Genomic and epigenomic integrative subtypes of renal cell carcinoma in a Japanese cohort.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
16 Dec 2023
Historique:
received: 06 06 2023
accepted: 01 12 2023
medline: 17 12 2023
pubmed: 17 12 2023
entrez: 16 12 2023
Statut: epublish

Résumé

Renal cell carcinoma (RCC) comprises several histological types characterised by different genomic and epigenomic aberrations; however, the molecular pathogenesis of each type still requires further exploration. We perform whole-genome sequencing of 128 Japanese RCC cases of different histology to elucidate the significant somatic alterations and mutagenesis processes. We also perform transcriptomic and epigenomic sequencing to identify distinguishing features, including assay for transposase-accessible chromatin sequencing (ATAC-seq) and methyl sequencing. Genomic analysis reveals that the mutational signature differs among the histological types, suggesting that different carcinogenic factors drive each histology. From the ATAC-seq results, master transcription factors are identified for each histology. Furthermore, clear cell RCC is classified into three epi-subtypes, one of which expresses highly immune checkpoint molecules with frequent loss of chromosome 14q. These genomic and epigenomic features may lead to the development of effective therapeutic strategies for RCC.

Identifiants

pubmed: 38104198
doi: 10.1038/s41467-023-44159-1
pii: 10.1038/s41467-023-44159-1
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

8383

Subventions

Organisme : Japan Agency for Medical Research and Development (AMED)
ID : 21ck0106547h000

Informations de copyright

© 2023. The Author(s).

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Auteurs

Akihiko Fukagawa (A)

Division of Cancer Genomics, National Cancer Center Research Institute, Tokyo, Japan.
Department of Pathology, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan.

Natsuko Hama (N)

Division of Cancer Genomics, National Cancer Center Research Institute, Tokyo, Japan.

Yasushi Totoki (Y)

Division of Cancer Genomics, National Cancer Center Research Institute, Tokyo, Japan.
Department of Cancer Genome Informatics, Graduate School of Medicine, Osaka University, Osaka, Japan.

Hiromi Nakamura (H)

Division of Cancer Genomics, National Cancer Center Research Institute, Tokyo, Japan.

Yasuhito Arai (Y)

Division of Cancer Genomics, National Cancer Center Research Institute, Tokyo, Japan.

Mihoko Saito-Adachi (M)

Division of Cancer Genomics, National Cancer Center Research Institute, Tokyo, Japan.

Akiko Maeshima (A)

Department of Diagnostic Pathology, National Cancer Center Hospital, Tokyo, Japan.

Yoshiyuki Matsui (Y)

Department of Urology, National Cancer Center Hospital, Tokyo, Japan.

Shinichi Yachida (S)

Department of Cancer Genome Informatics, Graduate School of Medicine, Osaka University, Osaka, Japan.

Tetsuo Ushiku (T)

Department of Pathology, Graduate School of Medicine, The University of Tokyo, Tokyo, Japan.

Tatsuhiro Shibata (T)

Division of Cancer Genomics, National Cancer Center Research Institute, Tokyo, Japan. tshibata2010@gmail.com.
Laboratory of Molecular Medicine, The Institute of Medical Science, The University of Tokyo, Tokyo, Japan. tshibata2010@gmail.com.

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