Unraveling the role of miRNAs as biomarkers in Chagas cardiomyopathy: Insights into molecular pathophysiology.


Journal

PLoS neglected tropical diseases
ISSN: 1935-2735
Titre abrégé: PLoS Negl Trop Dis
Pays: United States
ID NLM: 101291488

Informations de publication

Date de publication:
Feb 2024
Historique:
medline: 1 2 2024
pubmed: 1 2 2024
entrez: 1 2 2024
Statut: epublish

Résumé

Chagas cardiomyopathy (ChCM) is a severe form of Chagas disease and a major cause of cardiovascular morbidity and mortality. The dysregulation of the immune response leads to cardiac remodeling and functional disruptions, resulting in life-threatening complications. Conventional diagnostic methods have limitations, and therapeutic response evaluation is challenging. MicroRNAs (miRNAs), important regulators of gene expression, show potential as biomarkers for diagnosis and prognosis. This review aims to summarize experimental findings on miRNA expression in ChCM and explore the potential of these miRNAs as biomarkers of Chagas disease. The search was conducted in the US National Library of Medicine MEDLINE/PubMed public database using the terms "Chagas cardiomyopathy" OR "Chagas disease" AND "microRNA" OR "miRNA" OR "miR." Additionally, bioinformatics analysis was performed to investigate miRNA-target interactions and explore enrichment pathways of gene ontology biological processes and molecular functions. The miR-21, miR-146b, miR-146a, and miR-155 consistently exhibited up-regulation, whereas miR-145 was down-regulated in ChCM. These specific miRNAs have been linked to fibrosis, immune response, and inflammatory processes in heart tissue. Moreover, the findings from various studies indicate that these miRNAs have the potential as biomarkers for the disease and could be targeted in therapeutic strategies for ChCM. In this review, we point out miR-21, miR-146b, miR-146a, miR-155, and miR-145-5p role in the complex mechanisms of ChCM. These miRNAs have been shown as potential biomarkers for precise diagnosis, reliable prognostic evaluation, and effective treatment strategies in the ChCM.

Sections du résumé

BACKGROUND BACKGROUND
Chagas cardiomyopathy (ChCM) is a severe form of Chagas disease and a major cause of cardiovascular morbidity and mortality. The dysregulation of the immune response leads to cardiac remodeling and functional disruptions, resulting in life-threatening complications. Conventional diagnostic methods have limitations, and therapeutic response evaluation is challenging. MicroRNAs (miRNAs), important regulators of gene expression, show potential as biomarkers for diagnosis and prognosis.
AIM OBJECTIVE
This review aims to summarize experimental findings on miRNA expression in ChCM and explore the potential of these miRNAs as biomarkers of Chagas disease.
METHODS METHODS
The search was conducted in the US National Library of Medicine MEDLINE/PubMed public database using the terms "Chagas cardiomyopathy" OR "Chagas disease" AND "microRNA" OR "miRNA" OR "miR." Additionally, bioinformatics analysis was performed to investigate miRNA-target interactions and explore enrichment pathways of gene ontology biological processes and molecular functions.
RESULTS RESULTS
The miR-21, miR-146b, miR-146a, and miR-155 consistently exhibited up-regulation, whereas miR-145 was down-regulated in ChCM. These specific miRNAs have been linked to fibrosis, immune response, and inflammatory processes in heart tissue. Moreover, the findings from various studies indicate that these miRNAs have the potential as biomarkers for the disease and could be targeted in therapeutic strategies for ChCM.
CONCLUSION CONCLUSIONS
In this review, we point out miR-21, miR-146b, miR-146a, miR-155, and miR-145-5p role in the complex mechanisms of ChCM. These miRNAs have been shown as potential biomarkers for precise diagnosis, reliable prognostic evaluation, and effective treatment strategies in the ChCM.

Identifiants

pubmed: 38300899
doi: 10.1371/journal.pntd.0011865
pii: PNTD-D-23-00901
doi:

Types de publication

Journal Article Review

Langues

eng

Sous-ensembles de citation

IM

Pagination

e0011865

Informations de copyright

Copyright: © 2024 Ribeiro et al. This is an open access article distributed under the terms of the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original author and source are credited.

Déclaration de conflit d'intérêts

The authors have declared that no competing interests exist.

Auteurs

Heriks Gomes Ribeiro (HG)

Department of Clinical and Toxicological Analysis, Federal University of Rio Grande do Norte, Natal, Brazil.

Ony Araújo Galdino (OA)

Department of Clinical and Toxicological Analysis, Federal University of Rio Grande do Norte, Natal, Brazil.

Karla Simone Costa de Souza (KSC)

Department of Clinical and Toxicological Analysis, Federal University of Rio Grande do Norte, Natal, Brazil.

Antonia Pereira Rosa Neta (AP)

Department of Clinical and Toxicological Analysis, Federal University of Rio Grande do Norte, Natal, Brazil.

Hui Tzu Lin-Wang (HT)

Molecular Biology Laboratory, Dante Pazzanese Institute of Cardiology, São Paulo, Brazil.

Edecio Cunha-Neto (E)

Laboratory of Immunology, Heart Institute (InCor), University of São Paulo, School of Medicine, São Paulo, Brazil.
Division of Clinical Immunology and Allergy, University of São Paulo, School of Medicine, São Paulo, Brazil.
Institute for Investigation in Immunology (iii), INCT, São Paulo, Brazil.

Adriana Augusto de Rezende (AA)

Department of Clinical and Toxicological Analysis, Federal University of Rio Grande do Norte, Natal, Brazil.

Vivian Nogueira Silbiger (VN)

Department of Clinical and Toxicological Analysis, Federal University of Rio Grande do Norte, Natal, Brazil.
Translational Medicine, The Hospital for Sick Children, Toronto, Ontario, Canada.

Classifications MeSH