced movement. Brain 127, 2717–2731 (2004).
pubmed: 15329354 doi: 10.1093/brain/awh288
Tan, H. et al. Decoding voluntary movements and postural tremor based on thalamic LFPs as a basis for closed-loop stimulation for essential tremor. Brain Stimul. 12, 858–867 (2019).
pubmed: 30827864 pmcid: 6600875 doi: 10.1016/j.brs.2019.02.011
Pedrosa, D. J. et al. Thalamomuscular coherence in essential tremor: Hen or egg in the emergence of tremor?. J. Neurosci. 34, 14475–14483 (2014).
pubmed: 25339758 pmcid: 6608397 doi: 10.1523/JNEUROSCI.0087-14.2014
Kane, A., Hutchison, W. D., Hodaie, M., Lozano, A. M. & Dostrovsky, J. O. Enhanced synchronization of thalamic theta band local field potentials in patients with essential tremor. Experim. Neurol. 217, 171–176 (2009).
doi: 10.1016/j.expneurol.2009.02.005
Holdefer, R. N., Cohen, B. A. & Greene, K. A. Intraoperative local field recording for deep brain stimulation in Parkinson’s disease and essential tremor. Mov. Disord. 25, 2067–2075 (2010).
pubmed: 20721922 doi: 10.1002/mds.23232
Opri, E. et al. Chronic embedded cortico-thalamic closed-loop deep brain stimulation for the treatment of essential tremor. Sci. Transl. Med. 12, eaay7680 (2020).
pubmed: 33268512 pmcid: 8182660 doi: 10.1126/scitranslmed.aay7680
Beck, A. T. & Beck, R. W. Screening depressed patients in family practice. A rapid technic. Postgrad. Med. 52, 81–85 (1972).
pubmed: 4635613 doi: 10.1080/00325481.1972.11713319
Gardner, R., Oliver-Muñoz, S., Fisher, L. & Empting, L. Mattis Dementia Rating Scale: Internal reliability study using a diffusely impaired population. J. Clin. Neuropsychol. 3, 271–275 (1981).
pubmed: 7328179 doi: 10.1080/01688638108403130
Stacy, M. A. et al. Assessment of interrater and intrarater reliability of the Fahn-Tolosa-Marin Tremor Rating Scale in essential tremor. Mov. Disord. 22, 833–838 (2007).
pubmed: 17343274 doi: 10.1002/mds.21412
Engelhardt, J. et al. A phase 2 randomized trial of asleep versus awake subthalamic nucleus deep brain stimulation for Parkinson’s disease. Stereotact. Funct. Neurosurg. 99, 230–240 (2021).
pubmed: 33254172 doi: 10.1159/000511424
Yelnik, J. et al. A three-dimensional, histological and deformable atlas of the human basal ganglia. I. Atlas construction based on immunohistochemical and MRI data. Neuroimage 34, 618–638 (2007).
pubmed: 17110133 doi: 10.1016/j.neuroimage.2006.09.026
Stanslaski, S. et al. Design and validation of a fully implantable, chronic, closed-loop neuromodulation device with concurrent sensing and stimulation. IEEE Trans. Neural Syst. Rehabil. Eng. 20, 410–421 (2012).
pubmed: 22275720 doi: 10.1109/TNSRE.2012.2183617
Delorme, A. & Makeig, S. EEGLAB: An open source toolbox for analysis of single-trial EEG dynamics including independent component analysis. J. Neurosci. Methods 134, 9–21 (2004).
pubmed: 15102499 doi: 10.1016/j.jneumeth.2003.10.009
Prieto, T. E., Myklebust, J. B., Hoffmann, R. G., Lovett, E. G. & Myklebust, B. M. Measures of postural steadiness: Differences between healthy young and elderly adults. IEEE Trans. Biomed. Eng. 43, 956–966 (1996).
pubmed: 9214811 doi: 10.1109/10.532130
Marsden, J. F., Ashby, P., Limousin-Dowsey, P., Rothwell, J. C. & Brown, P. Coherence between cerebellar thalamus, cortex and muscle in man: Cerebellar thalamus interactions. Brain 123(Pt 7), 1459–1470 (2000).
pubmed: 10869057 doi: 10.1093/brain/123.7.1459
He, F. et al. Nonlinear interactions in the thalamocortical loop in essential tremor: A model-based frequency domain analysis. Neuroscience 324, 377–389 (2016).
pubmed: 26987955 doi: 10.1016/j.neuroscience.2016.03.028
Pedrosa, D. J. et al. Essential tremor and tremor in Parkinson’s disease are associated with distinct ‘tremor clusters’ in the ventral thalamus. Experim. Neurol. 237, 435–443 (2012).
doi: 10.1016/j.expneurol.2012.07.002
Hua, S. E. & Lenz, F. A. Posture-related oscillations in human cerebellar thalamus in essential tremor are enabled by voluntary motor circuits. J. Neurophysiol. 93, 117–127 (2005).
pubmed: 15317839 doi: 10.1152/jn.00527.2004
Milosevic, L. et al. Physiological mechanisms of thalamic ventral intermediate nucleus stimulation for tremor suppression. Brain J. Neurol. 141, 2142–2155 (2018).
doi: 10.1093/brain/awy139
Crone, N. E. et al. Functional mapping of human sensorimotor cortex with electrocorticographic spectral analysis. I. Alpha and beta event-related desynchronization. Brain 121(Pt 12), 2271–2299 (1998).
pubmed: 9874480 doi: 10.1093/brain/121.12.2271
Pfurtscheller, G. & Lopes da Silva, F. H. Event-related EEG/MEG synchronization and desynchronization: Basic principles. Clin. Neurophysiol. 110, 1842–1857 (1999).
pubmed: 10576479 doi: 10.1016/S1388-2457(99)00141-8
Kühn, A. A. et al. Event-related beta desynchronization in human subthalamic nucleus correlates with motor performance. Brain 127, 735–746 (2004).
pubmed: 14960502 doi: 10.1093/brain/awh106
Takemi, M., Masakado, Y., Liu, M. & Ushiba, J. Event-related desynchronization reflects downregulation of intracortical inhibition in human primary motor cortex. J. Neurophysiol. 110, 1158–1166 (2013).
pubmed: 23761697 doi: 10.1152/jn.01092.2012
Alegre, M. et al. Movement-related changes in oscillatory activity in the human subthalamic nucleus: Ipsilateral vs. contralateral movements. Eur. J. Neurosci. 22, 2315–2324 (2005).
pubmed: 16262669 doi: 10.1111/j.1460-9568.2005.04409.x
Basha, D. et al. Beta oscillatory neurons in the motor thalamus of movement disorder and pain patients. Exp. Neurol. 261, 782–790 (2014).
pubmed: 25205228 doi: 10.1016/j.expneurol.2014.08.024
Brücke, C. et al. Thalamic gamma oscillations correlate with reaction time in a Go/noGo task in patients with essential tremor. Neuroimage 75, 36–45 (2013).
pubmed: 23466935 doi: 10.1016/j.neuroimage.2013.02.038
Kühn, A. A. et al. Pathological synchronisation in the subthalamic nucleus of patients with Parkinson’s disease relates to both bradykinesia and rigidity. Exp. Neurol. 215, 380–387 (2009).
pubmed: 19070616 doi: 10.1016/j.expneurol.2008.11.008
Weinberger, M. et al. Beta oscillatory activity in the subthalamic nucleus and its relation to dopaminergic response in Parkinson’s disease. J. Neurophysiol. 96, 3248–3256 (2006).
pubmed: 17005611 doi: 10.1152/jn.00697.2006
Ray, N. J. et al. Local field potential beta activity in the subthalamic nucleus of patients with Parkinson’s disease is associated with improvements in bradykinesia after dopamine and deep brain stimulation. Exp. Neurol. 213, 108–113 (2008).
pubmed: 18619592 doi: 10.1016/j.expneurol.2008.05.008
Giannicola, G. et al. The effects of levodopa and ongoing deep brain stimulation on subthalamic beta oscillations in Parkinson’s disease. Exp. Neurol. 226, 120–127 (2010).
pubmed: 20713047 doi: 10.1016/j.expneurol.2010.08.011
Little, S. et al. Bilateral functional connectivity of the basal ganglia in patients with Parkinson’s disease and its modulation by dopaminergic treatment. PLoS One 8, e82762 (2013).
pubmed: 24376574 pmcid: 3869733 doi: 10.1371/journal.pone.0082762
Quinn, E. J. et al. Beta oscillations in freely moving Parkinson’s subjects are attenuated during deep brain stimulation. Mov. Disord. 30, 1750–1758 (2015).
pubmed: 26360123 doi: 10.1002/mds.26376
Kühn, A. A. et al. High-frequency stimulation of the subthalamic nucleus suppresses oscillatory beta activity in patients with Parkinson’s disease in parallel with improvement in motor performance. J. Neurosci. 28, 6165–6173 (2008).
pubmed: 18550758 pmcid: 6670522 doi: 10.1523/JNEUROSCI.0282-08.2008
Bronte-Stewart, H. et al. The STN beta-band profile in Parkinson’s disease is stationary and shows prolonged attenuation after deep brain stimulation. Exp. Neurol. 215, 20–28 (2009).
pubmed: 18929561 doi: 10.1016/j.expneurol.2008.09.008
Wingeier, B. et al. Intra-operative STN DBS attenuates the prominent beta rhythm in the STN in Parkinson’s disease. Exp. Neurol. 197, 244–251 (2006).
pubmed: 16289053 doi: 10.1016/j.expneurol.2005.09.016
Ferleger, B. I. et al. Fully implanted adaptive deep brain stimulation in freely moving essential tremor patients. J. Neural Eng. 17, 056026 (2020).
pubmed: 33055369 doi: 10.1088/1741-2552/abb416
He, S. et al. Closed-loop DBS triggered by real-time movement and tremor decoding based on thalamic LFPs for essential tremor. In 2020 42nd Annual International Conference of the IEEE Engineering in Medicine & Biology Society (EMBC) (eds He, S. et al.) 3602–3605 (IEEE, 2020). https://doi.org/10.1109/EMBC44109.2020.9175433 .
doi: 10.1109/EMBC44109.2020.9175433
He, S. et al. Closed-loop deep brain stimulation for essential tremor based on thalamic local field potentials. Mov. Disord. 36, 863–873 (2021).
pubmed: 33547859 pmcid: 7610625 doi: 10.1002/mds.28513
Buijink, A. W. G. et al. Thalamic local field potentials recorded using the deep brain stimulation pulse generator. Clin. Neurophysiol. Pract. 7, 103–106 (2022).
pubmed: 35345863 pmcid: 8956842 doi: 10.1016/j.cnp.2022.03.002

Auteurs

Dominique Guehl (D)

Service de Neurophysiologie Clinique de l'enfant et de l'adulte, Hôpital Pellegrin, Pôle des Neurosciences Cliniques, CHU de Bordeaux, Bordeaux, France. dominique.guehl@chu-bordeaux.fr.
Institut des Maladies Neurodégénératives, Univ. Bordeaux, CNRS, IMN, UMR 5293, F-33000, Bordeaux, France. dominique.guehl@chu-bordeaux.fr.

Etienne Guillaud (E)

Institute of Cognitive and Integrative Neurosciences, Univ. Bordeaux, CNRS, INCIA, UMR 5287, F-33000, Bordeaux, France.

Nicolas Langbour (N)

Centre de Recherche en Psychiatrie, CH de la Milétrie, 86000, Poitiers, France.

Emilie Doat (E)

Institute of Cognitive and Integrative Neurosciences, Univ. Bordeaux, CNRS, INCIA, UMR 5287, F-33000, Bordeaux, France.

Nicolas Auzou (N)

Institut des Maladies Neurodégénératives Clinique (IMNc), Pôle des Neurosciences Cliniques, CHU de Bordeaux, Bordeaux, France.

Edouard Courtin (E)

Service de Neurophysiologie Clinique de l'enfant et de l'adulte, Hôpital Pellegrin, Pôle des Neurosciences Cliniques, CHU de Bordeaux, Bordeaux, France.

Olivier Branchard (O)

Service de Neurochirurgie, CHU de Bordeaux, Bordeaux, France.

Julien Engelhardt (J)

Service de Neurochirurgie, CHU de Bordeaux, Bordeaux, France.

Abdelhamid Benazzouz (A)

Institut des Maladies Neurodégénératives, Univ. Bordeaux, CNRS, IMN, UMR 5293, F-33000, Bordeaux, France.

Alexandre Eusebio (A)

Department of Neurology and Movement Disorders, APHM, Hôpitaux Universitaire de Marseille, Marseille, France.
Institut de Neurosciences de la Timone, UMR 7289, Aix Marseille Univ, CNRS, Marseille, France.

Emmanuel Cuny (E)

Service de Neurochirurgie, CHU de Bordeaux, Bordeaux, France.

Pierre Burbaud (P)

Service de Neurophysiologie Clinique de l'enfant et de l'adulte, Hôpital Pellegrin, Pôle des Neurosciences Cliniques, CHU de Bordeaux, Bordeaux, France.
Institut des Maladies Neurodégénératives, Univ. Bordeaux, CNRS, IMN, UMR 5293, F-33000, Bordeaux, France.

Classifications MeSH