The presence of exudative thickening of Bowman's capsule predict poor prognosis in diabetic kidney disease.

Bowman’s capsule Diabetic kidney disease Matrix Parietal epithelial cells

Journal

Diabetes research and clinical practice
ISSN: 1872-8227
Titre abrégé: Diabetes Res Clin Pract
Pays: Ireland
ID NLM: 8508335

Informations de publication

Date de publication:
23 Feb 2024
Historique:
received: 03 01 2024
revised: 10 02 2024
accepted: 22 02 2024
medline: 26 2 2024
pubmed: 26 2 2024
entrez: 25 2 2024
Statut: aheadofprint

Résumé

The relationship between Bowman's capsule thickening and progression of diabetic kidney disease (DKD) remains uncertain. Renal biopsy specimens from 145 DKD patients and 20 control subjects were evaluated for Bowman's capsule thickness. Immunohistochemical staining assessed col4α2, laminin β1, and albumin expression. In a discovery cohort of 111 DKD patients with eGFR ≥ 30 ml/min/1.73 m Three types of thickening were observed: fibrotic, exudative, and periglomerular fibrosis. Parietal epithelial cell matrix protein accumulation contributed to fibrotic thickening, while albumin was present in exudative thickening. Bowman's capsule was significantly thicker in DKD patients (5.74 ± 2.09 μm) compared to controls (3.38 ± 0.43 μm, P < 0.01). In discovery cohort, the group of exudative thickning had a poorer prognosis(median time 20 months vs 57 months, P = 0.000). Cox multivariate analysis revealed that exudative thickening of Bowman's capsule were associated with a poor prognosis. The validation cohorts confirmed the result. Various mechanisms contribute to Bowman's capsule thickening in DKD. The proportion of exudative thickening may serve as a valuable prognostic indicator for DKD patients.

Sections du résumé

BACKGROUND BACKGROUND
The relationship between Bowman's capsule thickening and progression of diabetic kidney disease (DKD) remains uncertain.
METHODS METHODS
Renal biopsy specimens from 145 DKD patients and 20 control subjects were evaluated for Bowman's capsule thickness. Immunohistochemical staining assessed col4α2, laminin β1, and albumin expression. In a discovery cohort of 111 DKD patients with eGFR ≥ 30 ml/min/1.73 m
RESULTS RESULTS
Three types of thickening were observed: fibrotic, exudative, and periglomerular fibrosis. Parietal epithelial cell matrix protein accumulation contributed to fibrotic thickening, while albumin was present in exudative thickening. Bowman's capsule was significantly thicker in DKD patients (5.74 ± 2.09 μm) compared to controls (3.38 ± 0.43 μm, P < 0.01). In discovery cohort, the group of exudative thickning had a poorer prognosis(median time 20 months vs 57 months, P = 0.000). Cox multivariate analysis revealed that exudative thickening of Bowman's capsule were associated with a poor prognosis. The validation cohorts confirmed the result.
CONCLUSIONS CONCLUSIONS
Various mechanisms contribute to Bowman's capsule thickening in DKD. The proportion of exudative thickening may serve as a valuable prognostic indicator for DKD patients.

Identifiants

pubmed: 38403176
pii: S0168-8227(24)00078-0
doi: 10.1016/j.diabres.2024.111594
pii:
doi:

Types de publication

Journal Article

Langues

eng

Sous-ensembles de citation

IM

Pagination

111594

Informations de copyright

Copyright © 2024. Published by Elsevier B.V.

Déclaration de conflit d'intérêts

Declaration of competing interest The authors declare that they have no known competing financial interests or personal relationships that could have appeared to influence the work reported in this paper.

Auteurs

Jiayue Lu (J)

Department of Nephrology, Molecular Cell Lab for Kidney Disease, Shanghai Peritoneal Dialysis Research Center, Renji Hospital, Uremia Diagnosis and Treatment Center, Shanghai Jiaotong University, School of Medicine, China.

Kewei Xie (K)

Department of Nephrology, Molecular Cell Lab for Kidney Disease, Shanghai Peritoneal Dialysis Research Center, Renji Hospital, Uremia Diagnosis and Treatment Center, Shanghai Jiaotong University, School of Medicine, China.

Xiajing Che (X)

Department of Nephrology, Molecular Cell Lab for Kidney Disease, Shanghai Peritoneal Dialysis Research Center, Renji Hospital, Uremia Diagnosis and Treatment Center, Shanghai Jiaotong University, School of Medicine, China.

Ahui Song (A)

Department of Nephrology, Molecular Cell Lab for Kidney Disease, Shanghai Peritoneal Dialysis Research Center, Renji Hospital, Uremia Diagnosis and Treatment Center, Shanghai Jiaotong University, School of Medicine, China.

Minfang Zhang (M)

Department of Nephrology, Molecular Cell Lab for Kidney Disease, Shanghai Peritoneal Dialysis Research Center, Renji Hospital, Uremia Diagnosis and Treatment Center, Shanghai Jiaotong University, School of Medicine, China.

Chaojun Qi (C)

Department of Nephrology, Molecular Cell Lab for Kidney Disease, Shanghai Peritoneal Dialysis Research Center, Renji Hospital, Uremia Diagnosis and Treatment Center, Shanghai Jiaotong University, School of Medicine, China.

Renhua Lu (R)

Department of Nephrology, Molecular Cell Lab for Kidney Disease, Shanghai Peritoneal Dialysis Research Center, Renji Hospital, Uremia Diagnosis and Treatment Center, Shanghai Jiaotong University, School of Medicine, China.

Yin Fan (Y)

Department of Nephrology, Shanghai Sixth People's Hospital, Shanghai Jiao Tong University, School of Medicine, China.

Nan Liu (N)

Department of Nephrology, The First Affiliated Hospital of China Medical University, China.

Zhaohui Huang (Z)

Department of Nephrology, The First Affiliated Hospital of China Medical University, China.

Yali Peng (Y)

Department of Nephrology, The First Affiliated Hospital of China Medical University, China.

Qiuling Fan (Q)

Department of Nephrology, Shanghai General Hospital, Shanghai Jiao Tong University, School of Medicine, China.

Niansong Wang (N)

Department of Nephrology, Shanghai Sixth People's Hospital, Shanghai Jiao Tong University, School of Medicine, China.

Li Yao (L)

Department of Nephrology, The First Affiliated Hospital of China Medical University, China.

Leyi Gu (L)

Department of Nephrology, Molecular Cell Lab for Kidney Disease, Shanghai Peritoneal Dialysis Research Center, Renji Hospital, Uremia Diagnosis and Treatment Center, Shanghai Jiaotong University, School of Medicine, China. Electronic address: guleyi@aliyun.com.

Classifications MeSH