Modulation of Epstein-Barr-Virus (EBV)-Associated Cancers by Co-Infections.
Burkitt’s lymphoma
Epstein Barr virus (EBV)
Helicobacter pylori
Kaposi-sarcoma-associated herpesvirus (KSHV)
Plasmodium falciparum
human cytomegalovirus (HCMV)
human immunodeficiency virus (HIV)
human papilloma virus (HPV)
malaria
primary effusion lymphoma (PEL)
Journal
Cancers
ISSN: 2072-6694
Titre abrégé: Cancers (Basel)
Pays: Switzerland
ID NLM: 101526829
Informations de publication
Date de publication:
07 Dec 2023
07 Dec 2023
Historique:
received:
16
11
2023
revised:
04
12
2023
accepted:
05
12
2023
medline:
23
12
2023
pubmed:
23
12
2023
entrez:
23
12
2023
Statut:
epublish
Résumé
The oncogenic and persistent Epstein Barr virus (EBV) is carried by more than 95% of the human adult population. While asymptomatic in most of these, EBV can cause a wide variety of malignancies of lymphoid or epithelial cell origin. Some of these are also associated with co-infections that either increase EBV-induced tumorigenesis or weaken its immune control. The respective pathogens include Kaposi-sarcoma-associated herpesvirus (KSHV),
Identifiants
pubmed: 38136285
pii: cancers15245739
doi: 10.3390/cancers15245739
pii:
doi:
Types de publication
Journal Article
Review
Langues
eng
Subventions
Organisme : Swiss National Science Foundation
ID : 310030_204470/1
Pays : Switzerland
Organisme : Swiss National Science Foundation
ID : 310030L_197952/1
Pays : Switzerland