p4EBP1 staining predicts outcome in ER-positive endocrine-resistant metastatic breast cancer patients treated with everolimus and exemestane.


Journal

British journal of cancer
ISSN: 1532-1827
Titre abrégé: Br J Cancer
Pays: England
ID NLM: 0370635

Informations de publication

Date de publication:
05 Jan 2024
Historique:
received: 07 06 2023
accepted: 11 12 2023
revised: 27 11 2023
medline: 6 1 2024
pubmed: 6 1 2024
entrez: 5 1 2024
Statut: aheadofprint

Résumé

To identify patients most likely to respond to everolimus, a mammalian target of rapamycin (mTOR) inhibitor, a prospective biomarker study was conducted in hormone receptor-positive endocrine-resistant metastatic breast cancer patients treated with exemestane-everolimus therapy. Metastatic tumor biopsies were processed for immunohistochemical staining (p4EBP1, PTEN, pAKT, LKB1, and pS6K). ESR1, PIK3CA and AKT1 gene mutations were detected by NGS. The primary endpoint was the association between the p4EBP1 expression and clinical benefit rate (CBR) at 6 months of everolimus plus exemestane treatment. Of 150 patients included, 107 were evaluable for the primary endpoint. p4EBP1 staining above the median (Allred score ≥6) was associated with a higher CBR at 6 months (62% versus 40% in high-p4EBP1 versus low-p4EBP1, χ2 test, p = 0.026) and a longer progression-free survival (PFS) (median PFS of 9.2 versus 5.8 months in high-p4EBP1 versus low-p4EBP1; p = 0.02). When tested with other biomarkers, only p4EBP1 remained a significant predictive marker of PFS in multivariate analysis (hazard ratio, 0.591; p = 0.01). This study identified a subset of patients with hormone receptor-positive endocrine-resistant metastatic breast cancer and poor outcome who would derive less benefit from everolimus and exemestane. p4EBP1 may be a useful predictive biomarker in routine clinical practice. NCT02444390.

Sections du résumé

BACKGROUND BACKGROUND
To identify patients most likely to respond to everolimus, a mammalian target of rapamycin (mTOR) inhibitor, a prospective biomarker study was conducted in hormone receptor-positive endocrine-resistant metastatic breast cancer patients treated with exemestane-everolimus therapy.
METHODS METHODS
Metastatic tumor biopsies were processed for immunohistochemical staining (p4EBP1, PTEN, pAKT, LKB1, and pS6K). ESR1, PIK3CA and AKT1 gene mutations were detected by NGS. The primary endpoint was the association between the p4EBP1 expression and clinical benefit rate (CBR) at 6 months of everolimus plus exemestane treatment.
RESULTS RESULTS
Of 150 patients included, 107 were evaluable for the primary endpoint. p4EBP1 staining above the median (Allred score ≥6) was associated with a higher CBR at 6 months (62% versus 40% in high-p4EBP1 versus low-p4EBP1, χ2 test, p = 0.026) and a longer progression-free survival (PFS) (median PFS of 9.2 versus 5.8 months in high-p4EBP1 versus low-p4EBP1; p = 0.02). When tested with other biomarkers, only p4EBP1 remained a significant predictive marker of PFS in multivariate analysis (hazard ratio, 0.591; p = 0.01).
CONCLUSIONS CONCLUSIONS
This study identified a subset of patients with hormone receptor-positive endocrine-resistant metastatic breast cancer and poor outcome who would derive less benefit from everolimus and exemestane. p4EBP1 may be a useful predictive biomarker in routine clinical practice.
CLINICAL TRIAL REGISTRATION BACKGROUND
NCT02444390.

Identifiants

pubmed: 38182687
doi: 10.1038/s41416-023-02549-8
pii: 10.1038/s41416-023-02549-8
doi:

Banques de données

ClinicalTrials.gov
['NCT02444390']

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© 2024. The Author(s), under exclusive licence to Springer Nature Limited.

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Auteurs

Hélène Vanacker (H)

Centre Léon Bérard, Lyon, France.

Isabelle Treilleux (I)

Centre Léon Bérard, Lyon, France.

Camille Schiffler (C)

Centre Léon Bérard, Lyon, France.

Ivan Bieche (I)

Institut Curie, Paris, France.

Mario Campone (M)

Institut de cancérologie de l'ouest Pays de Loire Nantes-Angers, Saint-Herblain, France.

Anne Patsouris (A)

Institut de cancérologie de l'ouest Pays de Loire Nantes-Angers, Saint-Herblain, France.

Monica Arnedos (M)

Gustave Roussy, Villejuif, France.

Paul H Cottu (PH)

Institut Curie, Paris, France.

Jean-Philippe Jacquin (JP)

Institut de Cancérologie Lucien Neuwirth, Saint-Priest-En-Jarez, France.

Florence Dalenc (F)

ICR, Institut Universitaire du Cancer de Toulouse, Oncopole, Toulouse, France.

Antoine Pinton (A)

Institut Curie, Paris, France.

Nicolas Servant (N)

Institut Curie, Paris, France.

Valéry Attignon (V)

Centre Léon Bérard, Lyon, France.

Etienne Rouleau (E)

Gustave Roussy, Villejuif, France.

Alain Morel (A)

Institut de cancérologie de l'ouest Pays de Loire Nantes-Angers, Saint-Herblain, France.
Univ Angers, Nantes Université, Inserm, CNRS, CRCI2NA, SFR ICAT, F-49000, Angers, France.

François Legrand (F)

UNICANCER R&D, Paris, France.

Marta Jimenez (M)

UNICANCER R&D, Paris, France.

Fabrice Andre (F)

Gustave Roussy, Villejuif, France.

Thomas Bachelot (T)

Centre Léon Bérard, Lyon, France. thomas.bachelot@lyon.unicancer.fr.

Classifications MeSH