Statin therapy and outcome in Takotsubo syndrome patients: Results from the multicenter international GEIST registry.

Endothelial dysfunction Outcome Prognosis Statin Stress cardiomyopathy Takotsubo syndrome

Journal

Atherosclerosis
ISSN: 1879-1484
Titre abrégé: Atherosclerosis
Pays: Ireland
ID NLM: 0242543

Informations de publication

Date de publication:
13 Dec 2023
Historique:
received: 04 10 2023
revised: 28 11 2023
accepted: 07 12 2023
medline: 22 12 2023
pubmed: 22 12 2023
entrez: 22 12 2023
Statut: aheadofprint

Résumé

Several studies have shown that endothelial dysfunction plays a role in the pathogenesis of Takotsubo syndrome (TTS). Given the potential benefit of statin therapy on endothelial dysfunction, we hypothesized that such treatment could improve outcome. Aim of our study was to evaluate clinical characteristics and outcome of TTS patients treated with statin therapy. Patients were enrolled in the international multicenter GEIST (GErman Italian Spanish Takotsubo) registry. Demographic data, clinical features and drug therapy at discharge were recorded. Primary study outcome was the occurrence of all-cause death at follow-up. Study population included 2429 consecutive TTS patients: 1293 (53.2%) discharged on statin and 1136 (46.8%) without statin. Patients with statin were older (age 72 ± 11 vs 69 ± 13 years, p < 0.001), with higher prevalence of hypertension (74.3% vs 60.3%, p < 0.001), diabetes (21.1% vs 14.7%, p < 0.001), dyslipidemia (56.1% vs 23.3%, p < 0.001), history of coronary artery disease (13.3% vs 6.3%, p < 0.001) and lower rates of in-hospital complications (14.7% vs 19.3%, p = 0.003). Survival analysis showed similar mortality rates between groups (log rank p = 0.803). At univariable analysis, statin therapy at discharge was not associated with lower mortality (HR: 0.97, 95% CI 0.74-1.26, p = 0.803). At multivariable analysis age (HR: 1.06 95% CI 1.04-1.08, p < 0.001), male sex (HR: 1.83, 95% CI 1.20-2.80, p = 0.005), diabetes (HR: 2.55, 95% CI 1.83-3.54 p < 0.001), malignancies (HR: 2.41, 95% CI 1.68-3.44, p < 0.001) and physical trigger (HR: 2.24, 95% CI 1.62-3.10, p < 0.001) were associated with increased mortality. Statin therapy after a TTS event was not associated with better prognosis at follow-up.

Sections du résumé

BACKGROUND AND AIMS OBJECTIVE
Several studies have shown that endothelial dysfunction plays a role in the pathogenesis of Takotsubo syndrome (TTS). Given the potential benefit of statin therapy on endothelial dysfunction, we hypothesized that such treatment could improve outcome. Aim of our study was to evaluate clinical characteristics and outcome of TTS patients treated with statin therapy.
METHODS METHODS
Patients were enrolled in the international multicenter GEIST (GErman Italian Spanish Takotsubo) registry. Demographic data, clinical features and drug therapy at discharge were recorded. Primary study outcome was the occurrence of all-cause death at follow-up.
RESULTS RESULTS
Study population included 2429 consecutive TTS patients: 1293 (53.2%) discharged on statin and 1136 (46.8%) without statin. Patients with statin were older (age 72 ± 11 vs 69 ± 13 years, p < 0.001), with higher prevalence of hypertension (74.3% vs 60.3%, p < 0.001), diabetes (21.1% vs 14.7%, p < 0.001), dyslipidemia (56.1% vs 23.3%, p < 0.001), history of coronary artery disease (13.3% vs 6.3%, p < 0.001) and lower rates of in-hospital complications (14.7% vs 19.3%, p = 0.003). Survival analysis showed similar mortality rates between groups (log rank p = 0.803). At univariable analysis, statin therapy at discharge was not associated with lower mortality (HR: 0.97, 95% CI 0.74-1.26, p = 0.803). At multivariable analysis age (HR: 1.06 95% CI 1.04-1.08, p < 0.001), male sex (HR: 1.83, 95% CI 1.20-2.80, p = 0.005), diabetes (HR: 2.55, 95% CI 1.83-3.54 p < 0.001), malignancies (HR: 2.41, 95% CI 1.68-3.44, p < 0.001) and physical trigger (HR: 2.24, 95% CI 1.62-3.10, p < 0.001) were associated with increased mortality.
CONCLUSIONS CONCLUSIONS
Statin therapy after a TTS event was not associated with better prognosis at follow-up.

Identifiants

pubmed: 38134646
pii: S0021-9150(23)05342-X
doi: 10.1016/j.atherosclerosis.2023.117421
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

117421

Informations de copyright

Copyright © 2023 The Authors. Published by Elsevier B.V. All rights reserved.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

Auteurs

Giuseppina Novo (G)

Department of Health Promotion, Mother and Child Care, Internal Medicine and Medical Specialties (ProMISE), University of Palermo, Italy. Electronic address: giuseppina.novo@unipa.it.

Luca Arcari (L)

Institute of Cardiology, Madre Giuseppina Vannini Hospital, Rome, Italy, Department of Cardiology; Department of Clinical Internal, Anesthesiological and Cardiovascular Sciences, Sapienza University, Rome, Italy.

Thomas Stiermaier (T)

University Heart Center Lübeck, Medical Clinic II (Cardiology/Angiology/Intensive Care Medicine) and German Center for Cardiovascular Research (DZHK), Partner Site Hamburg/Kiel/Lübeck, Lübeck, Germany.

Chiara Alaimo (C)

Department of Health Promotion, Mother and Child Care, Internal Medicine and Medical Specialties (ProMISE), University of Palermo, Italy.

Ibrahim El-Battrawy (I)

Department of Cardiology and Angiology, Bergmannsheil University Hospitals, Ruhr University of Bochum, Bochum, Germany; First Department of Medicine, Faculty of Medicine, University Medical Centre Mannheim (UMM), University of Heidelberg, Mannheim, Germany; German Center for Cardiovascular Research, Partner Site, Heidelberg-Mannheim, Mannheim, Germany.

Luca Cacciotti (L)

Institute of Cardiology, Madre Giuseppina Vannini Hospital, Rome, Italy, Department of Cardiology.

Federico Guerra (F)

Cardiology and Arrhythmology Clinic, Marche Polytechnic University, University Hospital "Umberto I - Lancisi - Salesi", Ancona, Italy.

Beatrice Musumeci (B)

Cardiology Department, Clinical and Molecular Medicine Department, Faculty of Medicine and Psychology, Sapienza University of Rome, Rome, Italy.

Enrica Mariano (E)

University of Rome Tor Vergata, Division of Cardiology, Rome, Italy.

Giuseppe Parisi (G)

Department of Cardiology, San Paolo Hospital, Bari, Italy.

Roberta Montisci (R)

Clinical Cardiology, Department of Medical Science and Public Health, University of Cagliari, Cagliari, Italy.

Ravi Vazirani (R)

Cardiovascular Institute. Hospital Clínico Universitario San Carlos, Madrid, Spain.

Alberto Perez Castellanos (A)

Cardiology Department, Health Research Institute of the Balearic Islands (IdISBa), Hospital Universitari Son Espases, Palma, Spain.

Aitor Uribarri (A)

Cardiology Department, Hospital Universitari Vall d'Hebron, Barcelona, Spain.

Miguel Corbi-Pascual (M)

Cardiology Department, Hospital Universitario de Albacete. Albacete, Spain.

Jorge Salamanca (J)

Department of Cardiology, Hospital Universitario de la Princesa. Instituto de Investigación Sanitaria Princesa (IIS-IP), Madrid, Spain.

Ibrahim Akin (I)

First Department of Medicine, Faculty of Medicine, University Medical Centre Mannheim (UMM), University of Heidelberg, Mannheim, Germany.

Holger Thiele (H)

Heart Center Leipzig at University of Leipzig, Department of Internal Medicine/Cardiology and Leipzig Heart Institute, Leipzig, Germany.

Natale Daniele Brunetti (ND)

University of Foggia, Department of Medical and Surgical Sciences, Foggia, Italy.

Ingo Eitel (I)

University Heart Center Lübeck, Medical Clinic II (Cardiology/Angiology/Intensive Care Medicine) and German Center for Cardiovascular Research (DZHK), Partner Site Hamburg/Kiel/Lübeck, Lübeck, Germany.

Iván J Núñez Gil (IJ)

Cardiovascular Institute. Hospital Clínico Universitario San Carlos, Madrid, Spain.

Francesco Santoro (F)

University of Foggia, Department of Medical and Surgical Sciences, Foggia, Italy.

Classifications MeSH