Vitamin D insufficiency in infants with increased risk of developing type 1 diabetes: a secondary analysis of the POInT Study.

Endocrinology

Journal

BMJ paediatrics open
ISSN: 2399-9772
Titre abrégé: BMJ Paediatr Open
Pays: England
ID NLM: 101715309

Informations de publication

Date de publication:
12 Jan 2024
Historique:
received: 02 08 2023
accepted: 07 11 2023
medline: 13 1 2024
pubmed: 13 1 2024
entrez: 12 1 2024
Statut: epublish

Résumé

Vitamin D insufficiency (VDI) may be a factor in the development of type 1 diabetes (T1D). The aim of this study is to investigate the presence and persistence of VDI in a large cohort of infants with increased risk of developing T1D, in light of the differences in local supplementation guidelines. In the POInT Study, a multicentre primary prevention study between February 2018 and March 2021 in Germany, Poland, Belgium, England and Sweden, including infants aged 4-7 months at high genetic risk of developing β-cell autoantibodies, vitamin D levels were analysed at each study visit from inclusion (4-7 months) until 3 years, with an interval of 2 months (first three visits) or 4-6 months (visits 4-8). The protocol actively promotes vitamin D sufficiency to optimise immune tolerance. VDI was defined as a concentration below 30 ng/mL and was treated according to local guidelines of participating centres. Recovery from VDI was defined as a concentration above or equal to 30 ng/mL on the subsequent visit after VDI. 1050 infants were included, of which 5937 vitamin D levels were available for analyses. VDI was observed in 1464 (24.7%) visits and 507 (46.1%) of these were not resolved at the next visit. The risk of having VDI was independently associated with season (higher in winter), weight (higher with increased weight), age (higher with increased age) and country (higher in England). The risk of not recovering from VDI was independently associated with the season of the previously determined VDI, which was higher if VDI was identified in winter. VDI is frequent in infants with increased risk of developing T1D. Treatment guidelines for VDI do not seem effective. Increasing supplementation dosages in this patient population seems warranted, especially during winter, and increasing dosages more aggressively after VDI should be considered.

Sections du résumé

BACKGROUND BACKGROUND
Vitamin D insufficiency (VDI) may be a factor in the development of type 1 diabetes (T1D). The aim of this study is to investigate the presence and persistence of VDI in a large cohort of infants with increased risk of developing T1D, in light of the differences in local supplementation guidelines.
METHODS METHODS
In the POInT Study, a multicentre primary prevention study between February 2018 and March 2021 in Germany, Poland, Belgium, England and Sweden, including infants aged 4-7 months at high genetic risk of developing β-cell autoantibodies, vitamin D levels were analysed at each study visit from inclusion (4-7 months) until 3 years, with an interval of 2 months (first three visits) or 4-6 months (visits 4-8). The protocol actively promotes vitamin D sufficiency to optimise immune tolerance. VDI was defined as a concentration below 30 ng/mL and was treated according to local guidelines of participating centres. Recovery from VDI was defined as a concentration above or equal to 30 ng/mL on the subsequent visit after VDI.
RESULTS RESULTS
1050 infants were included, of which 5937 vitamin D levels were available for analyses. VDI was observed in 1464 (24.7%) visits and 507 (46.1%) of these were not resolved at the next visit. The risk of having VDI was independently associated with season (higher in winter), weight (higher with increased weight), age (higher with increased age) and country (higher in England). The risk of not recovering from VDI was independently associated with the season of the previously determined VDI, which was higher if VDI was identified in winter.
CONCLUSIONS CONCLUSIONS
VDI is frequent in infants with increased risk of developing T1D. Treatment guidelines for VDI do not seem effective. Increasing supplementation dosages in this patient population seems warranted, especially during winter, and increasing dosages more aggressively after VDI should be considered.

Identifiants

pubmed: 38216311
pii: 10.1136/bmjpo-2023-002212
doi: 10.1136/bmjpo-2023-002212
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Informations de copyright

© Author(s) (or their employer(s)) 2024. Re-use permitted under CC BY-NC. No commercial re-use. See rights and permissions. Published by BMJ.

Déclaration de conflit d'intérêts

Competing interests: None declared.

Auteurs

An Jacobs (A)

Department of Pediatric Endocrinology and Diabetes, KU Leuven University Hospitals Leuven, Leuven, Belgium an.k.jacobs@gmail.com.

Maarten Warnants (M)

Katholieke Universiteit Leuven, Leuven, Belgium.

Veronika Vollmuth (V)

Institute of Diabetes Research, Helmholtz Munich, German Center for Environmental Health, Munich, Germany.

Christiane Winkler (C)

Institute of Diabetes Research, Helmholtz Munich, German Center for Environmental Health, Munich, Germany.
Forschergruppe Diabetes at Klinikum rechts der Isar, School of Medicine, Technical University Munich, Munich, Germany.

Andreas Weiss (A)

Institute of Diabetes Research, Helmholtz Munich, German Center for Environmental Health, Munich, Germany.

Anette-Gabriele Ziegler (AG)

Institute of Diabetes Research, Helmholtz Munich, German Center for Environmental Health, Munich, Germany.
Forschergruppe Diabetes, Klinikum rechts der Isar, Technische Universität München, Munich, Germany.

Markus Lundgren (M)

Department of Clinical Sciences Malmö, Lund University, Malmö, Sweden.
Department of Pediatrics, Kristianstad Hospital, Kristianstad, Sweden.

Helena Elding Larsson (H)

Department of Pediatrics, Skåne University Hospital, Malmö, Sweden.
Department of Clinical Sciences Malmö, Lund University, Lund, Sweden.

Olga Kordonouri (O)

Auf der Bult Children's Hospital, Hannover, Germany.

Thekla von dem Berge (T)

Auf der Bult Children's Hospital, Hannover, Germany.

Marie-Luise Zielmann (ML)

Department of Pediatrics, Faculty of Medicine and University Hospital Carl Gustav Carus, Technische Universität Dresden, Dresden, Germany.

Ezio Bonifacio (E)

Center for Regenerative Therapies, Technische Universität Dresden, Dresden, Germany.

Angela Hommel (A)

Center for Regenerative Therapies, Technische Universität Dresden, Dresden, Germany.

Mariusz Ołtarzewski (M)

Department of Screening and Metabolic Diagnostics, Institute of Mother and Child, Warsaw, Poland.

Agnieszka Szypowska (A)

Department of Paediatrics, Medical University of Warsaw, Warsaw, Poland.

Rachel Besser (R)

Department of Paediatrics, University of Oxford, Oxford, UK.
Wellcome Centre for Human Genetics, Nuffield Department of Medicine, NIHR Oxford Biomedical Research Centre, University of Oxford, Oxford, UK.

John A Todd (JA)

Wellcome Centre for Human Genetics, Nuffield Department of Medicine, NIHR Oxford Biomedical Research Centre, University of Oxford, Oxford, UK.

Kristina Casteels (K)

Department of Pediatric Endocrinology and Diabetes, KU Leuven University Hospitals Leuven, Leuven, Belgium.
Department of Development and Regeneration, KU Leuven, Leuven, Belgium.

Classifications MeSH