CT109-SN-38, a Novel Antibody-drug Conjugate with Dual Specificity for CEACAM5 and 6, Elicits Potent Killing of Pancreatic Cancer Cells.

ADC CEACAM5 CEACAM6 and SN-3 antibody-drug conjugate pancreatic ductal adenocarcinoma

Journal

Current cancer drug targets
ISSN: 1873-5576
Titre abrégé: Curr Cancer Drug Targets
Pays: Netherlands
ID NLM: 101094211

Informations de publication

Date de publication:
04 Jan 2024
Historique:
received: 06 07 2023
revised: 18 08 2023
accepted: 03 10 2023
medline: 5 1 2024
pubmed: 5 1 2024
entrez: 5 1 2024
Statut: aheadofprint

Résumé

CEACAM5 and CEACAM6 are glycosylphosphatidylinositol (GPI)- linked members of the carcinoembryonic antigen-related cell adhesion molecule (CEACAM) family, which are frequently upregulated in epithelial cancers where they contribute to invasion, metastasis, immune evasion, and resistance to anoikis. CT109 is a novel antibody with dual specificity to both CEACAM5 and 6. In this study, we aimed to perform the preclinical characterization of CT109 and antibody- drug conjugate (ADCs) derivatives of CT109, focusing on CT109-SN-38. CT109's cognate epitope was characterized by scanning mutagenesis. CT109 specificity and internalization kinetics were assessed by immunoblot and flow cytometry, respectively. Cognate antigen expression prevalence in colorectal cancer and normal tissue arrays was determined by immunohistochemistry. CT109 conjugations were generated by the reaction of reduced CT109 cysteines with maleimide-functionalized payload linkers. In vitro cytotoxic activity of CT109 ADCs was characterized on antigen-positive and negative pancreatic ductal adenocarcinoma cell (PDAC) lines using a luminometric viability assay. In vivo efficacy of CT109-SN-38 was assessed on a PDAC tumor xenograft model at 10 and 25 mg/kg concentrations. CT109 showed to bind a glycoepitope centered on N309. CT109 is internalized in the CEACAM5+/CEACAM6+ double-positive PDAC line, BxPC-3, with a t1/2 of 2.3 hours. CT109 ADCs elicit a dose and antigen-dependent cytotoxic effect, with CT109-SN-38 exhibiting an IC50 value of 21 nM in BxPC-3 cells. In a BxPC-3 tumor xenograft model, CT109-SN-38 reduced tumor growth and induced regression in 3/10 mice at 25 mg/kg concentration. These data suggest that further preclinical and clinical development of CT109-SN-38 is warranted.

Sections du résumé

BACKGROUND BACKGROUND
CEACAM5 and CEACAM6 are glycosylphosphatidylinositol (GPI)- linked members of the carcinoembryonic antigen-related cell adhesion molecule (CEACAM) family, which are frequently upregulated in epithelial cancers where they contribute to invasion, metastasis, immune evasion, and resistance to anoikis. CT109 is a novel antibody with dual specificity to both CEACAM5 and 6.
OBJECTIVE OBJECTIVE
In this study, we aimed to perform the preclinical characterization of CT109 and antibody- drug conjugate (ADCs) derivatives of CT109, focusing on CT109-SN-38.
METHODS METHODS
CT109's cognate epitope was characterized by scanning mutagenesis. CT109 specificity and internalization kinetics were assessed by immunoblot and flow cytometry, respectively. Cognate antigen expression prevalence in colorectal cancer and normal tissue arrays was determined by immunohistochemistry. CT109 conjugations were generated by the reaction of reduced CT109 cysteines with maleimide-functionalized payload linkers. In vitro cytotoxic activity of CT109 ADCs was characterized on antigen-positive and negative pancreatic ductal adenocarcinoma cell (PDAC) lines using a luminometric viability assay. In vivo efficacy of CT109-SN-38 was assessed on a PDAC tumor xenograft model at 10 and 25 mg/kg concentrations.
RESULTS RESULTS
CT109 showed to bind a glycoepitope centered on N309. CT109 is internalized in the CEACAM5+/CEACAM6+ double-positive PDAC line, BxPC-3, with a t1/2 of 2.3 hours. CT109 ADCs elicit a dose and antigen-dependent cytotoxic effect, with CT109-SN-38 exhibiting an IC50 value of 21 nM in BxPC-3 cells. In a BxPC-3 tumor xenograft model, CT109-SN-38 reduced tumor growth and induced regression in 3/10 mice at 25 mg/kg concentration.
CONCLUSIONS CONCLUSIONS
These data suggest that further preclinical and clinical development of CT109-SN-38 is warranted.

Identifiants

pubmed: 38178674
pii: CCDT-EPUB-136984
doi: 10.2174/0115680096260614231115192343
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright© Bentham Science Publishers; For any queries, please email at epub@benthamscience.net.

Auteurs

Kelly C Arias Cardenas (KC)

Stromatis Pharma, 9501 Discovery Blvd Manassas, VA 20109, USA.

Clinton W Enos (CW)

Eastern Virginia Medical School, USA.

Mark R Spear (MR)

Stromatis Pharma, 9501 Discovery Blvd Manassas, VA 20109, USA.

Dana E Austin (DE)

Stromatis Pharma, 9501 Discovery Blvd Manassas, VA 20109, USA.

Raghad Almofeez (R)

Stromatis Pharma, 9501 Discovery Blvd Manassas, VA 20109, USA.

Stephanie Kortchak (S)

Stromatis Pharma, 9501 Discovery Blvd Manassas, VA 20109, USA.

Lauren Pincus (L)

Stromatis Pharma, 9501 Discovery Blvd Manassas, VA 20109, USA.

Hua-Bei Guo (HB)

University of Georgia Cancer Center, Department of Biochemistry and Molecular Biology and the Complex Carbohydrate Research Center (CCRC), USA.

Samuel Dolezal (S)

University of Georgia Cancer Center, Department of Biochemistry and Molecular Biology and the Complex Carbohydrate Research Center (CCRC), USA.

J Michael Pierce (JM)

University of Georgia Cancer Center, Department of Biochemistry and Molecular Biology and the Complex Carbohydrate Research Center (CCRC), USA.

Emma Furth (E)

Perelman School of Medicine at the University of Pennsylvania, USA.

Cyrille Gineste (C)

Vaxxinity Inc, Merritt Island, Fl, USA.

Yongjun Kwon (Y)

Institute: Food and Drug Administration, CDER, MD, USA.

Cohava Gelber (C)

Stromatis Pharma, 9501 Discovery Blvd Manassas, VA 20109, USA.

Classifications MeSH