Characteristics and prognosis factors of Pneumocystis jirovecii pneumonia according to underlying disease: a retrospective multicentre study.

Corticosteroids Immune-Mediated Inflammatory Diseases Outcome Pneumocystis jirovecii pneumonia Solid Tumors

Journal

Chest
ISSN: 1931-3543
Titre abrégé: Chest
Pays: United States
ID NLM: 0231335

Informations de publication

Date de publication:
10 Jan 2024
Historique:
received: 11 10 2023
revised: 15 12 2023
accepted: 05 01 2024
medline: 13 1 2024
pubmed: 13 1 2024
entrez: 12 1 2024
Statut: aheadofprint

Résumé

Pneumocystis jirovecii pneumonia (PcP) remains associated with high rates of mortality and the impact of immunocompromising underlying disease on the clinical presentation, severity and mortality of PcP has not been adequately evaluated. Does the underlying disease and immunosuppression causing Pneumocystis jirovecii pneumonia (PcP) impact the outcome and clinical presentation of the disease? In this multicentre retrospective observational study, conducted from January 2011 to December 2021, all consecutive patients admitted with a proven or probable diagnosis of PcP according to The European Organization for Research and Treatment of Cancer (EORTC) consensus definitions were included to assess the epidemiology and impact of underlying immunosuppressive diseases on overall and 90-day mortality. Overall, 481 patients were included in the study, 180 (37.4%) were defined as proven PcP and 301 (62.6%) as probable PcP. Patients with immune-mediated inflammatory diseases (IMID) or solid tumors had a statistically poorer prognosis than other patients with PcP at D90. In multivariate analysis, among HIV-negative population, solid tumor underlying disease (OR 5.47, 2.16-14.1; p<0.001), IMID (OR 2.19, 1.05-4.60; p=0.037), long term corticosteroid exposure (OR 2.07, 1.03-4.31; p=0.045), cysts in sputum/BAL smears (OR 1.92, 1.02-3.62; p=0.043), and SOFA score at admission (OR 1.58, 1.39-1.82; p<0.001) were independently associated with 90-day mortality. Prior corticotherapy was the only immunosuppressant associated with 90-day mortality (OR 1.67, 1.03-2.71; p=0.035), especially for a prednisone daily dose ≥ 10 mg (OR 1.80, 1.14-2.85; p=0.010). Among HIV-negative patients, long term corticosteroid prior to PcP diagnosis was independently associated with increased 90-day mortality, specifically in IMID patients. These results highlight both the needs for PcP prophylaxis in IMID patients as well as to early consider PcP curative treatment in severe pneumonia among IMID patients.

Sections du résumé

BACKGROUND BACKGROUND
Pneumocystis jirovecii pneumonia (PcP) remains associated with high rates of mortality and the impact of immunocompromising underlying disease on the clinical presentation, severity and mortality of PcP has not been adequately evaluated.
RESEARCH QUESTION OBJECTIVE
Does the underlying disease and immunosuppression causing Pneumocystis jirovecii pneumonia (PcP) impact the outcome and clinical presentation of the disease?
STUDY DESIGN AND METHODS METHODS
In this multicentre retrospective observational study, conducted from January 2011 to December 2021, all consecutive patients admitted with a proven or probable diagnosis of PcP according to The European Organization for Research and Treatment of Cancer (EORTC) consensus definitions were included to assess the epidemiology and impact of underlying immunosuppressive diseases on overall and 90-day mortality.
RESULTS RESULTS
Overall, 481 patients were included in the study, 180 (37.4%) were defined as proven PcP and 301 (62.6%) as probable PcP. Patients with immune-mediated inflammatory diseases (IMID) or solid tumors had a statistically poorer prognosis than other patients with PcP at D90. In multivariate analysis, among HIV-negative population, solid tumor underlying disease (OR 5.47, 2.16-14.1; p<0.001), IMID (OR 2.19, 1.05-4.60; p=0.037), long term corticosteroid exposure (OR 2.07, 1.03-4.31; p=0.045), cysts in sputum/BAL smears (OR 1.92, 1.02-3.62; p=0.043), and SOFA score at admission (OR 1.58, 1.39-1.82; p<0.001) were independently associated with 90-day mortality. Prior corticotherapy was the only immunosuppressant associated with 90-day mortality (OR 1.67, 1.03-2.71; p=0.035), especially for a prednisone daily dose ≥ 10 mg (OR 1.80, 1.14-2.85; p=0.010).
INTERPRETATION CONCLUSIONS
Among HIV-negative patients, long term corticosteroid prior to PcP diagnosis was independently associated with increased 90-day mortality, specifically in IMID patients. These results highlight both the needs for PcP prophylaxis in IMID patients as well as to early consider PcP curative treatment in severe pneumonia among IMID patients.

Identifiants

pubmed: 38215935
pii: S0012-3692(24)00022-9
doi: 10.1016/j.chest.2024.01.015
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

Copyright © 2024. Published by Elsevier Inc.

Auteurs

Romain Lécuyer (R)

Internal medicine and infectious diseases, Centre Hospitalier Bretagne-Atlantique, Vannes, France; Nantes Université, CHU Nantes, Cibles et médicaments des infections et de l'immunité, IICiMed, UR1155, F-44000 Nantes, France.

Nahéma Issa (N)

Intensive Care and Infectious Disease Unit, Groupe Saint-André, University Hospital, Bordeaux, France.

Fabrice Camou (F)

Intensive Care and Infectious Disease Unit, Groupe Saint-André, University Hospital, Bordeaux, France.

Rose-Anne Lavergne (RA)

Nantes Université, CHU Nantes, Cibles et médicaments des infections et de l'immunité, IICiMed, UR1155, F-44000 Nantes, France.

Frederic Gabriel (F)

Centre Hospitalier Universitaire de Bordeaux, Service de Parasitologie Mycologie, F-33000, Bordeaux, France.

Florent Morio (F)

Nantes Université, CHU Nantes, Cibles et médicaments des infections et de l'immunité, IICiMed, UR1155, F-44000 Nantes, France; Laboratoire de Parasitologie-Mycologie, Institut de Biologie, University Hospital, Nantes, France.

Emmanuel Canet (E)

Medical Intensive Care, University Hospital, Nantes, France.

François Raffi (F)

Department of Infectious Diseases, University Hospital of Nantes and Centre d'Investigation Clinique 1413, INSERM, Nantes, France.

David Boutoille (D)

Department of Infectious Diseases, University Hospital of Nantes and Centre d'Investigation Clinique 1413, INSERM, Nantes, France; Nantes Université, CHU Nantes, INSERM, Center for Research in Transplantation and Translational Immunology, UMR 1064, F-44000 Nantes, France.

Anne Cady (A)

Department of Microbiology, Centre Hospitalier Bretagne-Atlantique, Vannes, France.

Marie Gousseff (M)

Internal medicine and infectious diseases, Centre Hospitalier Bretagne-Atlantique, Vannes, France.

Yoann Crabol (Y)

Internal medicine and infectious diseases, Centre Hospitalier Bretagne-Atlantique, Vannes, France.

Antoine Néel (A)

CRTI UMR 1064, INSERM, Université de Nantes, Nantes, France; Department of Internal medicine, University Hospital, Nantes, France.

Benoît Tessoulin (B)

INSERM, U1232, Hematology Department, Nantes University Hospital, CRCI(2)NA, Nantes University, Nantes, France.

Benjamin Gaborit (B)

Department of Infectious Diseases, University Hospital of Nantes and Centre d'Investigation Clinique 1413, INSERM, Nantes, France; Nantes Université, CHU Nantes, INSERM, Center for Research in Transplantation and Translational Immunology, UMR 1064, F-44000 Nantes, France. Electronic address: benjamin.gaborit@chu-nantes.fr.

Classifications MeSH