Alteration in Effects of Endometriosis on Fecundity According to Pregnancy Experience in Mouse Model.

Endometriosis Fecundity Pregnancy Pregnancy experience

Journal

Reproductive sciences (Thousand Oaks, Calif.)
ISSN: 1933-7205
Titre abrégé: Reprod Sci
Pays: United States
ID NLM: 101291249

Informations de publication

Date de publication:
05 Jan 2024
Historique:
received: 04 10 2023
accepted: 08 12 2023
medline: 5 1 2024
pubmed: 5 1 2024
entrez: 5 1 2024
Statut: aheadofprint

Résumé

This study is aimed at identifying variations in the effect of endometriosis on fecundity in a mouse model based on prior pregnancy experience. Endometriosis is one of the most prevalent gynecological diseases and is known to impact female fecundity adversely. In this study, an endometriosis mouse model was established by allografting uterine horn tissue using Pelch's method. The effect of endometriosis on fecundity was confirmed in primiparous and multiparous female mice. As fecundity indicators, the pregnancy rate, number of litters, pregnancy period, and survival rate of the pups were investigated. As a result of the experiment, the pregnancy rate decreased, and the pregnancy period tended to be shorter in primiparous female mice. However, there was no significant change in the multiparous mice. In addition, it has been established that correlations exist between the size of lesions and certain fecundity indicators of the lesion, even among primiparous and multiparous females with endometriosis. The study attempted to demonstrate a link between pregnancy experience and fecundity changes caused by endometriosis by experimentally reproducing clinical results using mouse models. These results suggest strategies for identifying several pathophysiological characteristics of endometriosis.

Identifiants

pubmed: 38180609
doi: 10.1007/s43032-023-01426-2
pii: 10.1007/s43032-023-01426-2
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Subventions

Organisme : Korea Health Industry Development Institute
ID : HI22C1424
Pays : Republic of Korea
Organisme : Commercializations Promotion Agency for R and D Outcomes
ID : 1711177795

Informations de copyright

© 2024. The Author(s), under exclusive licence to Society for Reproductive Investigation.

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Auteurs

Wonhyoung Park (W)

Institute of Animal Molecular Biotechnology and Department of Biotechnology, College of Life Sciences and Biotechnology, Korea University, Seoul, 02841, Republic of Korea.

Miji Kim (M)

Institute of Animal Molecular Biotechnology and Department of Biotechnology, College of Life Sciences and Biotechnology, Korea University, Seoul, 02841, Republic of Korea.

Hee Seung Kim (HS)

Department of Obstetrics and Gynecology, Seoul National University Hospital, Seoul, 03080, Republic of Korea.
Department of Obstetrics and Gynecology, Seoul National University College of Medicine, Seoul, 03080, Republic of Korea.

Gwonhwa Song (G)

Institute of Animal Molecular Biotechnology and Department of Biotechnology, College of Life Sciences and Biotechnology, Korea University, Seoul, 02841, Republic of Korea.

Soo Jin Park (SJ)

Department of Obstetrics and Gynecology, Seoul National University Hospital, Seoul, 03080, Republic of Korea. soojin.mdpark@gmail.com.

Whasun Lim (W)

Department of Biological Sciences, College of Science, Sungkyunkwan University, Suwon, 16419, Republic of Korea. wlim@skku.edu.

Sunwoo Park (S)

Department of Plant & Biomaterials Science, Gyeongsang National University, Jinju, 52725, Republic of Korea. sw.park@gnu.ac.kr.
Department of GreenBio Science, Gyeongsang National University, Jinju, 52725, Republic of Korea. sw.park@gnu.ac.kr.

Classifications MeSH