Effect of the Factor XIa Inhibitor Asundexian According to Baseline Infarct Pattern and on MRI Covert Infarct Outcomes.

anticoagulants atherosclerosis cerebral infarction humans ischemic stroke

Journal

Stroke
ISSN: 1524-4628
Titre abrégé: Stroke
Pays: United States
ID NLM: 0235266

Informations de publication

Date de publication:
04 Jan 2024
Historique:
medline: 4 1 2024
pubmed: 4 1 2024
entrez: 4 1 2024
Statut: aheadofprint

Résumé

Exploratory analysis of the phase 2 PACIFIC-Stroke (Program of Anticoagulation via Inhibition of FXIa by the Oral Compound BAY 2433334-Non-Cardioembolic Stroke) randomized trial suggested that asundexian, an oral factor XIa inhibitor, prevents recurrent stroke and transient ischemic attacks in patients with atherosclerotic stroke. In this post hoc exploratory analysis, we hypothesized that asundexian would be more effective in patients enrolled with large, multiple, or cortical acute infarcts on magnetic resonance imaging than in patients enrolled with a single small subcortical acute infarct, and asundexian would prevent incident cortical covert infarcts. In this placebo-controlled double-blinded randomized controlled trial, patients with mild-to-moderate noncardioembolic ischemic stroke were assigned to asundexian (10, 20, or 50 mg once daily) or placebo, in addition to antiplatelet therapy. Brain magnetic resonance imagings were required within 72 hours of randomization and repeated at 26 weeks or at discontinuation of the study drug. Of 1808 randomized patients, 1780 (98.5%) had interpretable baseline magnetic resonance imagings, of which 1628 (91.5%) had ≥1 diffusion-weighted imaging positive acute infarcts. Magnetic resonance imaging follow-up was obtained in 1439 patients, of whom 1358 had no symptomatic stroke during the trial period. Compared with placebo, asundexian 50 mg daily conferred a trend toward reduced risk of recurrent ischemic stroke or incident covert infarcts (hazard ratio, 0.71 [95% CI, 0.45-1.11]) and recurrent ischemic stroke or transient ischemic attack (secondary outcome; hazard ratio, 0.59 [95% CI, 0.33-1.06]) that was not evident in patients with single small subcortical infarcts (hazard ratios, 1.14 [95% CI, 0.62-2.10] and 0.93 [95% CI, 0.28-3.06]). Incident cortical covert infarcts were reduced in patients taking asundexian 50 mg, but the difference was not statistically significant (crude incidence ratio, 0.56 [95% CI, 0.28-1.12]). These exploratory, unconfirmed results suggest that asundexian may prevent new embolic infarcts but not small artery occlusion. The hypothesis that subtypes of covert brain infarcts respond differently to anticoagulant prevention should be tested in future trials. URL: https://clinicaltrials.gov; Unique identifier: NCT04304508.

Sections du résumé

BACKGROUND UNASSIGNED
Exploratory analysis of the phase 2 PACIFIC-Stroke (Program of Anticoagulation via Inhibition of FXIa by the Oral Compound BAY 2433334-Non-Cardioembolic Stroke) randomized trial suggested that asundexian, an oral factor XIa inhibitor, prevents recurrent stroke and transient ischemic attacks in patients with atherosclerotic stroke. In this post hoc exploratory analysis, we hypothesized that asundexian would be more effective in patients enrolled with large, multiple, or cortical acute infarcts on magnetic resonance imaging than in patients enrolled with a single small subcortical acute infarct, and asundexian would prevent incident cortical covert infarcts.
METHODS UNASSIGNED
In this placebo-controlled double-blinded randomized controlled trial, patients with mild-to-moderate noncardioembolic ischemic stroke were assigned to asundexian (10, 20, or 50 mg once daily) or placebo, in addition to antiplatelet therapy. Brain magnetic resonance imagings were required within 72 hours of randomization and repeated at 26 weeks or at discontinuation of the study drug.
RESULTS UNASSIGNED
Of 1808 randomized patients, 1780 (98.5%) had interpretable baseline magnetic resonance imagings, of which 1628 (91.5%) had ≥1 diffusion-weighted imaging positive acute infarcts. Magnetic resonance imaging follow-up was obtained in 1439 patients, of whom 1358 had no symptomatic stroke during the trial period. Compared with placebo, asundexian 50 mg daily conferred a trend toward reduced risk of recurrent ischemic stroke or incident covert infarcts (hazard ratio, 0.71 [95% CI, 0.45-1.11]) and recurrent ischemic stroke or transient ischemic attack (secondary outcome; hazard ratio, 0.59 [95% CI, 0.33-1.06]) that was not evident in patients with single small subcortical infarcts (hazard ratios, 1.14 [95% CI, 0.62-2.10] and 0.93 [95% CI, 0.28-3.06]). Incident cortical covert infarcts were reduced in patients taking asundexian 50 mg, but the difference was not statistically significant (crude incidence ratio, 0.56 [95% CI, 0.28-1.12]).
CONCLUSIONS UNASSIGNED
These exploratory, unconfirmed results suggest that asundexian may prevent new embolic infarcts but not small artery occlusion. The hypothesis that subtypes of covert brain infarcts respond differently to anticoagulant prevention should be tested in future trials.
REGISTRATION UNASSIGNED
URL: https://clinicaltrials.gov; Unique identifier: NCT04304508.

Identifiants

pubmed: 38174569
doi: 10.1161/STROKEAHA.123.043198
doi:

Banques de données

ClinicalTrials.gov
['NCT04304508']

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Investigateurs

P Bailey (P)
T Kleinig (T)
D Cordato (D)
P Choi (P)
C Garcia-Esperon (C)
A Chew (A)
G Cloud (G)
V Stanislaus (V)
M Krause (M)
M Priglinger (M)
R Grimley (R)
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Auteurs

Eric E Smith (EE)

Department of Clinical Neurosciences, University of Calgary, AB, Canada (E.E.S., S.F.).

Ashkan Shoamanesh (A)

Department of Medicine (Neurology), Population Health Research Institute, McMaster University, Hamilton, ON, Canada (A.S., X.L., T.H., S.J.C., R.G.H.).

Lizhen Xu (L)

Department of Medicine (Neurology), Population Health Research Institute, McMaster University, Hamilton, ON, Canada (A.S., X.L., T.H., S.J.C., R.G.H.).

Laura Heenan (L)

Department of Statistics, Population Health Research Institute, Hamilton, ON, Canada (L.H.).

Feryal Saad (F)

Department of Clinical Neurosciences, University of Calgary, AB, Canada (E.E.S., S.F.).

Pablo Colorado (P)

Bayer US LLC Pharmaceuticals, Hanover, NJ (P.C.).

Chih-Hao Chen (CH)

Department of Neurology, National Taiwan University Hospital, Taipei (C.-H.C.).

Robin Lemmens (R)

Department of Neurosciences, Experimental Neurology, KU Leuven-University of Leuven, Belgium (R.L.).
Department of Neurology, University Hospitals Leuven, Belgium (R.L.).

Gian Marco De Marchis (GM)

Department of Neurology and Stroke Center, University Hospital Basel and University of Basel, Switzerland (G.M.D.M.).
Neurology Clinic and Stroke Center, Kantonsspital St. Gallen, Switzerland (G.M.D.M.).

Valeria Caso (V)

Department of Vascular and Emergency, Stroke Unit, Santa Maria de Misericordia Hospital, University of Perugia, Italy (V.C.).

Jaime Masjuan (J)

Stroke Unit, Department of Neurology, Instituto Ramón y Cajal de Investigación Sanitaria, Hospital Universitario Ramón y Cajal, Madrid, Spain (J.M.).
Department of Medicine, Universidad de Alcalá, Madrid, Spain (J.M.).

Teruyuki Hirano (T)

Department of Medicine (Neurology), Population Health Research Institute, McMaster University, Hamilton, ON, Canada (A.S., X.L., T.H., S.J.C., R.G.H.).
Department of Stroke and Cerebrovascular Medicine, School of Medicine, Kyorin University, Tokyo, Japan (T.H.).

Ivan Milanov (I)

Department of Neurology, Medical University, University Hospital for Neurology and Psychiatry Sveti Naum, Sofia, Bulgaria (I.M.).

Bruce C V Campbell (BCV)

Department of Medicine and Neurology, Melbourne Brain Centre at the Royal Melbourne Hospital, University of Melbourne, Parkville, VIC, Australia (B.C.V.C.).

Jean-Louis Mas (JL)

Department of Neurology, Groupe Hospitalo-Universitaire Paris, Hôpital Sainte-Anne, Université Paris-Cité, Inserm U1266, France (J.-L.M.).

Stuart J Connolly (SJ)

Department of Medicine (Neurology), Population Health Research Institute, McMaster University, Hamilton, ON, Canada (A.S., X.L., T.H., S.J.C., R.G.H.).

Hardi Mundl (H)

Bayer AG, Wuppertal, Germany (H.M.).

Robert G Hart (RG)

Department of Medicine (Neurology), Population Health Research Institute, McMaster University, Hamilton, ON, Canada (A.S., X.L., T.H., S.J.C., R.G.H.).

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