Farnesyl-transferase inhibitors show synergistic anticancer effects in combination with novel KRAS-G12C inhibitors.


Journal

British journal of cancer
ISSN: 1532-1827
Titre abrégé: Br J Cancer
Pays: England
ID NLM: 0370635

Informations de publication

Date de publication:
Apr 2024
Historique:
received: 01 06 2023
accepted: 15 01 2024
revised: 10 01 2024
pubmed: 27 1 2024
medline: 27 1 2024
entrez: 26 1 2024
Statut: ppublish

Résumé

Inhibition of mutant KRAS challenged cancer research for decades. Recently, allele-specific inhibitors were approved for the treatment of KRAS-G12C mutant lung cancer. However, de novo and acquired resistance limit their efficacy and several combinations are in clinical development. Our study shows the potential of combining G12C inhibitors with farnesyl-transferase inhibitors. Combinations of clinically approved farnesyl-transferase inhibitors and KRAS G12C inhibitors are tested on human lung, colorectal and pancreatic adenocarcinoma cells in vitro in 2D, 3D and subcutaneous xenograft models of lung adenocarcinoma. Treatment effects on migration, proliferation, apoptosis, farnesylation and RAS signaling were measured by histopathological analyses, videomicroscopy, cell cycle analyses, immunoblot, immunofluorescence and RAS pulldown. Combination of tipifarnib with sotorasib shows synergistic inhibitory effects on lung adenocarcinoma cells in vitro in 2D and 3D. Mechanistically, we present antiproliferative effect of the combination and interference with compensatory HRAS activation and RHEB and lamin farnesylation. Enhanced efficacy of sotorasib in combination with tipifarnib is recapitulated in the subcutaneous xenograft model of lung adenocarcinoma. Finally, combination of additional KRAS G1C and farnesyl-transferase inhibitors also shows synergism in lung, colorectal and pancreatic adenocarcinoma cellular models. Our findings warrant the clinical exploration of KRAS-G12C inhibitors in combination with farnesyl-transferase inhibitors.

Sections du résumé

BACKGROUND BACKGROUND
Inhibition of mutant KRAS challenged cancer research for decades. Recently, allele-specific inhibitors were approved for the treatment of KRAS-G12C mutant lung cancer. However, de novo and acquired resistance limit their efficacy and several combinations are in clinical development. Our study shows the potential of combining G12C inhibitors with farnesyl-transferase inhibitors.
METHODS METHODS
Combinations of clinically approved farnesyl-transferase inhibitors and KRAS G12C inhibitors are tested on human lung, colorectal and pancreatic adenocarcinoma cells in vitro in 2D, 3D and subcutaneous xenograft models of lung adenocarcinoma. Treatment effects on migration, proliferation, apoptosis, farnesylation and RAS signaling were measured by histopathological analyses, videomicroscopy, cell cycle analyses, immunoblot, immunofluorescence and RAS pulldown.
RESULTS RESULTS
Combination of tipifarnib with sotorasib shows synergistic inhibitory effects on lung adenocarcinoma cells in vitro in 2D and 3D. Mechanistically, we present antiproliferative effect of the combination and interference with compensatory HRAS activation and RHEB and lamin farnesylation. Enhanced efficacy of sotorasib in combination with tipifarnib is recapitulated in the subcutaneous xenograft model of lung adenocarcinoma. Finally, combination of additional KRAS G1C and farnesyl-transferase inhibitors also shows synergism in lung, colorectal and pancreatic adenocarcinoma cellular models.
DISCUSSION CONCLUSIONS
Our findings warrant the clinical exploration of KRAS-G12C inhibitors in combination with farnesyl-transferase inhibitors.

Identifiants

pubmed: 38278976
doi: 10.1038/s41416-024-02586-x
pii: 10.1038/s41416-024-02586-x
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1059-1072

Subventions

Organisme : Nemzeti Kutatási, Fejlesztési és Innovációs Hivatal (NKFI Office)
ID : ÚNKP-20-3-II
Organisme : Nemzeti Kutatási, Fejlesztési és Innovációs Hivatal (NKFI Office)
ID : ÚNKP-21-4-I-SE-24
Organisme : Nemzeti Kutatási, Fejlesztési és Innovációs Hivatal (NKFI Office)
ID : ÚNKP-22-4-II-SE-11

Informations de copyright

© 2024. The Author(s).

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Auteurs

Marcell Baranyi (M)

Department of Pathology, Forensic and Insurance Medicine, Semmelweis University, H-1091, Budapest, Hungary.
KINETO Lab Ltd, H-1037, Budapest, Hungary.

Eszter Molnár (E)

Department of Pathology, Forensic and Insurance Medicine, Semmelweis University, H-1091, Budapest, Hungary.

Luca Hegedűs (L)

Department of Thoracic Surgery, University Medicine Essen - Ruhrlandklinik, University Duisburg-Essen, D-45239, Essen, Germany.

Zsófia Gábriel (Z)

Department of Pathology, Forensic and Insurance Medicine, Semmelweis University, H-1091, Budapest, Hungary.
Pázmány Péter Catholic University Faculty of Information Technology and Bionics, H-1083, Budapest, Hungary.

Flóra Gréta Petényi (FG)

Department of Pathology, Forensic and Insurance Medicine, Semmelweis University, H-1091, Budapest, Hungary.
Pázmány Péter Catholic University Faculty of Information Technology and Bionics, H-1083, Budapest, Hungary.

Fanni Bordás (F)

Department of Pathology, Forensic and Insurance Medicine, Semmelweis University, H-1091, Budapest, Hungary.
Department of Anatomy, Cell and Developmental Biology, Eötvös Loránd University, H-1117, Budapest, Hungary.

Violetta Léner (V)

KINETO Lab Ltd, H-1037, Budapest, Hungary.

Ivan Ranđelović (I)

KINETO Lab Ltd, H-1037, Budapest, Hungary.
Department of Experimental Pharmacology and the National Tumor Biology Laboratory, National Institute of Oncology, H-1122, Budapest, Hungary.

Mihály Cserepes (M)

KINETO Lab Ltd, H-1037, Budapest, Hungary.
Department of Experimental Pharmacology and the National Tumor Biology Laboratory, National Institute of Oncology, H-1122, Budapest, Hungary.

József Tóvári (J)

Department of Experimental Pharmacology and the National Tumor Biology Laboratory, National Institute of Oncology, H-1122, Budapest, Hungary.

Balázs Hegedűs (B)

Department of Pathology, Forensic and Insurance Medicine, Semmelweis University, H-1091, Budapest, Hungary. Balazs.Hegedues@rlk.uk-essen.de.
Department of Thoracic Surgery, University Medicine Essen - Ruhrlandklinik, University Duisburg-Essen, D-45239, Essen, Germany. Balazs.Hegedues@rlk.uk-essen.de.

József Tímár (J)

Department of Pathology, Forensic and Insurance Medicine, Semmelweis University, H-1091, Budapest, Hungary.

Classifications MeSH