NAAA-regulated lipid signaling in monocytes controls the induction of hyperalgesic priming in mice.


Journal

Nature communications
ISSN: 2041-1723
Titre abrégé: Nat Commun
Pays: England
ID NLM: 101528555

Informations de publication

Date de publication:
24 Feb 2024
Historique:
received: 14 08 2023
accepted: 15 02 2024
medline: 26 2 2024
pubmed: 25 2 2024
entrez: 24 2 2024
Statut: epublish

Résumé

Circulating monocytes participate in pain chronification but the molecular events that cause their deployment are unclear. Using a mouse model of hyperalgesic priming (HP), we show that monocytes enable progression to pain chronicity through a mechanism that requires transient activation of the hydrolase, N-acylethanolamine acid amidase (NAAA), and the consequent suppression of NAAA-regulated lipid signaling at peroxisome proliferator-activated receptor-α (PPAR-α). Inhibiting NAAA in the 72 hours following administration of a priming stimulus prevented HP. This effect was phenocopied by NAAA deletion and depended on PPAR-α recruitment. Mice lacking NAAA in CD11b

Identifiants

pubmed: 38402219
doi: 10.1038/s41467-024-46139-5
pii: 10.1038/s41467-024-46139-5
pmc: PMC10894261
doi:

Substances chimiques

Amidohydrolases EC 3.5.-
Enzyme Inhibitors 0
Lipids 0
NAAA protein, human EC 3.5.1.-
PPAR alpha 0
NAAA protein, mouse EC 3.5.1.-

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

1705

Subventions

Organisme : NCCIH NIH HHS
ID : K99 AT012658
Pays : United States
Organisme : NIDA NIH HHS
ID : R01 DA055578
Pays : United States
Organisme : NINDS NIH HHS
ID : R01 NS120060
Pays : United States

Informations de copyright

© 2024. The Author(s).

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Auteurs

Yannick Fotio (Y)

Department of Anatomy and Neurobiology, University of California Irvine, Irvine, CA, USA.

Alex Mabou Tagne (A)

Department of Anatomy and Neurobiology, University of California Irvine, Irvine, CA, USA.

Erica Squire (E)

Department of Anatomy and Neurobiology, University of California Irvine, Irvine, CA, USA.

Hye-Lim Lee (HL)

Department of Anatomy and Neurobiology, University of California Irvine, Irvine, CA, USA.

Connor M Phillips (CM)

Department of Physiology and Biophysics, University of California Irvine, Irvine, CA, USA.

Kayla Chang (K)

Department of Anatomy and Neurobiology, University of California Irvine, Irvine, CA, USA.

Faizy Ahmed (F)

Department of Anatomy and Neurobiology, University of California Irvine, Irvine, CA, USA.

Andrew S Greenberg (AS)

Human Nutrition Research Center, Tufts University, Boston, MA, USA.

S Armando Villalta (SA)

Department of Physiology and Biophysics, University of California Irvine, Irvine, CA, USA.
Department of Neurology, University of California Irvine, Irvine, CA, USA.

Vanessa M Scarfone (VM)

Sue and Bill Gross Stem Cell Research Center, University of California Irvine, Irvine, CA, USA.

Gilberto Spadoni (G)

Dipartimento di Scienze Biomolecolari, Università di Urbino "Carlo Bo,", Urbino, Italy.

Marco Mor (M)

Dipartimento di Scienze degli Alimenti e del Farmaco, Università di Parma, Parma, Italy.

Daniele Piomelli (D)

Department of Anatomy and Neurobiology, University of California Irvine, Irvine, CA, USA. piomelli@hs.uci.edu.
Department of Biological Chemistry, University of California Irvine, Irvine, CA, USA. piomelli@hs.uci.edu.
Department of Pharmaceutical Sciences, University of California Irvine, Irvine, CA, USA. piomelli@hs.uci.edu.

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Classifications MeSH