Testosterone Recovery for Relugolix Versus Leuprolide in Men with Advanced Prostate Cancer: Results from the Phase 3 HERO Study.

Advanced prostate cancer Leuprolide Prostate-specific antigen Relugolix Testosterone recovery

Journal

European urology oncology
ISSN: 2588-9311
Titre abrégé: Eur Urol Oncol
Pays: Netherlands
ID NLM: 101724904

Informations de publication

Date de publication:
23 Dec 2023
Historique:
received: 19 04 2023
revised: 17 11 2023
accepted: 27 11 2023
medline: 25 12 2023
pubmed: 25 12 2023
entrez: 24 12 2023
Statut: aheadofprint

Résumé

In the HERO study, relugolix demonstrated sustained testosterone suppression superior to that of leuprolide acetate (97% vs 89%; difference 7.9% [95% confidence interval, 4.1-12%; p < 0.001]). To analyze testosterone recovery in a prespecified subset of men from the HERO study not indicated to continue androgen deprivation therapy. Men (N = 934) were randomized (2:1) to receive relugolix 120 mg orally daily or leuprolide acetate injections every 12 wk for 48 wk. Testosterone recovery was assessed in 184 men who completed 48 wk of treatment. During the 90-d recovery period, assessments included time to testosterone recovery (>280 ng/dl; ≥80% of baseline testosterone), serum levels of prostate-specific antigen and pituitary hormones, and adverse events. The cumulative incidence rate of testosterone recovery to >280 ng/dl at 90 d following drug discontinuation was significantly higher in the relugolix cohort (n = 137) than in the leuprolide acetate cohort (n = 47; 54% vs 3.2%; nominal p = 0.002). The median time to testosterone recovery was faster following relugolix treatment than with leuprolide acetate treatment (86.0 d vs 112.0 d). Compared with leuprolide acetate, more men treated with relugolix achieved ≥80% of baseline testosterone levels (39% vs 2.1%). Men ≤65 yr and those with baseline testosterone greater than the median had a higher incident rate of testosterone recovery. Adverse events were generally similar between treatment groups. One limitation is the short testosterone recovery follow-up period. Oral relugolix had faster and more complete recovery of testosterone to normal levels after treatment discontinuation than leuprolide acetate in a subset of men from the HERO study. The clinical implications of a faster testosterone recovery with relugolix may be significant for men being treated with androgen deprivation therapy and influence treatment decisions. The male hormone testosterone is reduced during androgen deprivation therapy for prostate cancer. Reduced testosterone levels cause side effects, impacting patient quality of life. When treatment is stopped, the side effects lessen over time as the levels of testosterone come back to pretreatment range (testosterone recovery). In this study, we found that the time to testosterone recovery was faster with relugolix than with leuprolide acetate.

Sections du résumé

BACKGROUND BACKGROUND
In the HERO study, relugolix demonstrated sustained testosterone suppression superior to that of leuprolide acetate (97% vs 89%; difference 7.9% [95% confidence interval, 4.1-12%; p < 0.001]).
OBJECTIVE OBJECTIVE
To analyze testosterone recovery in a prespecified subset of men from the HERO study not indicated to continue androgen deprivation therapy.
DESIGN, SETTING, AND PARTICIPANTS METHODS
Men (N = 934) were randomized (2:1) to receive relugolix 120 mg orally daily or leuprolide acetate injections every 12 wk for 48 wk.
OUTCOME MEASUREMENTS AND STATISTICAL ANALYSIS METHODS
Testosterone recovery was assessed in 184 men who completed 48 wk of treatment. During the 90-d recovery period, assessments included time to testosterone recovery (>280 ng/dl; ≥80% of baseline testosterone), serum levels of prostate-specific antigen and pituitary hormones, and adverse events.
RESULTS AND LIMITATIONS CONCLUSIONS
The cumulative incidence rate of testosterone recovery to >280 ng/dl at 90 d following drug discontinuation was significantly higher in the relugolix cohort (n = 137) than in the leuprolide acetate cohort (n = 47; 54% vs 3.2%; nominal p = 0.002). The median time to testosterone recovery was faster following relugolix treatment than with leuprolide acetate treatment (86.0 d vs 112.0 d). Compared with leuprolide acetate, more men treated with relugolix achieved ≥80% of baseline testosterone levels (39% vs 2.1%). Men ≤65 yr and those with baseline testosterone greater than the median had a higher incident rate of testosterone recovery. Adverse events were generally similar between treatment groups. One limitation is the short testosterone recovery follow-up period.
CONCLUSIONS CONCLUSIONS
Oral relugolix had faster and more complete recovery of testosterone to normal levels after treatment discontinuation than leuprolide acetate in a subset of men from the HERO study. The clinical implications of a faster testosterone recovery with relugolix may be significant for men being treated with androgen deprivation therapy and influence treatment decisions.
PATIENT SUMMARY RESULTS
The male hormone testosterone is reduced during androgen deprivation therapy for prostate cancer. Reduced testosterone levels cause side effects, impacting patient quality of life. When treatment is stopped, the side effects lessen over time as the levels of testosterone come back to pretreatment range (testosterone recovery). In this study, we found that the time to testosterone recovery was faster with relugolix than with leuprolide acetate.

Identifiants

pubmed: 38143206
pii: S2588-9311(23)00290-0
doi: 10.1016/j.euo.2023.11.024
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2023 European Association of Urology. Published by Elsevier B.V. All rights reserved.

Auteurs

Ronald Tutrone (R)

Chesapeake Urology, Towson, MD, USA. Electronic address: rtutrone@uniteduro.com.

Fred Saad (F)

University of Montreal Hospital Centre, Montreal, QC, Canada.

Daniel J George (DJ)

Duke Cancer Institute Center for Prostate and Urologic Cancers, Duke University, Durham, NC, USA.

Bertrand Tombal (B)

Institut de Recherche Clinique, Université Catholique de Louvain, Brussels, Belgium.

James L Bailen (JL)

First Urology, Jeffersonville, IN, USA.

Michael S Cookson (MS)

Department of Urology, The University of Oklahoma Health Sciences Center, Oklahoma City, OK, USA.

Daniel R Saltzstein (DR)

Urology San Antonio, San Antonio, TX, USA.

Sarah Hanson (S)

Pfizer, Inc., New York, NY, USA.

Bruce Brown (B)

Myovant Sciences, Inc., Brisbane, CA, USA.

Sophia Lu (S)

Myovant Sciences, Inc., Brisbane, CA, USA.

Mark Fallick (M)

Myovant Sciences, Inc., Brisbane, CA, USA.

Neal D Shore (ND)

Carolina Urologic Research Center and GenesisCare USA, Myrtle Beach, SC, USA.

Classifications MeSH