Regulation of RAS palmitoyltransferases by accessory proteins and palmitoylation.


Journal

Nature structural & molecular biology
ISSN: 1545-9985
Titre abrégé: Nat Struct Mol Biol
Pays: United States
ID NLM: 101186374

Informations de publication

Date de publication:
Mar 2024
Historique:
received: 02 03 2023
accepted: 17 11 2023
pubmed: 6 1 2024
medline: 6 1 2024
entrez: 5 1 2024
Statut: ppublish

Résumé

Palmitoylation of cysteine residues at the C-terminal hypervariable regions in human HRAS and NRAS, which is necessary for RAS signaling, is catalyzed by the acyltransferase DHHC9 in complex with its accessory protein GCP16. The molecular basis for the acyltransferase activity and the regulation of DHHC9 by GCP16 is not clear. Here we report the cryo-electron microscopy structures of the human DHHC9-GCP16 complex and its yeast counterpart-the Erf2-Erf4 complex, demonstrating that GCP16 and Erf4 are not directly involved in the catalytic process but stabilize the architecture of DHHC9 and Erf2, respectively. We found that a phospholipid binding to an arginine-rich region of DHHC9 and palmitoylation on three residues (C24, C25 and C288) were essential for the catalytic activity of the DHHC9-GCP16 complex. Moreover, we showed that GCP16 also formed complexes with DHHC14 and DHHC18 to catalyze RAS palmitoylation. These findings provide insights into the regulatory mechanism of RAS palmitoyltransferases.

Identifiants

pubmed: 38182928
doi: 10.1038/s41594-023-01183-5
pii: 10.1038/s41594-023-01183-5
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

436-446

Informations de copyright

© 2024. The Author(s), under exclusive licence to Springer Nature America, Inc.

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Auteurs

Anlan Yang (A)

College of Life Sciences, Zhejiang University, Hangzhou, China.
Key Laboratory of Structural Biology of Zhejiang Province, School of Life Sciences, Westlake University, Hangzhou, China.

Shengjie Liu (S)

Key Laboratory of Structural Biology of Zhejiang Province, School of Life Sciences, Westlake University, Hangzhou, China.
Fudan University, Shanghai, China.

Yuqi Zhang (Y)

Key Laboratory of Structural Biology of Zhejiang Province, School of Life Sciences, Westlake University, Hangzhou, China.

Jia Chen (J)

Key Laboratory of Structural Biology of Zhejiang Province, School of Life Sciences, Westlake University, Hangzhou, China.
Mass Spectrometry & Metabolomics Core Facility, Westlake University, Hangzhou, China.

Yujing Fan (Y)

Westlake Four-Dimensional Dynamic Metabolomics (Meta4D) Laboratory, Westlake Laboratory of Life Sciences and Biomedicine, Hangzhou, China.

Fengxiang Wang (F)

Westlake Four-Dimensional Dynamic Metabolomics (Meta4D) Laboratory, Westlake Laboratory of Life Sciences and Biomedicine, Hangzhou, China.
School of Life Sciences, Westlake University, Hangzhou, China.

Yilong Zou (Y)

Westlake Four-Dimensional Dynamic Metabolomics (Meta4D) Laboratory, Westlake Laboratory of Life Sciences and Biomedicine, Hangzhou, China.
School of Life Sciences, Westlake University, Hangzhou, China.

Shan Feng (S)

Key Laboratory of Structural Biology of Zhejiang Province, School of Life Sciences, Westlake University, Hangzhou, China.
Mass Spectrometry & Metabolomics Core Facility, Westlake University, Hangzhou, China.

Jianping Wu (J)

Key Laboratory of Structural Biology of Zhejiang Province, School of Life Sciences, Westlake University, Hangzhou, China. wujianping@westlake.edu.cn.
Westlake Laboratory of Life Sciences and Biomedicine, Hangzhou, China. wujianping@westlake.edu.cn.
Institute of Biology, Westlake Institute for Advanced Study, Hangzhou, China. wujianping@westlake.edu.cn.

Qi Hu (Q)

Key Laboratory of Structural Biology of Zhejiang Province, School of Life Sciences, Westlake University, Hangzhou, China. huqi@westlake.edu.cn.
Institute of Biology, Westlake Institute for Advanced Study, Hangzhou, China. huqi@westlake.edu.cn.
Westlake AI Therapeutics Lab, Westlake Laboratory of Life Sciences and Biomedicine, Hangzhou, China. huqi@westlake.edu.cn.

Classifications MeSH